Cortical myoclonus in levodopa‐responsive parkinsonism
Identifieur interne : 007F49 ( Main/Merge ); précédent : 007F48; suivant : 007F50Cortical myoclonus in levodopa‐responsive parkinsonism
Auteurs : John N. Caviness [États-Unis] ; Charles H. Adler [États-Unis] ; Stephanie Newman [États-Unis] ; Richard J. Caselli [États-Unis] ; Manfred D. Muenter [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 1998-05.
English descriptors
- KwdEn :
- Aged, Antiparkinson Agents (administration & dosage), Brain Mapping, Cerebral Cortex (drug effects), Cerebral Cortex (physiopathology), Cortex, Electroencephalography (drug effects), Electromyography (drug effects), Electrophysiology, Follow-Up Studies, Humans, Levodopa (administration & dosage), Male, Myoclonus, Myoclonus (drug therapy), Myoclonus (physiopathology), Parkinson Disease (drug therapy), Parkinson Disease (physiopathology), Parkinson's disease, Parkinsonism.
- MESH :
- chemical , administration & dosage : Antiparkinson Agents, Levodopa.
- drug effects : Cerebral Cortex, Electroencephalography, Electromyography.
- drug therapy : Myoclonus, Parkinson Disease.
- physiopathology : Cerebral Cortex, Myoclonus, Parkinson Disease.
- Aged, Brain Mapping, Follow-Up Studies, Humans, Male.
Abstract
We observed myoclonic movements of the fingers and wrists in two patients with a levodopa‐responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (< 50 ms) myoclonic electromyographic discharges showed a time‐locked relationship to a focal premovement electroencephalographic potential. Somatosensoryevoked potentials were not enlarged and long‐latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.
Url:
DOI: 10.1002/mds.870130327
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Caviness</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Adler, Charles H" sort="Adler, Charles H" uniqKey="Adler C" first="Charles H." last="Adler">Charles H. 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Caselli</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Muenter, Manfred D" sort="Muenter, Manfred D" uniqKey="Muenter M" first="Manfred D." last="Muenter">Manfred D. Muenter</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1998-05">1998-05</date><biblScope unit="vol">13</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="540">540</biblScope><biblScope unit="page" to="544">544</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">D06F7283C58F171A52AAEF4618E141466A448B8B</idno><idno type="DOI">10.1002/mds.870130327</idno><idno type="ArticleID">MDS870130327</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Antiparkinson Agents (administration & dosage)</term><term>Brain Mapping</term><term>Cerebral Cortex (drug effects)</term><term>Cerebral Cortex (physiopathology)</term><term>Cortex</term><term>Electroencephalography (drug effects)</term><term>Electromyography (drug effects)</term><term>Electrophysiology</term><term>Follow-Up Studies</term><term>Humans</term><term>Levodopa (administration & dosage)</term><term>Male</term><term>Myoclonus</term><term>Myoclonus (drug therapy)</term><term>Myoclonus (physiopathology)</term><term>Parkinson Disease (drug therapy)</term><term>Parkinson Disease (physiopathology)</term><term>Parkinson's disease</term><term>Parkinsonism</term></keywords><keywords scheme="MESH" type="chemical" qualifier="administration & dosage" xml:lang="en"><term>Antiparkinson Agents</term><term>Levodopa</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cerebral Cortex</term><term>Electroencephalography</term><term>Electromyography</term></keywords><keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Cerebral Cortex</term><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Brain Mapping</term><term>Follow-Up Studies</term><term>Humans</term><term>Male</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa‐responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (< 50 ms) myoclonic electromyographic discharges showed a time‐locked relationship to a focal premovement electroencephalographic potential. Somatosensoryevoked potentials were not enlarged and long‐latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div></front></TEI><double doi="10.1002/mds.870130327"><ISTEX><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Cortical myoclonus in levodopa‐responsive parkinsonism</title><author><name sortKey="Caviness, John N" sort="Caviness, John N" uniqKey="Caviness J" first="John N." last="Caviness">John N. Caviness</name></author><author><name sortKey="Adler, Charles H" sort="Adler, Charles H" uniqKey="Adler C" first="Charles H." last="Adler">Charles H. Adler</name></author><author><name sortKey="Newman, Stephanie" sort="Newman, Stephanie" uniqKey="Newman S" first="Stephanie" last="Newman">Stephanie Newman</name></author><author><name sortKey="Caselli, Richard J" sort="Caselli, Richard J" uniqKey="Caselli R" first="Richard J." last="Caselli">Richard J. Caselli</name></author><author><name sortKey="Muenter, Manfred D" sort="Muenter, Manfred D" uniqKey="Muenter M" first="Manfred D." last="Muenter">Manfred D. Muenter</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:D06F7283C58F171A52AAEF4618E141466A448B8B</idno><date when="1998" year="1998">1998</date><idno type="doi">10.1002/mds.870130327</idno><idno type="url">https://api.istex.fr/document/D06F7283C58F171A52AAEF4618E141466A448B8B/fulltext/pdf</idno><idno type="wicri:Area/Istex/Corpus">003136</idno><idno type="wicri:Area/Istex/Curation">003136</idno><idno type="wicri:Area/Istex/Checkpoint">003772</idno><idno type="wicri:doubleKey">0885-3185:1998:Caviness J:cortical:myoclonus:in</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Cortical myoclonus in levodopa‐responsive parkinsonism</title><author><name sortKey="Caviness, John N" sort="Caviness, John N" uniqKey="Caviness J" first="John N." last="Caviness">John N. Caviness</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Adler, Charles H" sort="Adler, Charles H" uniqKey="Adler C" first="Charles H." last="Adler">Charles H. Adler</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Newman, Stephanie" sort="Newman, Stephanie" uniqKey="Newman S" first="Stephanie" last="Newman">Stephanie Newman</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Caselli, Richard J" sort="Caselli, Richard J" uniqKey="Caselli R" first="Richard J." last="Caselli">Richard J. Caselli</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author><author><name sortKey="Muenter, Manfred D" sort="Muenter, Manfred D" uniqKey="Muenter M" first="Manfred D." last="Muenter">Manfred D. Muenter</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Scottsdale, Arizona</wicri:regionArea><placeName><region type="state">Arizona</region></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1998-05">1998-05</date><biblScope unit="vol">13</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="540">540</biblScope><biblScope unit="page" to="544">544</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">D06F7283C58F171A52AAEF4618E141466A448B8B</idno><idno type="DOI">10.1002/mds.870130327</idno><idno type="ArticleID">MDS870130327</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Cortex</term><term>Electrophysiology</term><term>Myoclonus</term><term>Parkinson's disease</term><term>Parkinsonism</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa‐responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (< 50 ms) myoclonic electromyographic discharges showed a time‐locked relationship to a focal premovement electroencephalographic potential. Somatosensoryevoked potentials were not enlarged and long‐latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div></front></TEI></ISTEX><PubMed><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Cortical myoclonus in levodopa-responsive parkinsonism.</title><author><name sortKey="Caviness, J N" sort="Caviness, J N" uniqKey="Caviness J" first="J N" last="Caviness">J N Caviness</name><affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259, USA.</nlm:affiliation><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259</wicri:regionArea><wicri:noRegion>Arizona 85259</wicri:noRegion></affiliation></author><author><name sortKey="Adler, C H" sort="Adler, C H" uniqKey="Adler C" first="C H" last="Adler">C H Adler</name></author><author><name sortKey="Newman, S" sort="Newman, S" uniqKey="Newman S" first="S" last="Newman">S. Newman</name></author><author><name sortKey="Caselli, R J" sort="Caselli, R J" uniqKey="Caselli R" first="R J" last="Caselli">R J Caselli</name></author><author><name sortKey="Muenter, M D" sort="Muenter, M D" uniqKey="Muenter M" first="M D" last="Muenter">M D Muenter</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="1998">1998</date><idno type="RBID">pubmed:9613750</idno><idno type="pmid">9613750</idno><idno type="doi">10.1002/mds.870130327</idno><idno type="wicri:Area/PubMed/Corpus">004411</idno><idno type="wicri:Area/PubMed/Curation">004411</idno><idno type="wicri:Area/PubMed/Checkpoint">004468</idno><idno type="wicri:Area/Ncbi/Merge">004F72</idno><idno type="wicri:Area/Ncbi/Curation">004F72</idno><idno type="wicri:Area/Ncbi/Checkpoint">004F72</idno><idno type="wicri:doubleKey">0885-3185:1998:Caviness J:cortical:myoclonus:in</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Cortical myoclonus in levodopa-responsive parkinsonism.</title><author><name sortKey="Caviness, J N" sort="Caviness, J N" uniqKey="Caviness J" first="J N" last="Caviness">J N Caviness</name><affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259, USA.</nlm:affiliation><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259</wicri:regionArea><wicri:noRegion>Arizona 85259</wicri:noRegion></affiliation></author><author><name sortKey="Adler, C H" sort="Adler, C H" uniqKey="Adler C" first="C H" last="Adler">C H Adler</name></author><author><name sortKey="Newman, S" sort="Newman, S" uniqKey="Newman S" first="S" last="Newman">S. Newman</name></author><author><name sortKey="Caselli, R J" sort="Caselli, R J" uniqKey="Caselli R" first="R J" last="Caselli">R J Caselli</name></author><author><name sortKey="Muenter, M D" sort="Muenter, M D" uniqKey="Muenter M" first="M D" last="Muenter">M D Muenter</name></author></analytic><series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title><idno type="ISSN">0885-3185</idno><imprint><date when="1998" type="published">1998</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Antiparkinson Agents (administration & dosage)</term><term>Brain Mapping</term><term>Cerebral Cortex (drug effects)</term><term>Cerebral Cortex (physiopathology)</term><term>Electroencephalography (drug effects)</term><term>Electromyography (drug effects)</term><term>Follow-Up Studies</term><term>Humans</term><term>Levodopa (administration & dosage)</term><term>Male</term><term>Myoclonus (drug therapy)</term><term>Myoclonus (physiopathology)</term><term>Parkinson Disease (drug therapy)</term><term>Parkinson Disease (physiopathology)</term></keywords><keywords scheme="MESH" type="chemical" qualifier="administration & dosage" xml:lang="en"><term>Antiparkinson Agents</term><term>Levodopa</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cerebral Cortex</term><term>Electroencephalography</term><term>Electromyography</term></keywords><keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Cerebral Cortex</term><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Brain Mapping</term><term>Follow-Up Studies</term><term>Humans</term><term>Male</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa-responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (<50 ms) myoclonic electromyographic discharges showed a time-locked relationship to a focal premovement electroencephalographic potential. Somatosensory-evoked potentials were not enlarged and long-latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div></front></TEI></PubMed></double></record>Pour manipuler ce document sous Unix (Dilib)
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