Cortical myoclonus in levodopa-responsive parkinsonism.
Identifieur interne : 004F72 ( Ncbi/Checkpoint ); précédent : 004F71; suivant : 004F73Cortical myoclonus in levodopa-responsive parkinsonism.
Auteurs : J N Caviness [États-Unis] ; C H Adler ; S. Newman ; R J Caselli ; M D MuenterSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 1998.
English descriptors
- KwdEn :
- Aged, Antiparkinson Agents (administration & dosage), Brain Mapping, Cerebral Cortex (drug effects), Cerebral Cortex (physiopathology), Electroencephalography (drug effects), Electromyography (drug effects), Follow-Up Studies, Humans, Levodopa (administration & dosage), Male, Myoclonus (drug therapy), Myoclonus (physiopathology), Parkinson Disease (drug therapy), Parkinson Disease (physiopathology).
- MESH :
- chemical , administration & dosage : Antiparkinson Agents, Levodopa.
- drug effects : Cerebral Cortex, Electroencephalography, Electromyography.
- drug therapy : Myoclonus, Parkinson Disease.
- physiopathology : Cerebral Cortex, Myoclonus, Parkinson Disease.
- Aged, Brain Mapping, Follow-Up Studies, Humans, Male.
Abstract
We observed myoclonic movements of the fingers and wrists in two patients with a levodopa-responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (<50 ms) myoclonic electromyographic discharges showed a time-locked relationship to a focal premovement electroencephalographic potential. Somatosensory-evoked potentials were not enlarged and long-latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.
DOI: 10.1002/mds.870130327
PubMed: 9613750
Affiliations:
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Newman</name></author><author><name sortKey="Caselli, R J" sort="Caselli, R J" uniqKey="Caselli R" first="R J" last="Caselli">R J Caselli</name></author><author><name sortKey="Muenter, M D" sort="Muenter, M D" uniqKey="Muenter M" first="M D" last="Muenter">M D Muenter</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="1998">1998</date><idno type="RBID">pubmed:9613750</idno><idno type="pmid">9613750</idno><idno type="doi">10.1002/mds.870130327</idno><idno type="wicri:Area/PubMed/Corpus">004411</idno><idno type="wicri:Area/PubMed/Curation">004411</idno><idno type="wicri:Area/PubMed/Checkpoint">004468</idno><idno type="wicri:Area/Ncbi/Merge">004F72</idno><idno type="wicri:Area/Ncbi/Curation">004F72</idno><idno type="wicri:Area/Ncbi/Checkpoint">004F72</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Cortical myoclonus in levodopa-responsive parkinsonism.</title><author><name sortKey="Caviness, J N" sort="Caviness, J N" uniqKey="Caviness J" first="J N" last="Caviness">J N Caviness</name><affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259, USA.</nlm:affiliation><country xml:lang="fr">États-Unis</country><wicri:regionArea>Department of Neurology, Mayo Clinic Scottsdale, Arizona 85259</wicri:regionArea><wicri:noRegion>Arizona 85259</wicri:noRegion></affiliation></author><author><name sortKey="Adler, C H" sort="Adler, C H" uniqKey="Adler C" first="C H" last="Adler">C H Adler</name></author><author><name sortKey="Newman, S" sort="Newman, S" uniqKey="Newman S" first="S" last="Newman">S. Newman</name></author><author><name sortKey="Caselli, R J" sort="Caselli, R J" uniqKey="Caselli R" first="R J" last="Caselli">R J Caselli</name></author><author><name sortKey="Muenter, M D" sort="Muenter, M D" uniqKey="Muenter M" first="M D" last="Muenter">M D Muenter</name></author></analytic><series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title><idno type="ISSN">0885-3185</idno><imprint><date when="1998" type="published">1998</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Antiparkinson Agents (administration & dosage)</term><term>Brain Mapping</term><term>Cerebral Cortex (drug effects)</term><term>Cerebral Cortex (physiopathology)</term><term>Electroencephalography (drug effects)</term><term>Electromyography (drug effects)</term><term>Follow-Up Studies</term><term>Humans</term><term>Levodopa (administration & dosage)</term><term>Male</term><term>Myoclonus (drug therapy)</term><term>Myoclonus (physiopathology)</term><term>Parkinson Disease (drug therapy)</term><term>Parkinson Disease (physiopathology)</term></keywords><keywords scheme="MESH" type="chemical" qualifier="administration & dosage" xml:lang="en"><term>Antiparkinson Agents</term><term>Levodopa</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cerebral Cortex</term><term>Electroencephalography</term><term>Electromyography</term></keywords><keywords scheme="MESH" qualifier="drug therapy" xml:lang="en"><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Cerebral Cortex</term><term>Myoclonus</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Brain Mapping</term><term>Follow-Up Studies</term><term>Humans</term><term>Male</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We observed myoclonic movements of the fingers and wrists in two patients with a levodopa-responsive parkinsonian syndrome most consistent with Parkinson's disease. These patients were studied with electrophysiological techniques. Brief (<50 ms) myoclonic electromyographic discharges showed a time-locked relationship to a focal premovement electroencephalographic potential. Somatosensory-evoked potentials were not enlarged and long-latency reflexes were not grossly exaggerated. This pattern of electrophysiological findings can be distinguished from those previously found in other parkinsonian syndromes. These results provide evidence for a cortical origin of the myoclonus seen in these patients.</div></front></TEI><affiliations><list><country><li>États-Unis</li></country></list><tree><noCountry><name sortKey="Adler, C H" sort="Adler, C H" uniqKey="Adler C" first="C H" last="Adler">C H Adler</name><name sortKey="Caselli, R J" sort="Caselli, R J" uniqKey="Caselli R" first="R J" last="Caselli">R J Caselli</name><name sortKey="Muenter, M D" sort="Muenter, M D" uniqKey="Muenter M" first="M D" last="Muenter">M D Muenter</name><name sortKey="Newman, S" sort="Newman, S" uniqKey="Newman S" first="S" last="Newman">S. Newman</name></noCountry><country name="États-Unis"><noRegion><name sortKey="Caviness, J N" sort="Caviness, J N" uniqKey="Caviness J" first="J N" last="Caviness">J N Caviness</name></noRegion></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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