Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Identifieur interne : 001B01 ( Istex/Checkpoint ); précédent : 001B00; suivant : 001B02Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Auteurs : Julia Roggendorf [Allemagne] ; Lothar Burghaus [Allemagne] ; Wei-Chi Liu [Allemagne] ; Simon Weisenbach [Allemagne] ; Carsten Eggers [Allemagne] ; Gereon R. Fink [Allemagne] ; Ruediger Hilker [Allemagne]Source :
- Movement Disorders [ 0885-3185 ] ; 2007-04-30.
English descriptors
Abstract
We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society
Url:
DOI: 10.1002/mds.21394
Affiliations:
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ISTEX:C313BA1692775C1DAE3905305CDCD287D31556C4Le document en format XML
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<front><div type="abstract" xml:lang="en">We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society</div>
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