Expression and bioactivities of endothelin-1 in gingiva during cyclosporine A treatment.
Identifieur interne : 001E29 ( PubMed/Curation ); précédent : 001E28; suivant : 001E30Expression and bioactivities of endothelin-1 in gingiva during cyclosporine A treatment.
Auteurs : Y-T Chin [République populaire de Chine] ; H-P Tu ; Y-T Chen ; N-T Dai ; E-C Shen ; C-Y Chiang ; M M-J Fu ; E. FuSource :
- Journal of periodontal research [ 1600-0765 ] ; 2009.
Descripteurs français
- KwdFr :
- Animaux, Antagonistes du récepteur de type A de l'endothéline, Antagonistes du récepteur de type B de l'endothéline, Antigène nucléaire de prolifération cellulaire (analyse), Bouche édentée (anatomopathologie), Cellules cultivées, Ciclosporine (pharmacologie), Endothéline-1 (analyse), Endothéline-1 (métabolisme), Fibroblastes (), Fibroblastes (anatomopathologie), Gencive (), Gencive (anatomopathologie), Humains, Immunosuppresseurs (pharmacologie), Mâle, Nitric oxide synthase type II (analyse), Prolifération cellulaire (), Rats, Récepteur de l'endothéline de type B (analyse), Récepteur de type A de l'endothéline (analyse), Régulation positive.
- MESH :
- analyse : Antigène nucléaire de prolifération cellulaire, Endothéline-1, Nitric oxide synthase type II, Récepteur de l'endothéline de type B, Récepteur de type A de l'endothéline.
- anatomopathologie : Bouche édentée, Fibroblastes, Gencive.
- métabolisme : Endothéline-1.
- pharmacologie : Ciclosporine, Immunosuppresseurs.
- Animaux, Antagonistes du récepteur de type A de l'endothéline, Antagonistes du récepteur de type B de l'endothéline, Cellules cultivées, Fibroblastes, Gencive, Humains, Mâle, Prolifération cellulaire, Rats, Régulation positive.
English descriptors
- KwdEn :
- Animals, Cell Proliferation (drug effects), Cells, Cultured, Cyclosporine (pharmacology), Endothelin A Receptor Antagonists, Endothelin B Receptor Antagonists, Endothelin-1 (analysis), Endothelin-1 (metabolism), Fibroblasts (drug effects), Fibroblasts (pathology), Gingiva (drug effects), Gingiva (pathology), Humans, Immunosuppressive Agents (pharmacology), Male, Mouth, Edentulous (pathology), Nitric Oxide Synthase Type II (analysis), Proliferating Cell Nuclear Antigen (analysis), Rats, Receptor, Endothelin A (analysis), Receptor, Endothelin B (analysis), Up-Regulation.
- MESH :
- chemical , analysis : Endothelin-1, Nitric Oxide Synthase Type II, Proliferating Cell Nuclear Antigen, Receptor, Endothelin A, Receptor, Endothelin B.
- chemical , metabolism : Endothelin-1.
- chemical , pharmacology : Cyclosporine, Immunosuppressive Agents.
- drug effects : Cell Proliferation, Fibroblasts, Gingiva.
- pathology : Fibroblasts, Gingiva, Mouth, Edentulous.
- Animals, Cells, Cultured, Endothelin A Receptor Antagonists, Endothelin B Receptor Antagonists, Humans, Male, Rats, Up-Regulation.
Abstract
This study aimed to evaluate the expression and bioactivities of endothelin-1 (ET-1) in gingiva during cyclosporine A (CsA) treatment.
DOI: 10.1111/j.1600-0765.2007.01058.x
PubMed: 18973526
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pubmed:18973526Le document en format XML
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<term>Cells, Cultured</term>
<term>Cyclosporine (pharmacology)</term>
<term>Endothelin A Receptor Antagonists</term>
<term>Endothelin B Receptor Antagonists</term>
<term>Endothelin-1 (analysis)</term>
<term>Endothelin-1 (metabolism)</term>
<term>Fibroblasts (drug effects)</term>
<term>Fibroblasts (pathology)</term>
<term>Gingiva (drug effects)</term>
<term>Gingiva (pathology)</term>
<term>Humans</term>
<term>Immunosuppressive Agents (pharmacology)</term>
<term>Male</term>
<term>Mouth, Edentulous (pathology)</term>
<term>Nitric Oxide Synthase Type II (analysis)</term>
<term>Proliferating Cell Nuclear Antigen (analysis)</term>
<term>Rats</term>
<term>Receptor, Endothelin A (analysis)</term>
<term>Receptor, Endothelin B (analysis)</term>
<term>Up-Regulation</term>
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<keywords scheme="KwdFr" xml:lang="fr"><term>Animaux</term>
<term>Antagonistes du récepteur de type A de l'endothéline</term>
<term>Antagonistes du récepteur de type B de l'endothéline</term>
<term>Antigène nucléaire de prolifération cellulaire (analyse)</term>
<term>Bouche édentée (anatomopathologie)</term>
<term>Cellules cultivées</term>
<term>Ciclosporine (pharmacologie)</term>
<term>Endothéline-1 (analyse)</term>
<term>Endothéline-1 (métabolisme)</term>
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<term>Gencive (anatomopathologie)</term>
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<term>Récepteur de l'endothéline de type B (analyse)</term>
<term>Récepteur de type A de l'endothéline (analyse)</term>
<term>Régulation positive</term>
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<term>Nitric Oxide Synthase Type II</term>
<term>Proliferating Cell Nuclear Antigen</term>
<term>Receptor, Endothelin A</term>
<term>Receptor, Endothelin B</term>
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<term>Immunosuppressive Agents</term>
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<term>Récepteur de l'endothéline de type B</term>
<term>Récepteur de type A de l'endothéline</term>
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<keywords scheme="MESH" qualifier="anatomopathologie" xml:lang="fr"><term>Bouche édentée</term>
<term>Fibroblastes</term>
<term>Gencive</term>
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<keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Cell Proliferation</term>
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<term>Gingiva</term>
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<term>Cells, Cultured</term>
<term>Endothelin A Receptor Antagonists</term>
<term>Endothelin B Receptor Antagonists</term>
<term>Humans</term>
<term>Male</term>
<term>Rats</term>
<term>Up-Regulation</term>
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<keywords scheme="MESH" xml:lang="fr"><term>Animaux</term>
<term>Antagonistes du récepteur de type A de l'endothéline</term>
<term>Antagonistes du récepteur de type B de l'endothéline</term>
<term>Cellules cultivées</term>
<term>Fibroblastes</term>
<term>Gencive</term>
<term>Humains</term>
<term>Mâle</term>
<term>Prolifération cellulaire</term>
<term>Rats</term>
<term>Régulation positive</term>
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<front><div type="abstract" xml:lang="en">This study aimed to evaluate the expression and bioactivities of endothelin-1 (ET-1) in gingiva during cyclosporine A (CsA) treatment.</div>
</front>
</TEI>
<pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">18973526</PMID>
<DateCompleted><Year>2009</Year>
<Month>09</Month>
<Day>15</Day>
</DateCompleted>
<DateRevised><Year>2014</Year>
<Month>11</Month>
<Day>20</Day>
</DateRevised>
<Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1600-0765</ISSN>
<JournalIssue CitedMedium="Internet"><Volume>44</Volume>
<Issue>1</Issue>
<PubDate><Year>2009</Year>
<Month>Feb</Month>
</PubDate>
</JournalIssue>
<Title>Journal of periodontal research</Title>
<ISOAbbreviation>J. Periodont. Res.</ISOAbbreviation>
</Journal>
<ArticleTitle>Expression and bioactivities of endothelin-1 in gingiva during cyclosporine A treatment.</ArticleTitle>
<Pagination><MedlinePgn>35-42</MedlinePgn>
</Pagination>
<ELocationID EIdType="doi" ValidYN="Y">10.1111/j.1600-0765.2007.01058.x</ELocationID>
<Abstract><AbstractText Label="BACKGROUND AND OBJECTIVE" NlmCategory="OBJECTIVE">This study aimed to evaluate the expression and bioactivities of endothelin-1 (ET-1) in gingiva during cyclosporine A (CsA) treatment.</AbstractText>
<AbstractText Label="MATERIAL AND METHODS" NlmCategory="METHODS">After establishing edentulous ridges, experimental rats were fed 30 mg/kg/day CsA while control animals received mineral oil for 4 weeks, after which a reverse transcription-polymerase chain reaction (RT-PCR) and/or immunohistochemistry was used to examine the expression of ET-1, its receptors, proliferating cell nuclear antigen (PCNA) and inducible nitric oxide synthase (iNOS) in gingivae. The roles of the endothelin receptors A and B (ET(A) and ET(B)) in CsA-enhanced expression of PCNA and iNOS were examined in cultured human gingival fibroblasts pretreated with receptor antagonists, by immunocytochemistry and RT-PCR, respectively.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">The mRNA expression of ET-1, ET(A) and ET(B), as well as of PCNA and iNOS, was significantly greater in edentulous gingiva that received CsA compared with control gingiva. Immunohistochemistry revealed more cells positively stained for ET-1 and its receptors in the tissues of CsA-treated rats than in those of control rats. In fibroblast cultures, enhanced mRNA expression of ET-1, ET(A) and ET(B) was observed after CsA treatment at the concentrations of 10 and 100 ng/mL. Cyclosporine A-enhanced PCNA expression was somewhat reduced by blockade of ET(A), but not ET(B), whereas iNOS expression was somewhat reduced by blockade of ET(B).</AbstractText>
<AbstractText Label="CONCLUSION" NlmCategory="CONCLUSIONS">Based on the present findings, we suggest that: (1) CsA upregulates the gingival expression of ET-1 and its receptors; and (2) ET(A) and ET(B) have different bioactivities, ET(A) being involved in cell proliferation and ET(B) being associated with iNOS expression.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Chin</LastName>
<ForeName>Y-T</ForeName>
<Initials>YT</Initials>
<AffiliationInfo><Affiliation>Graduate Institute of Life Sciences, National Defense Medical Center, Taipei, Taiwan, ROC.</Affiliation>
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<Author ValidYN="Y"><LastName>Tu</LastName>
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<Author ValidYN="Y"><LastName>Chen</LastName>
<ForeName>Y-T</ForeName>
<Initials>YT</Initials>
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<Author ValidYN="Y"><LastName>Dai</LastName>
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<Author ValidYN="Y"><LastName>Shen</LastName>
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<ForeName>M M-J</ForeName>
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<MedlineTA>J Periodontal Res</MedlineTA>
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<NameOfSubstance UI="D065130">Endothelin A Receptor Antagonists</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>0</RegistryNumber>
<NameOfSubstance UI="D065131">Endothelin B Receptor Antagonists</NameOfSubstance>
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<NameOfSubstance UI="D019332">Endothelin-1</NameOfSubstance>
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<NameOfSubstance UI="D007166">Immunosuppressive Agents</NameOfSubstance>
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</Chemical>
<Chemical><RegistryNumber>83HN0GTJ6D</RegistryNumber>
<NameOfSubstance UI="D016572">Cyclosporine</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>EC 1.14.13.39</RegistryNumber>
<NameOfSubstance UI="C496319">NOS2 protein, human</NameOfSubstance>
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<Chemical><RegistryNumber>EC 1.14.13.39</RegistryNumber>
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