Bilateral pedunculopontine nuclei strokes presenting as freezing of gait
Identifieur interne : 002974 ( Main/Exploration ); précédent : 002973; suivant : 002975Bilateral pedunculopontine nuclei strokes presenting as freezing of gait
Auteurs : Sheng-Han Kuo [États-Unis] ; Christopher Kenney [États-Unis] ; Joseph Jankovic [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 2008-03-15.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Congélation.
English descriptors
- KwdEn :
- Aged, Cognition Disorders (diagnosis), Cognition Disorders (etiology), Freezing, Freezing Reaction, Cataleptic (physiology), Functional Laterality (physiology), Gait (physiology), Humans, Locomotion (physiology), Magnetic Resonance Imaging, Male, Nervous system diseases, Neuropsychological Tests, Parkinson Disease (diagnosis), Parkinson Disease (etiology), Parkinson Disease (physiopathology), Pedunculopontine Tegmental Nucleus (blood supply), Pedunculopontine Tegmental Nucleus (pathology), Pedunculopontine Tegmental Nucleus (physiopathology), Stroke, Stroke (complications), Stroke (pathology), Stroke (physiopathology), Supranuclear Palsy, Progressive (diagnosis), Supranuclear Palsy, Progressive (etiology), Supranuclear Palsy, Progressive (physiopathology), Videotape Recording, freezing of gait, pedunculopontine nucleus, stroke.
- MESH :
- blood supply : Pedunculopontine Tegmental Nucleus.
- complications : Stroke.
- diagnosis : Cognition Disorders, Parkinson Disease, Supranuclear Palsy, Progressive.
- etiology : Cognition Disorders, Parkinson Disease, Supranuclear Palsy, Progressive.
- pathology : Pedunculopontine Tegmental Nucleus, Stroke.
- physiology : Freezing Reaction, Cataleptic, Functional Laterality, Gait, Locomotion.
- physiopathology : Parkinson Disease, Pedunculopontine Tegmental Nucleus, Stroke, Supranuclear Palsy, Progressive.
- Aged, Humans, Magnetic Resonance Imaging, Male, Neuropsychological Tests, Videotape Recording.
Abstract
The penduculopontine nucleus (PPN) has been suggested to play an important role in locomotion, based on animal studies, but its function in humans has not been well defined. Autopsy studies have suggested that PPN pathology correlates with gait dysfunction in Parkinson's disease and in progressive supranuclear palsy but direct clinical evidence is lacking. We report a patient with bilateral PPN infarcts whose dominant clinical feature was freezing of gait, thus providing evidence that PPN is involved in human locomotion and that damage to the PPN may lead to abnormal gait. © 2008 Movement Disorder Society
Url:
DOI: 10.1002/mds.21917
Affiliations:
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Le document en format XML
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Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2008-03-15">2008-03-15</date><biblScope unit="vol">23</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="616">616</biblScope><biblScope unit="page" to="619">619</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">C89997473CF70FB2E4FE0E3B24B5D070F395B813</idno><idno type="DOI">10.1002/mds.21917</idno><idno type="ArticleID">MDS21917</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Cognition Disorders (diagnosis)</term><term>Cognition Disorders (etiology)</term><term>Freezing</term><term>Freezing Reaction, Cataleptic (physiology)</term><term>Functional Laterality (physiology)</term><term>Gait (physiology)</term><term>Humans</term><term>Locomotion (physiology)</term><term>Magnetic Resonance Imaging</term><term>Male</term><term>Nervous system diseases</term><term>Neuropsychological Tests</term><term>Parkinson Disease (diagnosis)</term><term>Parkinson Disease (etiology)</term><term>Parkinson Disease (physiopathology)</term><term>Pedunculopontine Tegmental Nucleus (blood supply)</term><term>Pedunculopontine Tegmental Nucleus (pathology)</term><term>Pedunculopontine Tegmental Nucleus (physiopathology)</term><term>Stroke</term><term>Stroke (complications)</term><term>Stroke (pathology)</term><term>Stroke (physiopathology)</term><term>Supranuclear Palsy, Progressive (diagnosis)</term><term>Supranuclear Palsy, Progressive (etiology)</term><term>Supranuclear Palsy, Progressive (physiopathology)</term><term>Videotape Recording</term><term>freezing of gait</term><term>pedunculopontine nucleus</term><term>stroke</term></keywords><keywords scheme="MESH" qualifier="blood supply" xml:lang="en"><term>Pedunculopontine Tegmental Nucleus</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Stroke</term></keywords><keywords scheme="MESH" qualifier="diagnosis" xml:lang="en"><term>Cognition Disorders</term><term>Parkinson Disease</term><term>Supranuclear Palsy, Progressive</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Cognition Disorders</term><term>Parkinson Disease</term><term>Supranuclear Palsy, Progressive</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Pedunculopontine Tegmental Nucleus</term><term>Stroke</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Freezing Reaction, Cataleptic</term><term>Functional Laterality</term><term>Gait</term><term>Locomotion</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Parkinson Disease</term><term>Pedunculopontine Tegmental Nucleus</term><term>Stroke</term><term>Supranuclear Palsy, Progressive</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Humans</term><term>Magnetic Resonance Imaging</term><term>Male</term><term>Neuropsychological Tests</term><term>Videotape Recording</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Accident cérébrovasculaire</term><term>Congélation</term><term>Pathologie du système nerveux</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Congélation</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">The penduculopontine nucleus (PPN) has been suggested to play an important role in locomotion, based on animal studies, but its function in humans has not been well defined. Autopsy studies have suggested that PPN pathology correlates with gait dysfunction in Parkinson's disease and in progressive supranuclear palsy but direct clinical evidence is lacking. We report a patient with bilateral PPN infarcts whose dominant clinical feature was freezing of gait, thus providing evidence that PPN is involved in human locomotion and that damage to the PPN may lead to abnormal gait. © 2008 Movement Disorder Society</div></front></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Texas</li></region><settlement><li>Houston</li></settlement><orgName><li>Baylor College of Medicine</li></orgName></list><tree><country name="États-Unis"><region name="Texas"><name sortKey="Kuo, Sheng An" sort="Kuo, Sheng An" uniqKey="Kuo S" first="Sheng-Han" last="Kuo">Sheng-Han Kuo</name></region><name sortKey="Jankovic, Joseph" sort="Jankovic, Joseph" uniqKey="Jankovic J" first="Joseph" last="Jankovic">Joseph Jankovic</name><name sortKey="Kenney, Christopher" sort="Kenney, Christopher" uniqKey="Kenney C" first="Christopher" last="Kenney">Christopher Kenney</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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