MovDisordV3, PascalFrancis, Curation, bibRecord, 001794

Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome

Identifieur interne : 001794 ( PascalFrancis/Curation ); précédent : 001793; suivant : 001795

Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome

Auteurs : Steve W. Wu [États-Unis] ; Brent Graham [États-Unis] ; Michael J. Gelfand [États-Unis] ; Ralph E. Gruppo [États-Unis] ; Argirios Dinopolous [États-Unis] ; Donald L. Gilbert [États-Unis]

Source :

Movement disorders [ 0885-3185 ] ; 2007.

RBID : Pascal:07-0491135

Descripteurs français

Pascal (Inist)
- Système nerveux pathologie, Chorée syndrome, Tomoscintigraphie, Tomographie émission positon, Antiphospholipide syndrome, Stéroïde, Thrombocyte.

English descriptors

KwdEn :
- Antiphospholipid antibody syndrome, Chorea, Emission tomography, Nervous system diseases, Platelet, Positron emission tomography, Steroid.

Abstract

A fourteen-year-old right-handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti-β₂-glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right-sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom-free for 26 months.

A01	`01`	`1`		`@0 0885-3185`
A03		`1`		`@0 Mov. disord.`
A05				`@2 22`
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A08	`01`	`1`	`ENG`	`@1 Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome`
A11	`01`	`1`		`@1 WU (Steve W.)`
A11	`02`	`1`		`@1 GRAHAM (Brent)`
A11	`03`	`1`		`@1 GELFAND (Michael J.)`
A11	`04`	`1`		`@1 GRUPPO (Ralph E.)`
A11	`05`	`1`		`@1 DINOPOLOUS (Argirios)`
A11	`06`	`1`		`@1 GILBERT (Donald L.)`
A14	`01`			`@1 Division of Neurology, Cincinnati Children's Hospital Medical Center @2 Cincinnati, Ohio @3 USA @Z 1 aut. @Z 5 aut. @Z 6 aut.`
A14	`02`			`@1 Division of Rheumatology, Cincinnati Children's Hospital Medical Center @2 Cincinnati, Ohio @3 USA @Z 2 aut.`
A14	`03`			`@1 Division of Radiology, Cincinnati Children's Hospital Medical Center @2 Cincinnati, Ohio @3 USA @Z 3 aut.`
A14	`04`			`@1 Division of Hematology, Cincinnati Children's Hospital Medical Center @2 Cincinnati, Ohio @3 USA @Z 4 aut.`
A20				`@1 1813-1815`
A21				`@1 2007`
A23	`01`			`@0 ENG`
A43	`01`			`@1 INIST @2 20953 @5 354000143464810220`
A44				`@0 0000 @1 © 2007 INIST-CNRS. All rights reserved.`
A45				`@0 9 ref.`
A47	`01`	`1`		`@0 07-0491135`
A60				`@1 P @3 CC`
A61				`@0 A`
A64	`01`	`1`		`@0 Movement disorders`
A66	`01`			`@0 USA`
C01	`01`		`ENG`	@0 A fourteen-year-old right-handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti-β₂-glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right-sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom-free for 26 months.
C02	`01`	`X`		`@0 002B17`
C02	`02`	`X`		`@0 002B24B07`
C02	`03`	`X`		`@0 002B17F`
C03	`01`	`X`	`FRE`	`@0 Système nerveux pathologie @5 01`
C03	`01`	`X`	`ENG`	`@0 Nervous system diseases @5 01`
C03	`01`	`X`	`SPA`	`@0 Sistema nervioso patología @5 01`
C03	`02`	`X`	`FRE`	`@0 Chorée syndrome @5 02`
C03	`02`	`X`	`ENG`	`@0 Chorea @5 02`
C03	`02`	`X`	`SPA`	`@0 Corea síndrome @5 02`
C03	`03`	`X`	`FRE`	`@0 Tomoscintigraphie @5 09`
C03	`03`	`X`	`ENG`	`@0 Emission tomography @5 09`
C03	`03`	`X`	`SPA`	`@0 Tomocentelleografía @5 09`
C03	`04`	`X`	`FRE`	`@0 Tomographie émission positon @5 10`
C03	`04`	`X`	`ENG`	`@0 Positron emission tomography @5 10`
C03	`04`	`X`	`SPA`	`@0 Tomografía emisión positrones @5 10`
C03	`05`	`X`	`FRE`	`@0 Antiphospholipide syndrome @5 11`
C03	`05`	`X`	`ENG`	`@0 Antiphospholipid antibody syndrome @5 11`
C03	`05`	`X`	`SPA`	`@0 Antifosfolípido síndrome @5 11`
C03	`06`	`X`	`FRE`	`@0 Stéroïde @5 12`
C03	`06`	`X`	`ENG`	`@0 Steroid @5 12`
C03	`06`	`X`	`SPA`	`@0 Esteroide @5 12`
C03	`07`	`X`	`FRE`	`@0 Thrombocyte @5 78`
C03	`07`	`X`	`ENG`	`@0 Platelet @5 78`
C03	`07`	`X`	`SPA`	`@0 Trombocito @5 78`
C07	`01`	`X`	`FRE`	`@0 Encéphale pathologie @5 37`
C07	`01`	`X`	`ENG`	`@0 Cerebral disorder @5 37`
C07	`01`	`X`	`SPA`	`@0 Encéfalo patología @5 37`
C07	`02`	`X`	`FRE`	`@0 Extrapyramidal syndrome @5 38`
C07	`02`	`X`	`ENG`	`@0 Extrapyramidal syndrome @5 38`
C07	`02`	`X`	`SPA`	`@0 Extrapiramidal síndrome @5 38`
C07	`03`	`X`	`FRE`	`@0 Mouvement involontaire @5 39`
C07	`03`	`X`	`ENG`	`@0 Involuntary movement @5 39`
C07	`03`	`X`	`SPA`	`@0 Movimiento involuntario @5 39`
C07	`04`	`X`	`FRE`	`@0 Système nerveux central pathologie @5 40`
C07	`04`	`X`	`ENG`	`@0 Central nervous system disease @5 40`
C07	`04`	`X`	`SPA`	`@0 Sistema nervosio central patología @5 40`
C07	`05`	`X`	`FRE`	`@0 Trouble neurologique @5 41`
C07	`05`	`X`	`ENG`	`@0 Neurological disorder @5 41`
C07	`05`	`X`	`SPA`	`@0 Trastorno neurológico @5 41`
C07	`06`	`X`	`FRE`	`@0 Appareil circulatoire pathologie @5 42`
C07	`06`	`X`	`ENG`	`@0 Cardiovascular disease @5 42`
C07	`06`	`X`	`SPA`	`@0 Aparato circulatorio patología @5 42`
C07	`07`	`X`	`FRE`	`@0 Hémopathie @5 43`
C07	`07`	`X`	`ENG`	`@0 Hemopathy @5 43`
C07	`07`	`X`	`SPA`	`@0 Hemopatía @5 43`
C07	`08`	`X`	`FRE`	`@0 Maladie autoimmune @5 44`
C07	`08`	`X`	`ENG`	`@0 Autoimmune disease @5 44`
C07	`08`	`X`	`SPA`	`@0 Enfermedad autoinmune @5 44`
C07	`09`	`X`	`FRE`	`@0 Vaisseau sanguin pathologie @5 45`
C07	`09`	`X`	`ENG`	`@0 Vascular disease @5 45`
C07	`09`	`X`	`SPA`	`@0 Vaso sanguíneo patología @5 45`
C07	`10`	`X`	`FRE`	`@0 Immunopathologie @5 46`
C07	`10`	`X`	`ENG`	`@0 Immunopathology @5 46`
C07	`10`	`X`	`SPA`	`@0 Inmunopatología @5 46`
N21				`@1 323`
N44	`01`			`@1 OTO`
N82				`@1 OTO`

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Le document en format XML

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<author><name sortKey="Graham, Brent" sort="Graham, Brent" uniqKey="Graham B" first="Brent" last="Graham">Brent Graham</name>
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<author><name sortKey="Gelfand, Michael J" sort="Gelfand, Michael J" uniqKey="Gelfand M" first="Michael J." last="Gelfand">Michael J. Gelfand</name>
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<author><name sortKey="Gruppo, Ralph E" sort="Gruppo, Ralph E" uniqKey="Gruppo R" first="Ralph E." last="Gruppo">Ralph E. Gruppo</name>
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<author><name sortKey="Dinopolous, Argirios" sort="Dinopolous, Argirios" uniqKey="Dinopolous A" first="Argirios" last="Dinopolous">Argirios Dinopolous</name>
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<author><name sortKey="Gilbert, Donald L" sort="Gilbert, Donald L" uniqKey="Gilbert D" first="Donald L." last="Gilbert">Donald L. Gilbert</name>
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<seriesStmt><title level="j" type="main">Movement disorders</title>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Antiphospholipid antibody syndrome</term>
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<term>Platelet</term>
<term>Positron emission tomography</term>
<term>Steroid</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Système nerveux pathologie</term>
<term>Chorée syndrome</term>
<term>Tomoscintigraphie</term>
<term>Tomographie émission positon</term>
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<front><div type="abstract" xml:lang="en">A fourteen-year-old right-handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti-β<sub>2</sub>
-glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right-sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom-free for 26 months.</div>
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<fA14 i1="01"><s1>Division of Neurology, Cincinnati Children's Hospital Medical Center</s1>
<s2>Cincinnati, Ohio</s2>
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<fA14 i1="02"><s1>Division of Rheumatology, Cincinnati Children's Hospital Medical Center</s1>
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<fC01 i1="01" l="ENG"><s0>A fourteen-year-old right-handed male with a history of attention deficit hyperactivity disorder (ADHD) presented with alternating hemichorea. Laboratory findings included elevated anticardiolipin IgG and anti-β<sub>2</sub>
-glycoprotein I IgG, which were consistent with primary antiphospholipid antibody syndrome. Positron emission tomography (PET) imaging revealed altered striatal metabolism in his left putamen while he was exhibiting right-sided hemichorea. His symptoms resolved on prednisone; however, his antiphospholipid antibody profile remained markedly abnormal despite being symptom-free for 26 months.</s0>
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<s5>02</s5>
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<s5>09</s5>
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<s5>09</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA"><s0>Tomocentelleografía</s0>
<s5>09</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE"><s0>Tomographie émission positon</s0>
<s5>10</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG"><s0>Positron emission tomography</s0>
<s5>10</s5>
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<fC03 i1="04" i2="X" l="SPA"><s0>Tomografía emisión positrones</s0>
<s5>10</s5>
</fC03>
<fC03 i1="05" i2="X" l="FRE"><s0>Antiphospholipide syndrome</s0>
<s5>11</s5>
</fC03>
<fC03 i1="05" i2="X" l="ENG"><s0>Antiphospholipid antibody syndrome</s0>
<s5>11</s5>
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</fC03>
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<s5>12</s5>
</fC03>
<fC03 i1="07" i2="X" l="FRE"><s0>Thrombocyte</s0>
<s5>78</s5>
</fC03>
<fC03 i1="07" i2="X" l="ENG"><s0>Platelet</s0>
<s5>78</s5>
</fC03>
<fC03 i1="07" i2="X" l="SPA"><s0>Trombocito</s0>
<s5>78</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE"><s0>Encéphale pathologie</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG"><s0>Cerebral disorder</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA"><s0>Encéfalo patología</s0>
<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE"><s0>Extrapyramidal syndrome</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG"><s0>Extrapyramidal syndrome</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA"><s0>Extrapiramidal síndrome</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Mouvement involontaire</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Involuntary movement</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Movimiento involuntario</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Système nerveux central pathologie</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Central nervous system disease</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Sistema nervosio central patología</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Trouble neurologique</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG"><s0>Neurological disorder</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA"><s0>Trastorno neurológico</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE"><s0>Appareil circulatoire pathologie</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG"><s0>Cardiovascular disease</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA"><s0>Aparato circulatorio patología</s0>
<s5>42</s5>
</fC07>
<fC07 i1="07" i2="X" l="FRE"><s0>Hémopathie</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG"><s0>Hemopathy</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA"><s0>Hemopatía</s0>
<s5>43</s5>
</fC07>
<fC07 i1="08" i2="X" l="FRE"><s0>Maladie autoimmune</s0>
<s5>44</s5>
</fC07>
<fC07 i1="08" i2="X" l="ENG"><s0>Autoimmune disease</s0>
<s5>44</s5>
</fC07>
<fC07 i1="08" i2="X" l="SPA"><s0>Enfermedad autoinmune</s0>
<s5>44</s5>
</fC07>
<fC07 i1="09" i2="X" l="FRE"><s0>Vaisseau sanguin pathologie</s0>
<s5>45</s5>
</fC07>
<fC07 i1="09" i2="X" l="ENG"><s0>Vascular disease</s0>
<s5>45</s5>
</fC07>
<fC07 i1="09" i2="X" l="SPA"><s0>Vaso sanguíneo patología</s0>
<s5>45</s5>
</fC07>
<fC07 i1="10" i2="X" l="FRE"><s0>Immunopathologie</s0>
<s5>46</s5>
</fC07>
<fC07 i1="10" i2="X" l="ENG"><s0>Immunopathology</s0>
<s5>46</s5>
</fC07>
<fC07 i1="10" i2="X" l="SPA"><s0>Inmunopatología</s0>
<s5>46</s5>
</fC07>
<fN21><s1>323</s1>
</fN21>
<fN44 i1="01"><s1>OTO</s1>
</fN44>
<fN82><s1>OTO</s1>
</fN82>
</pA>
</standard>
</inist>
</record>

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   |texte=   Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome
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	Movement Disorders (revue)
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Movement Disorders (revue)

Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome

Clinical and positron emission tomography findings of chorea associated with primary antiphospholipid antibody syndrome

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