Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Identifieur interne : 003835 ( Istex/Corpus ); précédent : 003834; suivant : 003836Belly dancer's syndrome following central pontine and extrapontine myelinolysis
Auteurs : Julia Roggendorf ; Lothar Burghaus ; Wei-Chi Liu ; Simon Weisenbach ; Carsten Eggers ; Gereon R. Fink ; Ruediger HilkerSource :
- Movement Disorders [ 0885-3185 ] ; 2007-04-30.
English descriptors
Abstract
We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society
Url:
DOI: 10.1002/mds.21394
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ISTEX:C313BA1692775C1DAE3905305CDCD287D31556C4Le document en format XML
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Fink</name><affiliation><mods:affiliation>Department of Neurology, University Hospital, Cologne, Germany</mods:affiliation></affiliation></author><author><name sortKey="Hilker, Ruediger" sort="Hilker, Ruediger" uniqKey="Hilker R" first="Ruediger" last="Hilker">Ruediger Hilker</name><affiliation><mods:affiliation>Department of Neurology, University Hospital, Cologne, Germany</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Movement Disorders</title><title level="j" type="abbrev">Mov. Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2007-04-30">2007-04-30</date><biblScope unit="vol">22</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="892">892</biblScope><biblScope unit="page" to="894">894</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">C313BA1692775C1DAE3905305CDCD287D31556C4</idno><idno type="DOI">10.1002/mds.21394</idno><idno type="ArticleID">MDS21394</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>belly dancer's syndrome</term><term>central pontine and extrapontine myelinolysis</term><term>hyponatriemia</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Julia Roggendorf MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Lothar Burghaus MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Wei‐Chi Liu MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Simon Weisenbach MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Carsten Eggers MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Gereon R. Fink MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item><json:item><name>Ruediger Hilker MD</name><affiliations><json:string>Department of Neurology, University Hospital, Cologne, Germany</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>central pontine and extrapontine myelinolysis</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>belly dancer's syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>hyponatriemia</value></json:item></subject><language><json:string>eng</json:string></language><abstract>We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. 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This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><!--Version 0.6 générée le 4-12-2015--><mods version="3.6"><titleInfo lang="en"><title>Belly dancer's syndrome following central pontine and extrapontine myelinolysis</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Belly Dancer's Diskinesia in CPM/EPM</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Belly dancer's syndrome following central pontine and extrapontine myelinolysis</title></titleInfo><name type="personal"><namePart type="given">Julia</namePart><namePart type="family">Roggendorf</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Lothar</namePart><namePart type="family">Burghaus</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Wei‐Chi</namePart><namePart type="family">Liu</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Simon</namePart><namePart type="family">Weisenbach</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Carsten</namePart><namePart type="family">Eggers</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Gereon R.</namePart><namePart type="family">Fink</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Ruediger</namePart><namePart type="family">Hilker</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Neurology, University Hospital, Cologne, Germany</affiliation><description>Correspondence: Department of Neurology, University Hospital, Kerpener Street 62, 50924 Cologne, Germany</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre authority="originalCategForm">article</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">Hoboken</placeTerm></place><dateIssued encoding="w3cdtf">2007-04-30</dateIssued><dateCaptured encoding="w3cdtf">2006-11-27</dateCaptured><dateValid encoding="w3cdtf">2006-12-18</dateValid><copyrightDate encoding="w3cdtf">2007</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="references">13</extent><extent unit="words">1354</extent></physicalDescription><abstract lang="en">We report on a woman with delayed‐onset of belly dancer's syndrome 5 months after central pontine and extrapontine myelinolysis (CPM/EPM) and severe hyponatriemia. This case demonstrates that basal ganglia lesions in EPM can be the underlying pathoanatomic substrate for the rarely observed belly dancer's syndrome. The sequential appearance of extrapyramidal symptoms might reflect an ongoing but ineffective or deficient remyelination process. The presence of CPM/EPM should be considered in patients with involuntary dyskinesias of the abdominal wall. © 2007 Movement Disorder Society</abstract><subject lang="en"><genre>Keywords</genre><topic>central pontine and extrapontine myelinolysis</topic><topic>belly dancer's syndrome</topic><topic>hyponatriemia</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title></titleInfo><titleInfo type="abbreviated"><title>Mov. Disord.</title></titleInfo><note type="content"> This article contains supplementary video clips, available online at http://www.interscience.wiley.com/jpages/0885‐3185/suppmat .</note><subject><genre>article category</genre><topic>Brief Report</topic></subject><identifier type="ISSN">0885-3185</identifier><identifier type="eISSN">1531-8257</identifier><identifier type="DOI">10.1002/(ISSN)1531-8257</identifier><identifier type="PublisherID">MDS</identifier><part><date>2007</date><detail type="volume"><caption>vol.</caption><number>22</number></detail><detail type="issue"><caption>no.</caption><number>6</number></detail><extent unit="pages"><start>892</start><end>894</end><total>3</total></extent></part></relatedItem><identifier type="istex">C313BA1692775C1DAE3905305CDCD287D31556C4</identifier><identifier type="DOI">10.1002/mds.21394</identifier><identifier type="ArticleID">MDS21394</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 2007 Movement Disorder Society</accessCondition><recordInfo><recordOrigin>Wiley Subscription Services, Inc., A Wiley Company</recordOrigin><recordContentSource>WILEY</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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|texte= Belly dancer's syndrome following central pontine and extrapontine myelinolysis
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