Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint
Identifieur interne : 002F60 ( Istex/Corpus ); précédent : 002F59; suivant : 002F61Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint
Auteurs : Manuel De Entrambasaguas ; Andrés Plaza-Costa ; Joaquín Casal ; Silvia ParraSource :
- Movement Disorders [ 0885-3185 ] ; 2007-07-15.
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Abstract
A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society
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DOI: 10.1002/mds.21488
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ISTEX:9B6AFF5D5804101273AA6A4184D63F57514C63ADLe document en format XML
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Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Manuel de Entrambasaguas MD</name><affiliations><json:string>Department of Clinical Neurophysiology, Hospital General de Castellón, Castellón, Spain</json:string></affiliations></json:item><json:item><name>Andrés Plaza‐Costa MD</name><affiliations><json:string>Associate Professor and Professor of the Master in Odontology in Special Patients, University of Valencia Medical and Dental School, Valencia, Spain</json:string></affiliations></json:item><json:item><name>Joaquín Casal MD</name><affiliations><json:string>Odontologist, Private Practice, Valencia, Spain</json:string></affiliations></json:item><json:item><name>Silvia Parra MD</name><affiliations><json:string>Department of Clinical Neurophysiology, Hospital Universitario La Fe, Valencia, Spain</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>bruxism</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>dystonia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>facial</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>neuropathy</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>splint</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>trigeminal</value></json:item></subject><language><json:string>eng</json:string></language><abstract>A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. 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Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. 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The maxillary splint is shown. Dystonia subsides when the patient puts the splint on. Needle electromyography shows spontaneous firing of motor unit action potentials (MUAPs), some of them polyphasic, from left depressor anguli oris (or triangularis) muscle, but not from right depressor anguli oris or left orbicularis oris. When the patient wears the splint, the spontaneous firing of MUAPs subsides and disappears.</caption></supportingInfoItem></supportingInformation><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>A 27‐year‐old woman with bruxism suffered a spider bite (<i>Loxosceles rufescens</i>) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 3-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Labial Dystonia Controlled with Splint</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint</title></titleInfo><name type="personal"><namePart type="given">Manuel</namePart><namePart type="family">de Entrambasaguas</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neurophysiology, Hospital General de Castellón, Castellón, Spain</affiliation><description>Correspondence: Department of Clinical Neurophysiology, Hospital General de Castellón, Avenida de Benicàssim s/n E‐12004 Castellón, Spain</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Andrés</namePart><namePart type="family">Plaza‐Costa</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Associate Professor and Professor of the Master in Odontology in Special Patients, University of Valencia Medical and Dental School, Valencia, Spain</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Joaquín</namePart><namePart type="family">Casal</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Odontologist, Private Practice, Valencia, Spain</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Silvia</namePart><namePart type="family">Parra</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neurophysiology, Hospital Universitario La Fe, Valencia, Spain</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">2007-07-15</dateIssued><dateCaptured encoding="w3cdtf">2007-01-13</dateCaptured><dateValid encoding="w3cdtf">2007-03-02</dateValid><copyrightDate encoding="w3cdtf">2007</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="references">11</extent><extent unit="words">2068</extent></physicalDescription><abstract lang="en">A 27‐year‐old woman with bruxism suffered a spider bite (Loxosceles rufescens) on the left cheek that caused severe local cellulitis, facial palsy, and painful hyperesthesia over the two lower trigeminal nerve divisions. Facial but not trigeminal neuropathy improved, and she developed a labial dystonia that only corrected while pressing the right medial incisor. A specially designed maxillary splint that continuously pressed it suppressed both dystonia and related spontaneous firing of motor unit potentials in electromyography. Overstimulation of the contralateral trigeminal territory possibly compensated for the altered left trigeminal nerve input, balanced proprioceptive influences at the central inhibitory–excitatory circuitry, and controlled dystonia. © 2007 Movement Disorder Society</abstract><subject lang="en"><genre>Keywords</genre><topic>bruxism</topic><topic>dystonia</topic><topic>facial</topic><topic>neuropathy</topic><topic>splint</topic><topic>trigeminal</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title><subTitle>Official Journal of the Movement Disorder Society</subTitle></titleInfo><titleInfo type="abbreviated"><title>Mov. Disord.</title></titleInfo><note type="content"> This article includes supplementary video clips, available online at http://www.interscience.wiley.com/jpages/0885‐3185/suppmat .Supporting Info Item: Segment 1.Dystonic downward deviation of the left labial angle is shown. The maxillary splint is shown. Dystonia subsides when the patient puts the splint on. Needle electromyography shows spontaneous firing of motor unit action potentials (MUAPs), some of them polyphasic, from left depressor anguli oris (or triangularis) muscle, but not from right depressor anguli oris or left orbicularis oris. When the patient wears the splint, the spontaneous firing of MUAPs subsides and disappears. - </note><subject><genre>article category</genre><topic>Brief Report</topic></subject><identifier type="ISSN">0885-3185</identifier><identifier type="eISSN">1531-8257</identifier><identifier type="DOI">10.1002/(ISSN)1531-8257</identifier><identifier type="PublisherID">MDS</identifier><part><date>2007</date><detail type="volume"><caption>vol.</caption><number>22</number></detail><detail type="issue"><caption>no.</caption><number>9</number></detail><extent unit="pages"><start>1355</start><end>1358</end><total>3</total></extent></part></relatedItem><identifier type="istex">9B6AFF5D5804101273AA6A4184D63F57514C63AD</identifier><identifier type="DOI">10.1002/mds.21488</identifier><identifier type="ArticleID">MDS21488</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 2007 Movement Disorder Society</accessCondition><recordInfo><recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin><recordContentSource>WILEY</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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|texte= Labial dystonia after facial and trigeminal neuropathy controlled with a maxillary splint
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