Cortical myoclonus in levodopa‐responsive parkinsonism
Identifieur interne : 007F49 ( Main/Merge ); précédent : 007F48; suivant : 007F50Cortical myoclonus in levodopa‐responsive parkinsonism
Auteurs : John N. Caviness [États-Unis] ; Charles H. Adler [États-Unis] ; Stephanie Newman [États-Unis] ; Richard J. Caselli [États-Unis] ; Manfred D. Muenter [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 1998-05.
English descriptors
- KwdEn :
- Aged, Antiparkinson Agents (administration & dosage), Brain Mapping, Cerebral Cortex (drug effects), Cerebral Cortex (physiopathology), Cortex, Electroencephalography (drug effects), Electromyography (drug effects), Electrophysiology, Follow-Up Studies, Humans, Levodopa (administration & dosage), Male, Myoclonus, Myoclonus (drug therapy), Myoclonus (physiopathology), Parkinson Disease (drug therapy), Parkinson Disease (physiopathology), Parkinson's disease, Parkinsonism.
- MESH :
- chemical , administration & dosage : Antiparkinson Agents, Levodopa.
- drug effects : Cerebral Cortex, Electroencephalography, Electromyography.
- drug therapy : Myoclonus, Parkinson Disease.
- physiopathology : Cerebral Cortex, Myoclonus, Parkinson Disease.
- Aged, Brain Mapping, Follow-Up Studies, Humans, Male.
Abstract
We observed myoclonic movements of the fingers and wrists in two patients with a levodopa‐responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (< 50 ms) myoclonic electromyographic discharges showed a time‐locked relationship to a focal premovement electroencephalographic potential. Somatosensoryevoked potentials were not enlarged and long‐latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.
Url:
DOI: 10.1002/mds.870130327
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<front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa‐responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (< 50 ms) myoclonic electromyographic discharges showed a time‐locked relationship to a focal premovement electroencephalographic potential. Somatosensoryevoked potentials were not enlarged and long‐latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div>
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<front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa-responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (<50 ms) myoclonic electromyographic discharges showed a time-locked relationship to a focal premovement electroencephalographic potential. Somatosensory-evoked potentials were not enlarged and long-latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div>
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