Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Identifieur interne : 002F93 ( Main/Exploration ); précédent : 002F92; suivant : 002F94Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Auteurs : Julia Roggendorf [Allemagne] ; Lothar Burghaus [Allemagne] ; Wei-Chi Liu [Allemagne] ; Simon Weisenbach [Allemagne] ; Carsten Eggers [Allemagne] ; Gereon R. Fink [Allemagne] ; Ruediger Hilker [Allemagne]Source :
- Movement Disorders [ 0885-3185 ] ; 2007-04-30.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
- Adult, Benzothiazoles (therapeutic use), Brain (pathology), Central pontine myelinolysis, Dancing, Female, Humans, Hyponatremia (etiology), Magnetic Resonance Imaging, Movement Disorders (drug therapy), Movement Disorders (etiology), Myelinolysis, Central Pontine (diagnosis), Nervous system diseases, Pons (pathology), Restless Legs Syndrome (drug therapy), belly dancer's syndrome, central pontine and extrapontine myelinolysis, hyponatriemia.
- MESH :
- chemical , therapeutic use : Benzothiazoles.
- diagnosis : Myelinolysis, Central Pontine.
- drug therapy : Movement Disorders, Restless Legs Syndrome.
- etiology : Hyponatremia, Movement Disorders.
- pathology : Brain, Pons.
- Adult, Dancing, Female, Humans, Magnetic Resonance Imaging.
Abstract
We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society
Url:
DOI: 10.1002/mds.21394
Affiliations:
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Le document en format XML
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<term>Dancing</term>
<term>Female</term>
<term>Humans</term>
<term>Hyponatremia (etiology)</term>
<term>Magnetic Resonance Imaging</term>
<term>Movement Disorders (drug therapy)</term>
<term>Movement Disorders (etiology)</term>
<term>Myelinolysis, Central Pontine (diagnosis)</term>
<term>Nervous system diseases</term>
<term>Pons (pathology)</term>
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<term>belly dancer's syndrome</term>
<term>central pontine and extrapontine myelinolysis</term>
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<front><div type="abstract" xml:lang="en">We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society</div>
</front>
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<tree><country name="Allemagne"><noRegion><name sortKey="Roggendorf, Julia" sort="Roggendorf, Julia" uniqKey="Roggendorf J" first="Julia" last="Roggendorf">Julia Roggendorf</name>
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<name sortKey="Burghaus, Lothar" sort="Burghaus, Lothar" uniqKey="Burghaus L" first="Lothar" last="Burghaus">Lothar Burghaus</name>
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<name sortKey="Fink, Gereon R" sort="Fink, Gereon R" uniqKey="Fink G" first="Gereon R." last="Fink">Gereon R. Fink</name>
<name sortKey="Hilker, Ruediger" sort="Hilker, Ruediger" uniqKey="Hilker R" first="Ruediger" last="Hilker">Ruediger Hilker</name>
<name sortKey="Liu, Wei Hi" sort="Liu, Wei Hi" uniqKey="Liu W" first="Wei-Chi" last="Liu">Wei-Chi Liu</name>
<name sortKey="Weisenbach, Simon" sort="Weisenbach, Simon" uniqKey="Weisenbach S" first="Simon" last="Weisenbach">Simon Weisenbach</name>
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