Enhanced cell fusion activity in porcine epidemic diarrhea virus adapted to suckling mice.
Identifieur interne : 002208 ( Ncbi/Merge ); précédent : 002207; suivant : 002209Enhanced cell fusion activity in porcine epidemic diarrhea virus adapted to suckling mice.
Auteurs : Kazuya Shirato [Japon] ; Madoka Maejima ; Asuka Hirai ; Yasushi Ami ; Natsumi Takeyama ; Kotaro Tsuchiya ; Kouich Kusanagi ; Tetsuo Nunoya ; Fumihiro TaguchiSource :
- Archives of virology [ 1432-8798 ] ; 2010.
Descripteurs français
- KwdFr :
- Adaptation biologique, Animaux, Cellules cultivées, Encéphale (virologie), Femelle, Fusion cellulaire, Glycoprotéine de spicule des coronavirus, Glycoprotéines membranaires (génétique), Grossesse, Modèles animaux de maladie humaine, Passage en série, Protéines de l'enveloppe virale (génétique), Souris, Souris de lignée ICR, Substitution d'acide aminé (génétique), Virulence, Virus de la diarrhée porcine épidémique (génétique), Virus de la diarrhée porcine épidémique (pathogénicité).
- MESH :
- génétique : Glycoprotéines membranaires, Protéines de l'enveloppe virale, Substitution d'acide aminé, Virus de la diarrhée porcine épidémique.
- pathogénicité : Virus de la diarrhée porcine épidémique.
- virologie : Encéphale.
- Adaptation biologique, Animaux, Cellules cultivées, Femelle, Fusion cellulaire, Glycoprotéine de spicule des coronavirus, Grossesse, Modèles animaux de maladie humaine, Passage en série, Souris, Souris de lignée ICR, Virulence.
English descriptors
- KwdEn :
- Adaptation, Biological, Amino Acid Substitution (genetics), Animals, Brain (virology), Cell Fusion, Cells, Cultured, Disease Models, Animal, Female, Membrane Glycoproteins (genetics), Mice, Mice, Inbred ICR, Porcine epidemic diarrhea virus (genetics), Porcine epidemic diarrhea virus (pathogenicity), Pregnancy, Serial Passage, Spike Glycoprotein, Coronavirus, Viral Envelope Proteins (genetics), Virulence.
- MESH :
- chemical , genetics : Membrane Glycoproteins, Viral Envelope Proteins.
- genetics : Amino Acid Substitution, Porcine epidemic diarrhea virus.
- pathogenicity : Porcine epidemic diarrhea virus.
- virology : Brain.
- Adaptation, Biological, Animals, Cell Fusion, Cells, Cultured, Disease Models, Animal, Female, Mice, Mice, Inbred ICR, Pregnancy, Serial Passage, Spike Glycoprotein, Coronavirus, Virulence.
Abstract
Porcine epidemic diarrhea virus (PEDV) is the major causative agent of fatal diarrhea in piglets. To study the pathogenic features of PEDV using a mouse model, PEDV with virulence in mice is required. In pursuit of this, we adapted a tissue-culture-passed PEDV MK strain to suckling mouse brains. PEDV obtained after ten passages through the brains (MK-p10) had increased virulence for mice, and its fusion activity in cultured cells exceeded that of the original strain. However, the replication kinetics of MK and MK-p10 did not differ from each other in the brain and in cultured cells. The spike (S) protein of MK-p10 had four amino acid substitutions relative to the original strain. One of these (an H-to-R substitution at residue 1,381) was first detected in PEDV isolated after eight passages, and both this virus (MK-p8) and MK-p10 showed enhanced syncytium formation relative to the original MK strain and viruses isolated after two, four, and six passages, suggesting the possibility that the H-to-R mutation was responsible for this activity. This mutation could be also involved in the increased virulence of PEDV observed for MK-p10.
DOI: 10.1007/s00705-010-0790-1
PubMed: 20827493
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pubmed:20827493Le document en format XML
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<term>Cell Fusion</term>
<term>Cells, Cultured</term>
<term>Disease Models, Animal</term>
<term>Female</term>
<term>Membrane Glycoproteins (genetics)</term>
<term>Mice</term>
<term>Mice, Inbred ICR</term>
<term>Porcine epidemic diarrhea virus (genetics)</term>
<term>Porcine epidemic diarrhea virus (pathogenicity)</term>
<term>Pregnancy</term>
<term>Serial Passage</term>
<term>Spike Glycoprotein, Coronavirus</term>
<term>Viral Envelope Proteins (genetics)</term>
<term>Virulence</term>
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<term>Cellules cultivées</term>
<term>Encéphale (virologie)</term>
<term>Femelle</term>
<term>Fusion cellulaire</term>
<term>Glycoprotéine de spicule des coronavirus</term>
<term>Glycoprotéines membranaires (génétique)</term>
<term>Grossesse</term>
<term>Modèles animaux de maladie humaine</term>
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<term>Protéines de l'enveloppe virale (génétique)</term>
<term>Souris</term>
<term>Souris de lignée ICR</term>
<term>Substitution d'acide aminé (génétique)</term>
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<term>Virus de la diarrhée porcine épidémique (génétique)</term>
<term>Virus de la diarrhée porcine épidémique (pathogénicité)</term>
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<term>Viral Envelope Proteins</term>
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<keywords scheme="MESH" qualifier="genetics" xml:lang="en"><term>Amino Acid Substitution</term>
<term>Porcine epidemic diarrhea virus</term>
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<keywords scheme="MESH" qualifier="génétique" xml:lang="fr"><term>Glycoprotéines membranaires</term>
<term>Protéines de l'enveloppe virale</term>
<term>Substitution d'acide aminé</term>
<term>Virus de la diarrhée porcine épidémique</term>
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<term>Cell Fusion</term>
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<term>Disease Models, Animal</term>
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<term>Mice</term>
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<term>Pregnancy</term>
<term>Serial Passage</term>
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<term>Virulence</term>
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<front><div type="abstract" xml:lang="en">Porcine epidemic diarrhea virus (PEDV) is the major causative agent of fatal diarrhea in piglets. To study the pathogenic features of PEDV using a mouse model, PEDV with virulence in mice is required. In pursuit of this, we adapted a tissue-culture-passed PEDV MK strain to suckling mouse brains. PEDV obtained after ten passages through the brains (MK-p10) had increased virulence for mice, and its fusion activity in cultured cells exceeded that of the original strain. However, the replication kinetics of MK and MK-p10 did not differ from each other in the brain and in cultured cells. The spike (S) protein of MK-p10 had four amino acid substitutions relative to the original strain. One of these (an H-to-R substitution at residue 1,381) was first detected in PEDV isolated after eight passages, and both this virus (MK-p8) and MK-p10 showed enhanced syncytium formation relative to the original MK strain and viruses isolated after two, four, and six passages, suggesting the possibility that the H-to-R mutation was responsible for this activity. This mutation could be also involved in the increased virulence of PEDV observed for MK-p10.</div>
</front>
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<Abstract><AbstractText>Porcine epidemic diarrhea virus (PEDV) is the major causative agent of fatal diarrhea in piglets. To study the pathogenic features of PEDV using a mouse model, PEDV with virulence in mice is required. In pursuit of this, we adapted a tissue-culture-passed PEDV MK strain to suckling mouse brains. PEDV obtained after ten passages through the brains (MK-p10) had increased virulence for mice, and its fusion activity in cultured cells exceeded that of the original strain. However, the replication kinetics of MK and MK-p10 did not differ from each other in the brain and in cultured cells. The spike (S) protein of MK-p10 had four amino acid substitutions relative to the original strain. One of these (an H-to-R substitution at residue 1,381) was first detected in PEDV isolated after eight passages, and both this virus (MK-p8) and MK-p10 showed enhanced syncytium formation relative to the original MK strain and viruses isolated after two, four, and six passages, suggesting the possibility that the H-to-R mutation was responsible for this activity. This mutation could be also involved in the increased virulence of PEDV observed for MK-p10.</AbstractText>
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<affiliations><list><country><li>Japon</li>
</country>
<region><li>Région de Kantō</li>
</region>
<settlement><li>Tokyo</li>
</settlement>
</list>
<tree><noCountry><name sortKey="Ami, Yasushi" sort="Ami, Yasushi" uniqKey="Ami Y" first="Yasushi" last="Ami">Yasushi Ami</name>
<name sortKey="Hirai, Asuka" sort="Hirai, Asuka" uniqKey="Hirai A" first="Asuka" last="Hirai">Asuka Hirai</name>
<name sortKey="Kusanagi, Kouich" sort="Kusanagi, Kouich" uniqKey="Kusanagi K" first="Kouich" last="Kusanagi">Kouich Kusanagi</name>
<name sortKey="Maejima, Madoka" sort="Maejima, Madoka" uniqKey="Maejima M" first="Madoka" last="Maejima">Madoka Maejima</name>
<name sortKey="Nunoya, Tetsuo" sort="Nunoya, Tetsuo" uniqKey="Nunoya T" first="Tetsuo" last="Nunoya">Tetsuo Nunoya</name>
<name sortKey="Taguchi, Fumihiro" sort="Taguchi, Fumihiro" uniqKey="Taguchi F" first="Fumihiro" last="Taguchi">Fumihiro Taguchi</name>
<name sortKey="Takeyama, Natsumi" sort="Takeyama, Natsumi" uniqKey="Takeyama N" first="Natsumi" last="Takeyama">Natsumi Takeyama</name>
<name sortKey="Tsuchiya, Kotaro" sort="Tsuchiya, Kotaro" uniqKey="Tsuchiya K" first="Kotaro" last="Tsuchiya">Kotaro Tsuchiya</name>
</noCountry>
<country name="Japon"><region name="Région de Kantō"><name sortKey="Shirato, Kazuya" sort="Shirato, Kazuya" uniqKey="Shirato K" first="Kazuya" last="Shirato">Kazuya Shirato</name>
</region>
</country>
</tree>
</affiliations>
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