PINK1 deficiency enhances autophagy and mitophagy induction
Identifieur interne : 000611 ( Main/Exploration ); précédent : 000610; suivant : 000612PINK1 deficiency enhances autophagy and mitophagy induction
Auteurs : Rubén G Mez-Sánchez [Espagne] ; Sokhna M S. Yakhine-Diop [Espagne] ; José M. Bravo-San Pedro [Espagne, France] ; Elisa Pizarro-Estrella [Espagne] ; Mario Rodríguez-Arribas [Espagne] ; Vicente Climent [Espagne] ; Francisco E. Martin-Cano [Espagne] ; María E. González-Soltero [Espagne] ; Anurag Tandon [Canada] ; José M. Fuentes [Espagne] ; Rosa A. González-Polo [Espagne]Source :
- Molecular & Cellular Oncology [ 2372-3548 ] ; 2015.
Abstract
Parkinson's disease (PD) is a neurodegenerative disorder with poorly understood etiology. Increasing evidence suggests that age-dependent compromise of the maintenance of mitochondrial function is a key risk factor. Several proteins encoded by PD-related genes are associated with mitochondria including
Url:
DOI: 10.1080/23723556.2015.1046579
PubMed: 27308585
PubMed Central: 4905358
Affiliations:
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Le document en format XML
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<author><name sortKey="Yakhine Diop, Sokhna M S" sort="Yakhine Diop, Sokhna M S" uniqKey="Yakhine Diop S" first="Sokhna M S" last="Yakhine-Diop">Sokhna M S. Yakhine-Diop</name>
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<author><name sortKey="Gonzalez Soltero, Maria E" sort="Gonzalez Soltero, Maria E" uniqKey="Gonzalez Soltero M" first="María E" last="González-Soltero">María E. González-Soltero</name>
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<author><name sortKey="Pizarro Estrella, Elisa" sort="Pizarro Estrella, Elisa" uniqKey="Pizarro Estrella E" first="Elisa" last="Pizarro-Estrella">Elisa Pizarro-Estrella</name>
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</nlm:aff>
<country xml:lang="fr">Espagne</country>
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</nlm:aff>
<country xml:lang="fr">Espagne</country>
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; Cáceres,<country>Spain</country>
</nlm:aff>
<country xml:lang="fr">Espagne</country>
<wicri:regionArea># see nlm:aff country strict</wicri:regionArea>
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<author><name sortKey="Gonzalez Soltero, Maria E" sort="Gonzalez Soltero, Maria E" uniqKey="Gonzalez Soltero M" first="María E" last="González-Soltero">María E. González-Soltero</name>
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; Plasencia,<country>Spain</country>
</nlm:aff>
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<author><name sortKey="Tandon, Anurag" sort="Tandon, Anurag" uniqKey="Tandon A" first="Anurag" last="Tandon">Anurag Tandon</name>
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<wicri:regionArea># see nlm:aff country strict</wicri:regionArea>
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</nlm:aff>
<country xml:lang="fr">Espagne</country>
<wicri:regionArea># see nlm:aff country strict</wicri:regionArea>
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<affiliation><nlm:aff id="af0010">These authors contributed equally to this work.</nlm:aff>
</affiliation>
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<author><name sortKey="Gonzalez Polo, Rosa A" sort="Gonzalez Polo, Rosa A" uniqKey="Gonzalez Polo R" first="Rosa A" last="González-Polo">Rosa A. González-Polo</name>
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</nlm:aff>
<country xml:lang="fr">Espagne</country>
<wicri:regionArea># see nlm:aff country strict</wicri:regionArea>
</affiliation>
<affiliation><nlm:aff id="af0010">These authors contributed equally to this work.</nlm:aff>
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<series><title level="j">Molecular & Cellular Oncology</title>
<idno type="ISSN">2372-3548</idno>
<idno type="eISSN">2372-3556</idno>
<imprint><date when="2015">2015</date>
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<front><div type="abstract" xml:lang="en"><p>Parkinson's disease (PD) is a neurodegenerative disorder with poorly understood etiology. Increasing evidence suggests that age-dependent compromise of the maintenance of mitochondrial function is a key risk factor. Several proteins encoded by PD-related genes are associated with mitochondria including <italic>PTEN-induced putative kinase 1</italic>
(<italic>PINK1</italic>
), which was first identified as a gene that is upregulated by PTEN. Loss-of-function <italic>PINK1</italic>
mutations induce mitochondrial dysfunction and, ultimately, neuronal cell death. To mitigate the negative effects of altered cellular functions cells possess a degradation mechanism called autophagy for recycling damaged components; selective elimination of dysfunctional mitochondria by autophagy is termed mitophagy. Our study indicates that autophagy and mitophagy are upregulated in <italic>PINK1</italic>
-deficient cells, and is the first report to demonstrate efficient fluxes by one-step analysis. We propose that autophagy is induced to maintain cellular homeostasis under conditions of non-regulated mitochondrial quality control.</p>
</div>
</front>
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<name sortKey="Gonzalez Soltero, Maria E" sort="Gonzalez Soltero, Maria E" uniqKey="Gonzalez Soltero M" first="María E" last="González-Soltero">María E. González-Soltero</name>
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<name sortKey="Yakhine Diop, Sokhna M S" sort="Yakhine Diop, Sokhna M S" uniqKey="Yakhine Diop S" first="Sokhna M S" last="Yakhine-Diop">Sokhna M S. Yakhine-Diop</name>
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<country name="France"><noRegion><name sortKey="Bravo San Pedro, Jose M" sort="Bravo San Pedro, Jose M" uniqKey="Bravo San Pedro J" first="José M" last="Bravo-San Pedro">José M. Bravo-San Pedro</name>
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<name sortKey="Bravo San Pedro, Jose M" sort="Bravo San Pedro, Jose M" uniqKey="Bravo San Pedro J" first="José M" last="Bravo-San Pedro">José M. Bravo-San Pedro</name>
<name sortKey="Bravo San Pedro, Jose M" sort="Bravo San Pedro, Jose M" uniqKey="Bravo San Pedro J" first="José M" last="Bravo-San Pedro">José M. Bravo-San Pedro</name>
<name sortKey="Bravo San Pedro, Jose M" sort="Bravo San Pedro, Jose M" uniqKey="Bravo San Pedro J" first="José M" last="Bravo-San Pedro">José M. Bravo-San Pedro</name>
</country>
<country name="Canada"><noRegion><name sortKey="Tandon, Anurag" sort="Tandon, Anurag" uniqKey="Tandon A" first="Anurag" last="Tandon">Anurag Tandon</name>
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</tree>
</affiliations>
</record>
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