Persistent organic pollutants and diabetes: a review of the epidemiological evidence.
Identifieur interne : 003317 ( PubMed/Checkpoint ); précédent : 003316; suivant : 003318Persistent organic pollutants and diabetes: a review of the epidemiological evidence.
Auteurs : D J Magliano [Australie] ; V H Y. Loh [Australie] ; J L Harding [Australie] ; J. Botton [France] ; J E Shaw [Australie]Source :
- Diabetes & metabolism [ 1878-1780 ] ; 2014.
Descripteurs français
- KwdFr :
- Diabète de type 2 (), Diabète de type 2 (métabolisme), Diabète de type 2 (épidémiologie), Diabète de type 2 (étiologie), Dibenzodioxines polychlorées (effets indésirables), Exposition environnementale (effets indésirables), Facteurs de risque, Femelle, Foie (métabolisme), Herbicides (effets indésirables), Humains, Hydrocarbures chlorés (effets indésirables), Insuline (sécrétion), Liaison aux protéines (), Mâle, Pancréas (métabolisme), Polluants atmosphériques d'origine professionnelle (effets indésirables), Polluants environnementaux (effets indésirables), Polychlorobiphényles (effets indésirables), Prévalence, Tissu adipeux (métabolisme), Transporteurs de glucose par diffusion facilitée (métabolisme), Épidémiosurveillance.
- MESH :
- effets indésirables : Dibenzodioxines polychlorées, Exposition environnementale, Herbicides, Hydrocarbures chlorés, Polluants atmosphériques d'origine professionnelle, Polluants environnementaux, Polychlorobiphényles.
- métabolisme : Diabète de type 2, Foie, Pancréas, Tissu adipeux, Transporteurs de glucose par diffusion facilitée.
- sécrétion : Insuline.
- épidémiologie : Diabète de type 2.
- étiologie : Diabète de type 2.
- Diabète de type 2, Facteurs de risque, Femelle, Humains, Liaison aux protéines, Mâle, Prévalence, Épidémiosurveillance.
English descriptors
- KwdEn :
- Adipose Tissue (metabolism), Air Pollutants, Occupational (adverse effects), Diabetes Mellitus, Type 2 (chemically induced), Diabetes Mellitus, Type 2 (epidemiology), Diabetes Mellitus, Type 2 (etiology), Diabetes Mellitus, Type 2 (metabolism), Environmental Exposure (adverse effects), Environmental Pollutants (adverse effects), Epidemiological Monitoring, Female, Glucose Transport Proteins, Facilitative (metabolism), Herbicides (adverse effects), Humans, Hydrocarbons, Chlorinated (adverse effects), Insulin (secretion), Liver (metabolism), Male, Pancreas (metabolism), Polychlorinated Biphenyls (adverse effects), Polychlorinated Dibenzodioxins (adverse effects), Prevalence, Protein Binding (drug effects), Risk Factors.
- MESH :
- chemical , adverse effects : Air Pollutants, Occupational, Environmental Pollutants, Herbicides, Hydrocarbons, Chlorinated, Polychlorinated Biphenyls, Polychlorinated Dibenzodioxins.
- adverse effects : Environmental Exposure.
- chemically induced : Diabetes Mellitus, Type 2.
- drug effects : Protein Binding.
- epidemiology : Diabetes Mellitus, Type 2.
- etiology : Diabetes Mellitus, Type 2.
- metabolism : Adipose Tissue, Diabetes Mellitus, Type 2, Glucose Transport Proteins, Facilitative, Liver, Pancreas.
- chemical , secretion : Insulin.
- Epidemiological Monitoring, Female, Humans, Male, Prevalence, Risk Factors.
Abstract
The prevalence of diabetes and obesity has increased rapidly over the last few decades in both developed and developing countries. While it is intuitively appealing to suggest that lifestyle risk factors such as decreased physical activity and adoption of poor diets can explain much of the increase, the evidence to support this is poor. Given this, there has been an impetus to look more widely than traditional lifestyle and biomedical risk factors, especially those risk factors, which arise from the environment. Since the industrial revolution, there has been an introduction of many chemicals into our environment, which have now become environmental pollutants. There has been growing interest in one key class of environmental pollutants known as persistent organic pollutants (POPs) and their potential role in the development of diabetes. This review will summarise and appraise the current epidemiological evidence relating POPs to diabetes and highlight gaps and flaws in this evidence.
DOI: 10.1016/j.diabet.2013.09.006
PubMed: 24262435
Affiliations:
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environnementale (effets indésirables)</term><term>Facteurs de risque</term><term>Femelle</term><term>Foie (métabolisme)</term><term>Herbicides (effets indésirables)</term><term>Humains</term><term>Hydrocarbures chlorés (effets indésirables)</term><term>Insuline (sécrétion)</term><term>Liaison aux protéines ()</term><term>Mâle</term><term>Pancréas (métabolisme)</term><term>Polluants atmosphériques d'origine professionnelle (effets indésirables)</term><term>Polluants environnementaux (effets indésirables)</term><term>Polychlorobiphényles (effets indésirables)</term><term>Prévalence</term><term>Tissu adipeux (métabolisme)</term><term>Transporteurs de glucose par diffusion facilitée (métabolisme)</term><term>Épidémiosurveillance</term></keywords><keywords scheme="MESH" type="chemical" qualifier="adverse effects" xml:lang="en"><term>Air Pollutants, Occupational</term><term>Environmental Pollutants</term><term>Herbicides</term><term>Hydrocarbons, Chlorinated</term><term>Polychlorinated Biphenyls</term><term>Polychlorinated Dibenzodioxins</term></keywords><keywords scheme="MESH" qualifier="adverse effects" xml:lang="en"><term>Environmental Exposure</term></keywords><keywords scheme="MESH" qualifier="chemically induced" xml:lang="en"><term>Diabetes Mellitus, Type 2</term></keywords><keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Protein Binding</term></keywords><keywords scheme="MESH" qualifier="effets indésirables" xml:lang="fr"><term>Dibenzodioxines polychlorées</term><term>Exposition environnementale</term><term>Herbicides</term><term>Hydrocarbures chlorés</term><term>Polluants atmosphériques d'origine professionnelle</term><term>Polluants environnementaux</term><term>Polychlorobiphényles</term></keywords><keywords scheme="MESH" qualifier="epidemiology" xml:lang="en"><term>Diabetes Mellitus, Type 2</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Diabetes Mellitus, Type 2</term></keywords><keywords scheme="MESH" 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Monitoring</term><term>Female</term><term>Humans</term><term>Male</term><term>Prevalence</term><term>Risk Factors</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Diabète de type 2</term><term>Facteurs de risque</term><term>Femelle</term><term>Humains</term><term>Liaison aux protéines</term><term>Mâle</term><term>Prévalence</term><term>Épidémiosurveillance</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">The prevalence of diabetes and obesity has increased rapidly over the last few decades in both developed and developing countries. While it is intuitively appealing to suggest that lifestyle risk factors such as decreased physical activity and adoption of poor diets can explain much of the increase, the evidence to support this is poor. Given this, there has been an impetus to look more widely than traditional lifestyle and biomedical risk factors, especially those risk factors, which arise from the environment. Since the industrial revolution, there has been an introduction of many chemicals into our environment, which have now become environmental pollutants. There has been growing interest in one key class of environmental pollutants known as persistent organic pollutants (POPs) and their potential role in the development of diabetes. This review will summarise and appraise the current epidemiological evidence relating POPs to diabetes and highlight gaps and flaws in this evidence.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">24262435</PMID><DateCreated><Year>2014</Year><Month>02</Month><Day>10</Day></DateCreated><DateCompleted><Year>2014</Year><Month>11</Month><Day>12</Day></DateCompleted><DateRevised><Year>2016</Year><Month>11</Month><Day>25</Day></DateRevised><Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1878-1780</ISSN><JournalIssue CitedMedium="Internet"><Volume>40</Volume><Issue>1</Issue><PubDate><Year>2014</Year><Month>Feb</Month></PubDate></JournalIssue><Title>Diabetes & metabolism</Title><ISOAbbreviation>Diabetes Metab.</ISOAbbreviation></Journal><ArticleTitle>Persistent organic pollutants and diabetes: a review of the epidemiological evidence.</ArticleTitle><Pagination><MedlinePgn>1-14</MedlinePgn></Pagination><ELocationID EIdType="doi" ValidYN="Y">10.1016/j.diabet.2013.09.006</ELocationID><ELocationID EIdType="pii" ValidYN="Y">S1262-3636(13)00179-1</ELocationID><Abstract><AbstractText>The prevalence of diabetes and obesity has increased rapidly over the last few decades in both developed and developing countries. While it is intuitively appealing to suggest that lifestyle risk factors such as decreased physical activity and adoption of poor diets can explain much of the increase, the evidence to support this is poor. Given this, there has been an impetus to look more widely than traditional lifestyle and biomedical risk factors, especially those risk factors, which arise from the environment. Since the industrial revolution, there has been an introduction of many chemicals into our environment, which have now become environmental pollutants. There has been growing interest in one key class of environmental pollutants known as persistent organic pollutants (POPs) and their potential role in the development of diabetes. This review will summarise and appraise the current epidemiological evidence relating POPs to diabetes and highlight gaps and flaws in this evidence.</AbstractText><CopyrightInformation>Copyright © 2013 Elsevier Masson SAS. All rights reserved.</CopyrightInformation></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Magliano</LastName><ForeName>D J</ForeName><Initials>DJ</Initials><AffiliationInfo><Affiliation>Baker IDI Heart and Diabetes Institute, Melbourne, Australia; Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australia. Electronic address: dianna.magliano@bakeridi.edu.au.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Loh</LastName><ForeName>V H Y</ForeName><Initials>VH</Initials><AffiliationInfo><Affiliation>Baker IDI Heart and Diabetes Institute, Melbourne, Australia.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Harding</LastName><ForeName>J L</ForeName><Initials>JL</Initials><AffiliationInfo><Affiliation>Baker IDI Heart and Diabetes Institute, Melbourne, Australia; Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australia.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Botton</LastName><ForeName>J</ForeName><Initials>J</Initials><AffiliationInfo><Affiliation>Inserm, CESP (Centre of Research for Epidemiology and Population Health), U1018, Lifelong Epidemiology of Obesity, Diabetes and Kidney Diseases Team, 94807 Villejuif, France; UMR-S 1018, University Paris-Sud, 94807 Villejuif, France.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Shaw</LastName><ForeName>J E</ForeName><Initials>JE</Initials><AffiliationInfo><Affiliation>Baker IDI Heart and Diabetes Institute, Melbourne, Australia; Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australia.</Affiliation></AffiliationInfo></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList><ArticleDate DateType="Electronic"><Year>2013</Year><Month>11</Month><Day>18</Day></ArticleDate></Article><MedlineJournalInfo><Country>France</Country><MedlineTA>Diabetes Metab</MedlineTA><NlmUniqueID>9607599</NlmUniqueID><ISSNLinking>1262-3636</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D000395">Air Pollutants, Occupational</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D004785">Environmental Pollutants</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D051246">Glucose Transport Proteins, Facilitative</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D006540">Herbicides</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D006843">Hydrocarbons, Chlorinated</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D007328">Insulin</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D000072317">Polychlorinated Dibenzodioxins</NameOfSubstance></Chemical><Chemical><RegistryNumber>DFC2HB4I0K</RegistryNumber><NameOfSubstance UI="D011078">Polychlorinated Biphenyls</NameOfSubstance></Chemical></ChemicalList><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000273" MajorTopicYN="N">Adipose Tissue</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="Y">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D000395" MajorTopicYN="N">Air Pollutants, Occupational</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="Y">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D003924" MajorTopicYN="N">Diabetes Mellitus, Type 2</DescriptorName><QualifierName UI="Q000139" MajorTopicYN="N">chemically induced</QualifierName><QualifierName UI="Q000453" MajorTopicYN="N">epidemiology</QualifierName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004781" MajorTopicYN="N">Environmental Exposure</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="Y">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004785" MajorTopicYN="N">Environmental Pollutants</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="Y">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D062665" MajorTopicYN="N">Epidemiological Monitoring</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D051246" MajorTopicYN="N">Glucose Transport Proteins, Facilitative</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006540" MajorTopicYN="N">Herbicides</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="N">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006843" MajorTopicYN="N">Hydrocarbons, Chlorinated</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="N">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D007328" MajorTopicYN="N">Insulin</DescriptorName><QualifierName UI="Q000557" MajorTopicYN="N">secretion</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008099" MajorTopicYN="N">Liver</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="Y">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D010179" MajorTopicYN="N">Pancreas</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="Y">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011078" MajorTopicYN="N">Polychlorinated Biphenyls</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="N">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D000072317" MajorTopicYN="N">Polychlorinated Dibenzodioxins</DescriptorName><QualifierName UI="Q000009" MajorTopicYN="N">adverse effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D015995" MajorTopicYN="N">Prevalence</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D011485" MajorTopicYN="N">Protein Binding</DescriptorName><QualifierName UI="Q000187" MajorTopicYN="N">drug effects</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D012307" MajorTopicYN="N">Risk Factors</DescriptorName></MeshHeading></MeshHeadingList><KeywordList Owner="NOTNLM"><Keyword MajorTopicYN="N">Diabetes</Keyword><Keyword MajorTopicYN="N">Dioxin</Keyword><Keyword MajorTopicYN="N">Persistent organic pollutants</Keyword><Keyword MajorTopicYN="N">Polychlorinated biphenyls</Keyword></KeywordList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="received"><Year>2013</Year><Month>08</Month><Day>19</Day></PubMedPubDate><PubMedPubDate PubStatus="revised"><Year>2013</Year><Month>09</Month><Day>26</Day></PubMedPubDate><PubMedPubDate PubStatus="accepted"><Year>2013</Year><Month>09</Month><Day>26</Day></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2013</Year><Month>11</Month><Day>23</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="pubmed"><Year>2013</Year><Month>11</Month><Day>23</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2014</Year><Month>11</Month><Day>13</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">24262435</ArticleId><ArticleId IdType="pii">S1262-3636(13)00179-1</ArticleId><ArticleId IdType="doi">10.1016/j.diabet.2013.09.006</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Australie</li><li>France</li></country><region><li>Victoria (État)</li><li>Île-de-France</li></region><settlement><li>Melbourne</li><li>Villejuif</li></settlement></list><tree><country name="Australie"><region name="Victoria (État)"><name sortKey="Magliano, D J" sort="Magliano, D J" uniqKey="Magliano D" first="D J" last="Magliano">D J Magliano</name></region><name sortKey="Harding, J L" sort="Harding, J L" uniqKey="Harding J" first="J L" last="Harding">J L Harding</name><name sortKey="Loh, V H Y" sort="Loh, V H Y" uniqKey="Loh V" first="V H Y" last="Loh">V H Y. Loh</name><name sortKey="Shaw, J E" sort="Shaw, J E" uniqKey="Shaw J" first="J E" last="Shaw">J E Shaw</name></country><country name="France"><region name="Île-de-France"><name sortKey="Botton, J" sort="Botton, J" uniqKey="Botton J" first="J" last="Botton">J. Botton</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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