Movement Disorders (revue)

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Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint.

Identifieur interne : 002731 ( PubMed/Corpus ); précédent : 002730; suivant : 002732

Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint.

Auteurs : Manuel De Entrambasaguas ; Andrés Plaza-Costa ; Joaquín Casal ; Silvia Parra

Source :

RBID : pubmed:17486646

English descriptors

Abstract

A 27-year-old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory-excitatory circuitry, and controlled dystonia.

DOI: 10.1002/mds.21488
PubMed: 17486646

Links to Exploration step

pubmed:17486646

Le document en format XML

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<term>Female</term>
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<div type="abstract" xml:lang="en">A 27-year-old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory-excitatory circuitry, and controlled dystonia.</div>
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