Crossing barriers: infections of the lung and the gut
Identifieur interne : 000E34 ( Pmc/Checkpoint ); précédent : 000E33; suivant : 000E35Crossing barriers: infections of the lung and the gut
Auteurs : P J OpenshawSource :
- Mucosal Immunology [ 1933-0219 ] ; 2008.
Abstract
Although known as respiratory pathogens, severe acute respiratory syndrome (SARS) and its sister coronaviruses frequently cause enteric symptoms. In addition, other classically non-enteric viruses (such as HIV and influenza) may also have enteric effects that are crucial in their pathogeneses. These effects can be due to direct infection of the gut mucosa, but can also be because of decreased antibacterial defenses, increased mucosal permeability, bacterial translocation, and systemic leak of endotoxin.
The online version of this article (doi:10.1038/mi.2008.79) contains supplementary material, which is available to authorized users.
Url:
DOI: 10.1038/mi.2008.79
PubMed: 19129753
PubMed Central: 7100545
Affiliations:
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PMC:7100545Le document en format XML
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<contrib-group><contrib contrib-type="author" corresp="yes"><name><surname>Openshaw</surname>
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<pub-date pub-type="epub"><day>24</day>
<month>12</month>
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<volume>2</volume>
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<fpage>100</fpage>
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<permissions><copyright-statement>© Society for Mucosal Immunology Ltd 2009</copyright-statement>
<license><license-p>This article is made available via the PMC Open Access Subset for unrestricted research re-use and secondary analysis in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the World Health Organization (WHO) declaration of COVID-19 as a global pandemic.</license-p>
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<abstract id="Abs1"><p id="Par1">Although known as respiratory pathogens, severe acute respiratory syndrome (SARS) and its sister coronaviruses frequently cause enteric symptoms. In addition, other classically non-enteric viruses (such as HIV and influenza) may also have enteric effects that are crucial in their pathogeneses. These effects can be due to direct infection of the gut mucosa, but can also be because of decreased antibacterial defenses, increased mucosal permeability, bacterial translocation, and systemic leak of endotoxin.</p>
<sec><title>Supplementary information</title>
<p>The online version of this article (doi:10.1038/mi.2008.79) contains supplementary material, which is available to authorized users.</p>
</sec>
</abstract>
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