SrasV1, Ncbi, Merge, bibRecord, 000F17

Association of RANTES with the replication of severe acute respiratory syndrome coronavirus in THP-1 cells.

Identifieur interne : 000F17 ( Ncbi/Merge ); précédent : 000F16; suivant : 000F18

Association of RANTES with the replication of severe acute respiratory syndrome coronavirus in THP-1 cells.

Auteurs : D. Li [Allemagne] ; N. Wu ; H. Yao ; A. Bader ; Norbert H. Brockmeyer ; P. Altmeyer

Source :

European journal of medical research [ 0949-2321 ] ; 2005.

RBID : pubmed:15851378

Descripteurs français

KwdFr :
- Animaux, Antiviraux (physiologie), Cellules CHO, Cellules Vero, Chimiokine CCL5 (physiologie), Cricetinae, Humains, Lignée cellulaire tumorale, Macrophages (virologie), Réplication virale (physiologie), Virus du SRAS (physiologie).
MESH :
- physiologie : Antiviraux, Chimiokine CCL5, Réplication virale, Virus du SRAS.
- virologie : Macrophages.
- Animaux, Cellules CHO, Cellules Vero, Cricetinae, Humains, Lignée cellulaire tumorale.

English descriptors

KwdEn :
- Animals, Antiviral Agents (physiology), CHO Cells, Cell Line, Tumor, Chemokine CCL5 (physiology), Chlorocebus aethiops, Cricetinae, Humans, Macrophages (virology), SARS Virus (physiology), Vero Cells, Virus Replication (physiology).
MESH :
- chemical , physiology : Antiviral Agents, Chemokine CCL5.
- physiology : SARS Virus, Virus Replication.
- virology : Macrophages.
- Animals, CHO Cells, Cell Line, Tumor, Chlorocebus aethiops, Cricetinae, Humans, Vero Cells.

Abstract

Severe acute respiratory syndrome (SARS) is a novel infectious disease which is characterized by an overaggressive immune response. Chemokines are important inflammatory mediators and regulate disease due to viral infection. In previous study, we found that SARS-CoV has the ability to replicate in mononuclear cells. In present work, we sought to characterize the replication of SARS-CoV at the presence of RANTES in THP-1 cells.

PubMed: 15851378

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to stream PubMed, to step Checkpoint: 002815

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pubmed:15851378

Le document en format XML

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<author><name sortKey="Li, D" sort="Li, D" uniqKey="Li D" first="D" last="Li">D. Li</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Dermatology and Allergology, Ruhr-University Bochum, St. Josef-Hospital, Gudrunstr. 56, D-44791 Bochum, Germany.</nlm:affiliation>
<country xml:lang="fr">Allemagne</country>
<wicri:regionArea>Department of Dermatology and Allergology, Ruhr-University Bochum, St. Josef-Hospital, Gudrunstr. 56, D-44791 Bochum</wicri:regionArea>
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<author><name sortKey="Bader, A" sort="Bader, A" uniqKey="Bader A" first="A" last="Bader">A. Bader</name>
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<author><name sortKey="Brockmeyer, Norbert H" sort="Brockmeyer, Norbert H" uniqKey="Brockmeyer N" first="Norbert H" last="Brockmeyer">Norbert H. Brockmeyer</name>
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<term>CHO Cells</term>
<term>Cell Line, Tumor</term>
<term>Chemokine CCL5 (physiology)</term>
<term>Chlorocebus aethiops</term>
<term>Cricetinae</term>
<term>Humans</term>
<term>Macrophages (virology)</term>
<term>SARS Virus (physiology)</term>
<term>Vero Cells</term>
<term>Virus Replication (physiology)</term>
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<keywords scheme="KwdFr" xml:lang="fr"><term>Animaux</term>
<term>Antiviraux (physiologie)</term>
<term>Cellules CHO</term>
<term>Cellules Vero</term>
<term>Chimiokine CCL5 (physiologie)</term>
<term>Cricetinae</term>
<term>Humains</term>
<term>Lignée cellulaire tumorale</term>
<term>Macrophages (virologie)</term>
<term>Réplication virale (physiologie)</term>
<term>Virus du SRAS (physiologie)</term>
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<keywords scheme="MESH" type="chemical" qualifier="physiology" xml:lang="en"><term>Antiviral Agents</term>
<term>Chemokine CCL5</term>
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<term>Chimiokine CCL5</term>
<term>Réplication virale</term>
<term>Virus du SRAS</term>
</keywords>
<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>SARS Virus</term>
<term>Virus Replication</term>
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<keywords scheme="MESH" qualifier="virologie" xml:lang="fr"><term>Macrophages</term>
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<term>Humains</term>
<term>Lignée cellulaire tumorale</term>
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<front><div type="abstract" xml:lang="en">Severe acute respiratory syndrome (SARS) is a novel infectious disease which is characterized by an overaggressive immune response. Chemokines are important inflammatory mediators and regulate disease due to viral infection. In previous study, we found that SARS-CoV has the ability to replicate in mononuclear cells. In present work, we sought to characterize the replication of SARS-CoV at the presence of RANTES in THP-1 cells.</div>
</front>
</TEI>
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<DateCompleted><Year>2005</Year>
<Month>09</Month>
<Day>27</Day>
</DateCompleted>
<DateRevised><Year>2019</Year>
<Month>12</Month>
<Day>10</Day>
</DateRevised>
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<JournalIssue CitedMedium="Print"><Volume>10</Volume>
<Issue>3</Issue>
<PubDate><Year>2005</Year>
<Month>Mar</Month>
<Day>29</Day>
</PubDate>
</JournalIssue>
<Title>European journal of medical research</Title>
<ISOAbbreviation>Eur. J. Med. Res.</ISOAbbreviation>
</Journal>
<ArticleTitle>Association of RANTES with the replication of severe acute respiratory syndrome coronavirus in THP-1 cells.</ArticleTitle>
<Pagination><MedlinePgn>117-20</MedlinePgn>
</Pagination>
<Abstract><AbstractText Label="BACKGROUND" NlmCategory="BACKGROUND">Severe acute respiratory syndrome (SARS) is a novel infectious disease which is characterized by an overaggressive immune response. Chemokines are important inflammatory mediators and regulate disease due to viral infection. In previous study, we found that SARS-CoV has the ability to replicate in mononuclear cells. In present work, we sought to characterize the replication of SARS-CoV at the presence of RANTES in THP-1 cells.</AbstractText>
<AbstractText Label="METHODS" NlmCategory="METHODS">To determine whether RANTES play an role in the process of SARS, THP-1 cells were incubated with heat-inactivated SARS-CoV and ELISA was used to test RANTES levels in the supernatants; Then the effect of dexamethasone on the induced secretion was evaluated. Real-time PCR was used to investigate the effort of RANTES on the replication of SARS-CoV in vitro. Macrophages, induced by THP-1 cells, were used as cell model.</AbstractText>
<AbstractText Label="FINDINGS" NlmCategory="RESULTS">Inactive SARS-CoV could induce THP-1 cells secret RANTES and this increase effect could not be suppressed by DXM. RANTES itself could inhibit the replication of SARS-CoV in THP-1 cells when it was added into the culture before or at the same time with the virus; No inhibition effect was shown when RANTES were added into the culture after SARS-CoV infected the cells.</AbstractText>
</Abstract>
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Association of RANTES with the replication of severe acute respiratory syndrome coronavirus in THP-1 cells.

Association of RANTES with the replication of severe acute respiratory syndrome coronavirus in THP-1 cells.

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki