Expression and bioactivities of endothelin-1 in gingiva during cyclosporine A treatment.
Identifieur interne : 005855 ( Main/Merge ); précédent : 005854; suivant : 005856Expression and bioactivities of endothelin-1 in gingiva during cyclosporine A treatment.
Auteurs : Y-T Chin [République populaire de Chine] ; H-P Tu ; Y-T Chen ; N-T Dai ; E-C Shen ; C-Y Chiang ; M M-J Fu ; E. FuSource :
- Journal of periodontal research [ 1600-0765 ] ; 2009.
Descripteurs français
- KwdFr :
- Animaux, Antagonistes du récepteur de type A de l'endothéline, Antagonistes du récepteur de type B de l'endothéline, Antigène nucléaire de prolifération cellulaire (analyse), Bouche édentée (anatomopathologie), Cellules cultivées, Ciclosporine (pharmacologie), Endothéline-1 (analyse), Endothéline-1 (métabolisme), Fibroblastes (), Fibroblastes (anatomopathologie), Gencive (), Gencive (anatomopathologie), Humains, Immunosuppresseurs (pharmacologie), Mâle, Nitric oxide synthase type II (analyse), Prolifération cellulaire (), Rats, Récepteur de l'endothéline de type B (analyse), Récepteur de type A de l'endothéline (analyse), Régulation positive.
- MESH :
- analyse : Antigène nucléaire de prolifération cellulaire, Endothéline-1, Nitric oxide synthase type II, Récepteur de l'endothéline de type B, Récepteur de type A de l'endothéline.
- anatomopathologie : Bouche édentée, Fibroblastes, Gencive.
- métabolisme : Endothéline-1.
- pharmacologie : Ciclosporine, Immunosuppresseurs.
- Animaux, Antagonistes du récepteur de type A de l'endothéline, Antagonistes du récepteur de type B de l'endothéline, Cellules cultivées, Fibroblastes, Gencive, Humains, Mâle, Prolifération cellulaire, Rats, Régulation positive.
English descriptors
- KwdEn :
- Animals, Cell Proliferation (drug effects), Cells, Cultured, Cyclosporine (pharmacology), Endothelin A Receptor Antagonists, Endothelin B Receptor Antagonists, Endothelin-1 (analysis), Endothelin-1 (metabolism), Fibroblasts (drug effects), Fibroblasts (pathology), Gingiva (drug effects), Gingiva (pathology), Humans, Immunosuppressive Agents (pharmacology), Male, Mouth, Edentulous (pathology), Nitric Oxide Synthase Type II (analysis), Proliferating Cell Nuclear Antigen (analysis), Rats, Receptor, Endothelin A (analysis), Receptor, Endothelin B (analysis), Up-Regulation.
- MESH :
- chemical , analysis : Endothelin-1, Nitric Oxide Synthase Type II, Proliferating Cell Nuclear Antigen, Receptor, Endothelin A, Receptor, Endothelin B.
- chemical , metabolism : Endothelin-1.
- chemical , pharmacology : Cyclosporine, Immunosuppressive Agents.
- drug effects : Cell Proliferation, Fibroblasts, Gingiva.
- pathology : Fibroblasts, Gingiva, Mouth, Edentulous.
- Animals, Cells, Cultured, Endothelin A Receptor Antagonists, Endothelin B Receptor Antagonists, Humans, Male, Rats, Up-Regulation.
Abstract
This study aimed to evaluate the expression and bioactivities of endothelin-1 (ET-1) in gingiva during cyclosporine A (CsA) treatment.
DOI: 10.1111/j.1600-0765.2007.01058.x
PubMed: 18973526
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pubmed:18973526Le document en format XML
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Fu</name></author></analytic><series><title level="j">Journal of periodontal research</title><idno type="eISSN">1600-0765</idno><imprint><date when="2009" type="published">2009</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term><term>Cell Proliferation (drug effects)</term><term>Cells, Cultured</term><term>Cyclosporine (pharmacology)</term><term>Endothelin A Receptor Antagonists</term><term>Endothelin B Receptor Antagonists</term><term>Endothelin-1 (analysis)</term><term>Endothelin-1 (metabolism)</term><term>Fibroblasts (drug effects)</term><term>Fibroblasts (pathology)</term><term>Gingiva (drug effects)</term><term>Gingiva (pathology)</term><term>Humans</term><term>Immunosuppressive Agents (pharmacology)</term><term>Male</term><term>Mouth, Edentulous (pathology)</term><term>Nitric Oxide Synthase Type II (analysis)</term><term>Proliferating Cell Nuclear Antigen (analysis)</term><term>Rats</term><term>Receptor, Endothelin A (analysis)</term><term>Receptor, Endothelin B (analysis)</term><term>Up-Regulation</term></keywords><keywords scheme="KwdFr" xml:lang="fr"><term>Animaux</term><term>Antagonistes du récepteur de type A de l'endothéline</term><term>Antagonistes du récepteur de type B de l'endothéline</term><term>Antigène nucléaire de prolifération cellulaire (analyse)</term><term>Bouche édentée (anatomopathologie)</term><term>Cellules cultivées</term><term>Ciclosporine (pharmacologie)</term><term>Endothéline-1 (analyse)</term><term>Endothéline-1 (métabolisme)</term><term>Fibroblastes ()</term><term>Fibroblastes (anatomopathologie)</term><term>Gencive ()</term><term>Gencive (anatomopathologie)</term><term>Humains</term><term>Immunosuppresseurs (pharmacologie)</term><term>Mâle</term><term>Nitric oxide synthase type II (analyse)</term><term>Prolifération cellulaire ()</term><term>Rats</term><term>Récepteur de l'endothéline de type B (analyse)</term><term>Récepteur de type A de l'endothéline (analyse)</term><term>Régulation positive</term></keywords><keywords scheme="MESH" type="chemical" qualifier="analysis" xml:lang="en"><term>Endothelin-1</term><term>Nitric Oxide Synthase Type II</term><term>Proliferating Cell Nuclear Antigen</term><term>Receptor, Endothelin A</term><term>Receptor, Endothelin B</term></keywords><keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Endothelin-1</term></keywords><keywords scheme="MESH" type="chemical" qualifier="pharmacology" xml:lang="en"><term>Cyclosporine</term><term>Immunosuppressive Agents</term></keywords><keywords scheme="MESH" qualifier="analyse" xml:lang="fr"><term>Antigène nucléaire de prolifération cellulaire</term><term>Endothéline-1</term><term>Nitric oxide synthase type II</term><term>Récepteur de l'endothéline de type B</term><term>Récepteur de type A de l'endothéline</term></keywords><keywords scheme="MESH" qualifier="anatomopathologie" xml:lang="fr"><term>Bouche édentée</term><term>Fibroblastes</term><term>Gencive</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cell Proliferation</term><term>Fibroblasts</term><term>Gingiva</term></keywords><keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Endothéline-1</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Fibroblasts</term><term>Gingiva</term><term>Mouth, Edentulous</term></keywords><keywords scheme="MESH" qualifier="pharmacologie" xml:lang="fr"><term>Ciclosporine</term><term>Immunosuppresseurs</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Animals</term><term>Cells, Cultured</term><term>Endothelin A Receptor Antagonists</term><term>Endothelin B Receptor Antagonists</term><term>Humans</term><term>Male</term><term>Rats</term><term>Up-Regulation</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Animaux</term><term>Antagonistes du récepteur de type A de l'endothéline</term><term>Antagonistes du récepteur de type B de l'endothéline</term><term>Cellules cultivées</term><term>Fibroblastes</term><term>Gencive</term><term>Humains</term><term>Mâle</term><term>Prolifération cellulaire</term><term>Rats</term><term>Régulation positive</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">This study aimed to evaluate the expression and bioactivities of endothelin-1 (ET-1) in gingiva during cyclosporine A (CsA) treatment.</div></front></TEI></record>Pour manipuler ce document sous Unix (Dilib)
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