Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome
Identifieur interne : 002F58 ( Main/Exploration ); précédent : 002F57; suivant : 002F59Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome
Auteurs : Steve W. Wu [États-Unis] ; Brent Graham [États-Unis] ; Michael J. Gelfand [États-Unis] ; Ralph E. Gruppo [États-Unis] ; Argirios Dinopolous [États-Unis] ; Donald L. Gilbert [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 2007-09-15.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
- Adolescent, Antiphospholipid antibody syndrome, Attention Deficit Disorder with Hyperactivity (complications), Cardiolipins (immunology), Chorea, Chorea (etiology), Chorea (immunology), Chorea (radionuclide imaging), Emission tomography, Fluorodeoxyglucose F18 (diagnostic use), Humans, Immunoglobulin G (blood), Male, Nervous system diseases, Neurologic Examination, PET imaging, Platelet, Positron emission tomography, Positron-Emission Tomography (methods), Steroid, antiphospholipid antibody syndrome, beta 2-Glycoprotein I (immunology), chorea, steroid.
- MESH :
- chemical , blood : Immunoglobulin G.
- chemical , diagnostic use : Fluorodeoxyglucose F18.
- chemical , immunology : Cardiolipins, beta 2-Glycoprotein I.
- complications : Attention Deficit Disorder with Hyperactivity.
- etiology : Chorea.
- immunology : Chorea.
- methods : Positron-Emission Tomography.
- radionuclide imaging : Chorea.
- Adolescent, Humans, Male, Neurologic Examination.
Abstract
A fourteen‐year‐old right‐handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti‐β2‐glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right‐sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom‐free for 26 months. © 2007 Movement Disorder Society
Url:
DOI: 10.1002/mds.21657
Affiliations:
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Le document en format XML
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<term>Antiphospholipid antibody syndrome</term>
<term>Attention Deficit Disorder with Hyperactivity (complications)</term>
<term>Cardiolipins (immunology)</term>
<term>Chorea</term>
<term>Chorea (etiology)</term>
<term>Chorea (immunology)</term>
<term>Chorea (radionuclide imaging)</term>
<term>Emission tomography</term>
<term>Fluorodeoxyglucose F18 (diagnostic use)</term>
<term>Humans</term>
<term>Immunoglobulin G (blood)</term>
<term>Male</term>
<term>Nervous system diseases</term>
<term>Neurologic Examination</term>
<term>PET imaging</term>
<term>Platelet</term>
<term>Positron emission tomography</term>
<term>Positron-Emission Tomography (methods)</term>
<term>Steroid</term>
<term>antiphospholipid antibody syndrome</term>
<term>beta 2-Glycoprotein I (immunology)</term>
<term>chorea</term>
<term>steroid</term>
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<keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Chorea</term>
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<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Chorea</term>
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<term>Stéroïde</term>
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<front><div type="abstract" xml:lang="en">A fourteen‐year‐old right‐handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti‐β2‐glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right‐sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom‐free for 26 months. © 2007 Movement Disorder Society</div>
</front>
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