A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.
Identifieur interne : 002434 ( Ncbi/Curation ); précédent : 002433; suivant : 002435A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.
Auteurs : Stewart T. Chang [États-Unis] ; Nicolas Tchitchek ; Debashis Ghosh ; Arndt Benecke ; Michael G. KatzeSource :
- BMC systems biology [ 1752-0509 ] ; 2011.
Descripteurs français
- KwdFr :
- Analyse de profil d'expression de gènes, Analyse de regroupements, Chimiokines (génétique), Chimiokines (métabolisme), Humains, Infections de l'appareil respiratoire (immunologie), Infections de l'appareil respiratoire (virologie), Régulation de l'expression des gènes viraux (génétique), Sous-type H1N1 du virus de la grippe A (génétique), Sous-type H1N1 du virus de la grippe A (métabolisme), Sous-type H1N1 du virus de la grippe A (pathogénicité), Sous-type H5N1 du virus de la grippe A (génétique), Sous-type H5N1 du virus de la grippe A (métabolisme), Sous-type H5N1 du virus de la grippe A (pathogénicité), Virulence, Virus du SRAS (génétique), Virus du SRAS (métabolisme), Virus du SRAS (pathogénicité).
- MESH :
- génétique : Chimiokines, Régulation de l'expression des gènes viraux, Sous-type H1N1 du virus de la grippe A, Sous-type H5N1 du virus de la grippe A, Virus du SRAS.
- immunologie : Infections de l'appareil respiratoire.
- métabolisme : Chimiokines, Sous-type H1N1 du virus de la grippe A, Sous-type H5N1 du virus de la grippe A, Virus du SRAS.
- pathogénicité : Sous-type H1N1 du virus de la grippe A, Sous-type H5N1 du virus de la grippe A, Virus du SRAS.
- virologie : Infections de l'appareil respiratoire.
- Analyse de profil d'expression de gènes, Analyse de regroupements, Humains, Virulence.
English descriptors
- KwdEn :
- Chemokines (genetics), Chemokines (metabolism), Cluster Analysis, Gene Expression Profiling, Gene Expression Regulation, Viral (genetics), Humans, Influenza A Virus, H1N1 Subtype (genetics), Influenza A Virus, H1N1 Subtype (metabolism), Influenza A Virus, H1N1 Subtype (pathogenicity), Influenza A Virus, H5N1 Subtype (genetics), Influenza A Virus, H5N1 Subtype (metabolism), Influenza A Virus, H5N1 Subtype (pathogenicity), Respiratory Tract Infections (immunology), Respiratory Tract Infections (virology), SARS Virus (genetics), SARS Virus (metabolism), SARS Virus (pathogenicity), Virulence.
- MESH :
- chemical , genetics : Chemokines.
- chemical , metabolism : Chemokines.
- genetics : Gene Expression Regulation, Viral, Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- immunology : Respiratory Tract Infections.
- metabolism : Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- pathogenicity : Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- virology : Respiratory Tract Infections.
- Cluster Analysis, Gene Expression Profiling, Humans, Virulence.
Abstract
During respiratory viral infections host injury occurs due in part to inappropriate host responses. In this study we sought to uncover the host transcriptional responses underlying differences between high- and low-pathogenic infections.
DOI: 10.1186/1752-0509-5-202
PubMed: 22189154
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pubmed:22189154Le document en format XML
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<front><div type="abstract" xml:lang="en">During respiratory viral infections host injury occurs due in part to inappropriate host responses. In this study we sought to uncover the host transcriptional responses underlying differences between high- and low-pathogenic infections.</div>
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