The latent state‐trait structure of resting EEG asymmetry: Replication and extension
Identifieur interne : 001373 ( Istex/Corpus ); précédent : 001372; suivant : 001374The latent state‐trait structure of resting EEG asymmetry: Replication and extension
Auteurs : Dirk Hagemann ; Johannes Hewig ; Jan Seifert ; Ewald Naumann ; Dieter BartussekSource :
- Psychophysiology [ 0048-5772 ] ; 2005-11.
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Abstract
Recent research on brain asymmetry suggested that resting electroencephalographic (EEG) asymmetry represents a superimposition of a trait‐like asymmetry with state‐dependent fluctuations. The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. These data replicate previous findings and substantiate that resting asymmetry has trait and state properties.
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DOI: 10.1111/j.1469-8986.2005.00367.x
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The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. These data replicate previous findings and substantiate that resting asymmetry has trait and state properties.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>Dirk Hagemann</name><affiliations><json:string>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</json:string></affiliations></json:item><json:item><name>Johannes Hewig</name><affiliations><json:string>Department of Psychology, Friedrich‐Schiller‐Universität Jena, Jena, Germany</json:string></affiliations></json:item><json:item><name>Jan Seifert</name><affiliations><json:string>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</json:string></affiliations></json:item><json:item><name>Ewald Naumann</name><affiliations><json:string>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</json:string></affiliations></json:item><json:item><name>Dieter Bartussek</name><affiliations><json:string>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Brain asymmetry</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Electroencephalography</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Individual differences</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>State‐trait distinction</value></json:item></subject><articleId><json:string>PSYP367</json:string></articleId><language><json:string>eng</json:string></language><originalGenre><json:string>article</json:string></originalGenre><abstract>Recent research on brain asymmetry suggested that resting electroencephalographic (EEG) asymmetry represents a superimposition of a trait‐like asymmetry with state‐dependent fluctuations. The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. 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Concepts, issues, and applications</title></host><title>Structural equation models with nonnormal variables</title></json:item></refBibs><genre><json:string>article</json:string></genre><host><volume>42</volume><publisherId><json:string>PSYP</json:string></publisherId><pages><total>13</total><last>752</last><first>740</first></pages><issn><json:string>0048-5772</json:string></issn><issue>6</issue><genre><json:string>journal</json:string></genre><language><json:string>unknown</json:string></language><eissn><json:string>1469-8986</json:string></eissn><title>Psychophysiology</title><doi><json:string>10.1111/(ISSN)1469-8986</json:string></doi></host><categories><wos><json:string>social science</json:string><json:string>psychology, experimental</json:string><json:string>psychology, biological</json:string><json:string>science</json:string><json:string>psychology</json:string><json:string>physiology</json:string><json:string>neurosciences</json:string></wos><scienceMetrix><json:string>health sciences</json:string><json:string>psychology & cognitive sciences</json:string><json:string>experimental psychology</json:string></scienceMetrix></categories><publicationDate>2005</publicationDate><copyrightDate>2005</copyrightDate><doi><json:string>10.1111/j.1469-8986.2005.00367.x</json:string></doi><id>3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F</id><score>0.9052455</score><fulltext><json:item><extension>pdf</extension><original>true</original><mimetype>application/pdf</mimetype><uri>https://api.istex.fr/document/3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F/fulltext/pdf</uri></json:item><json:item><extension>zip</extension><original>false</original><mimetype>application/zip</mimetype><uri>https://api.istex.fr/document/3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">The latent state‐trait structure of resting EEG asymmetry: Replication and extension</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><availability><p>WILEY</p></availability><date>2005</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">The latent state‐trait structure of resting EEG asymmetry: Replication and extension</title><author xml:id="author-1"><persName><forename type="first">Dirk</forename><surname>Hagemann</surname></persName><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation></author><author xml:id="author-2"><persName><forename type="first">Johannes</forename><surname>Hewig</surname></persName><affiliation>Department of Psychology, Friedrich‐Schiller‐Universität Jena, Jena, Germany</affiliation></author><author xml:id="author-3"><persName><forename type="first">Jan</forename><surname>Seifert</surname></persName><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation></author><author xml:id="author-4"><persName><forename type="first">Ewald</forename><surname>Naumann</surname></persName><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation></author><author xml:id="author-5"><persName><forename type="first">Dieter</forename><surname>Bartussek</surname></persName><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation></author></analytic><monogr><title level="j">Psychophysiology</title><idno type="pISSN">0048-5772</idno><idno type="eISSN">1469-8986</idno><idno type="DOI">10.1111/(ISSN)1469-8986</idno><imprint><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><date type="published" when="2005-11"></date><biblScope unit="volume">42</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="740">740</biblScope><biblScope unit="page" to="752">752</biblScope></imprint></monogr><idno type="istex">3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F</idno><idno type="DOI">10.1111/j.1469-8986.2005.00367.x</idno><idno type="ArticleID">PSYP367</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>2005</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Recent research on brain asymmetry suggested that resting electroencephalographic (EEG) asymmetry represents a superimposition of a trait‐like asymmetry with state‐dependent fluctuations. The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. These data replicate previous findings and substantiate that resting asymmetry has trait and state properties.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>keywords</head><item><term>Brain asymmetry</term></item><item><term>Electroencephalography</term></item><item><term>Individual differences</term></item><item><term>State‐trait distinction</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2005-11">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Inc</publisherName><publisherLoc>Malden, USA</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1469-8986</doi><issn type="print">0048-5772</issn><issn type="electronic">1469-8986</issn><idGroup><id type="product" value="PSYP"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PSYCHOPHYSIOLOGY">Psychophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="11006"><doi origin="wiley">10.1111/psyp.2005.42.issue-6</doi><numberingGroup><numbering type="journalVolume" number="42">42</numbering><numbering type="journalIssue" number="6">6</numbering></numberingGroup><coverDate startDate="2005-11">November 2005</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="13" status="forIssue"><doi origin="wiley">10.1111/j.1469-8986.2005.00367.x</doi><idGroup><id type="unit" value="PSYP367"></id><id type="supplier" value="367"></id></idGroup><countGroup><count type="pageTotal" number="13"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><eventGroup><event type="firstOnline" date="2005-12-16"></event><event type="publishedOnlineFinalForm" date="2005-12-16"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:FullText result:FullText" date="2010-03-03"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-07"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="740">740</numbering><numbering type="pageLast" number="752">752</numbering></numberingGroup><correspondenceTo>Address reprint requests to: Dirk Hagemann, Fachbereich I–Psychologie, Universität Trier, Universitätsring 15, 54286 Trier, Germany. E‐mail: <email normalForm="hagemann@uni-trier.de">hagemann@uni‐trier.de</email>.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PSYP.PSYP367.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>(Received March 31, 2005; Accepted September 8, 2005)</unparsedEditorialHistory><countGroup><count type="figureTotal" number="4"></count><count type="tableTotal" number="5"></count><count type="formulaTotal" number="0"></count><count type="referenceTotal" number="48"></count><count type="wordTotal" number="11834"></count><count type="linksCrossRef" number="99"></count></countGroup><titleGroup><title type="main">The latent state‐trait structure of resting EEG asymmetry: Replication and extension</title><title type="shortAuthors">D. Hagemann et al.</title><title type="short">State‐trait structure of EEG asymmetry</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>Dirk</givenNames><familyName>Hagemann</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a2"><personName><givenNames>Johannes</givenNames><familyName>Hewig</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>Jan</givenNames><familyName>Seifert</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>Ewald</givenNames><familyName>Naumann</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>Dieter</givenNames><familyName>Bartussek</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="DE"><unparsedAffiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="DE"><unparsedAffiliation>Department of Psychology, Friedrich‐Schiller‐Universität Jena, Jena, Germany</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Brain asymmetry</keyword><keyword xml:id="k2">Electroencephalography</keyword><keyword xml:id="k3">Individual differences</keyword><keyword xml:id="k4">State‐trait distinction</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">Abstract</title><p>Recent research on brain asymmetry suggested that resting electroencephalographic (EEG) asymmetry represents a superimposition of a trait‐like asymmetry with state‐dependent fluctuations. The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. These data replicate previous findings and substantiate that resting asymmetry has trait and state properties.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn2" numbered="no"><p>The authors are grateful to Rolf Steyer for useful suggestions and also gratefully acknowledge Renate Freudenreich and Helmut Peifer for technical support, and Katja Bartz, Patrick Britz, Sven Haarscheidt, Melanie Hahn, Henning Holle, Alexander Hug, Pascal Klingmann, Astrid Kronbergs, Sonja Römer, Mirjam Rupp, and Olaf Schweisthal for data acquisition and processing. The authors are also grateful to two anonymous reviewers for valuable suggestions. This research was supported by the Deutsche Forschungsgemeinschaft through grant Ha 3044/2‐1 to the first author.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>The latent state‐trait structure of resting EEG asymmetry: Replication and extension</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>State‐trait structure of EEG asymmetry</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>The latent state‐trait structure of resting EEG asymmetry: Replication and extension</title></titleInfo><name type="personal"><namePart type="given">Dirk</namePart><namePart type="family">Hagemann</namePart><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Johannes</namePart><namePart type="family">Hewig</namePart><affiliation>Department of Psychology, Friedrich‐Schiller‐Universität Jena, Jena, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Jan</namePart><namePart type="family">Seifert</namePart><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Ewald</namePart><namePart type="family">Naumann</namePart><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Dieter</namePart><namePart type="family">Bartussek</namePart><affiliation>Fachbereich I–Psychologie, Universität Trier, Trier, Germany</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Inc</publisher><place><placeTerm type="text">Malden, USA</placeTerm></place><dateIssued encoding="w3cdtf">2005-11</dateIssued><edition>(Received March 31, 2005; Accepted September 8, 2005)</edition><copyrightDate encoding="w3cdtf">2005</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">4</extent><extent unit="tables">5</extent><extent unit="references">48</extent><extent unit="words">11834</extent></physicalDescription><abstract lang="en">Recent research on brain asymmetry suggested that resting electroencephalographic (EEG) asymmetry represents a superimposition of a trait‐like asymmetry with state‐dependent fluctuations. The present study tested this hypothesis and additionally examined individual differences in state changes. A 61‐channel EEG was collected from 59 participants in a resting state on three occasions of measurement. An analysis of latent state‐trait models suggested that between 40% and 50% of the variance of anterior asymmetry measures was due to individual differences on a latent trait and approximately the same portion of the variance was due to occasion‐specific fluctuations. A further analysis of true intraindividual change models indicated that there were large individual differences in intraindividual change over time. These data replicate previous findings and substantiate that resting asymmetry has trait and state properties.</abstract><subject lang="en"><genre>keywords</genre><topic>Brain asymmetry</topic><topic>Electroencephalography</topic><topic>Individual differences</topic><topic>State‐trait distinction</topic></subject><relatedItem type="host"><titleInfo><title>Psychophysiology</title></titleInfo><genre type="journal">journal</genre><identifier type="ISSN">0048-5772</identifier><identifier type="eISSN">1469-8986</identifier><identifier type="DOI">10.1111/(ISSN)1469-8986</identifier><identifier type="PublisherID">PSYP</identifier><part><date>2005</date><detail type="volume"><caption>vol.</caption><number>42</number></detail><detail type="issue"><caption>no.</caption><number>6</number></detail><extent unit="pages"><start>740</start><end>752</end><total>13</total></extent></part></relatedItem><identifier type="istex">3BFF7E76F44E5F8D2E53033BF1F75AEE2954207F</identifier><identifier type="DOI">10.1111/j.1469-8986.2005.00367.x</identifier><identifier type="ArticleID">PSYP367</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Inc</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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