Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint
Identifieur interne : 003D03 ( Main/Merge ); précédent : 003D02; suivant : 003D04Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint
Auteurs : Manuel De Entrambasaguas [Espagne] ; Andrés Plaza-Costa [Espagne] ; Joaquín Casal [Espagne] ; Silvia Parra [Espagne]Source :
- Movement Disorders [ 0885-3185 ] ; 2007-07-15.
English descriptors
- KwdEn :
- Adult, Dystonia (etiology), Dystonia (pathology), Dystonia (rehabilitation), Facial Nerve Diseases (complications), Female, Humans, Labial Frenum (pathology), Maxilla (physiopathology), Palatal Expansion Technique, Trigeminal Nerve Diseases (complications), bruxism, dystonia, facial, neuropathy, splint, trigeminal.
- MESH :
- complications : Facial Nerve Diseases, Trigeminal Nerve Diseases.
- etiology : Dystonia.
- pathology : Dystonia, Labial Frenum.
- physiopathology : Maxilla.
- rehabilitation : Dystonia.
- Adult, Female, Humans, Palatal Expansion Technique.
Abstract
A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society
Url:
DOI: 10.1002/mds.21488
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<front><div type="abstract" xml:lang="en">A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society</div>
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<front><div type="abstract" xml:lang="en">A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society</div>
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<front><div type="abstract" xml:lang="en">A 27-year-old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory-excitatory circuitry, and controlled dystonia.</div>
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