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A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.

Identifieur interne : 001426 ( PubMed/Curation ); précédent : 001425; suivant : 001427

A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.

Auteurs : Stewart T. Chang [États-Unis] ; Nicolas Tchitchek ; Debashis Ghosh ; Arndt Benecke ; Michael G. Katze

Source :

BMC systems biology [ 1752-0509 ] ; 2011.

RBID : pubmed:22189154

Descripteurs français

English descriptors

KwdEn :
- Chemokines (genetics), Chemokines (metabolism), Cluster Analysis, Gene Expression Profiling, Gene Expression Regulation, Viral (genetics), Humans, Influenza A Virus, H1N1 Subtype (genetics), Influenza A Virus, H1N1 Subtype (metabolism), Influenza A Virus, H1N1 Subtype (pathogenicity), Influenza A Virus, H5N1 Subtype (genetics), Influenza A Virus, H5N1 Subtype (metabolism), Influenza A Virus, H5N1 Subtype (pathogenicity), Respiratory Tract Infections (immunology), Respiratory Tract Infections (virology), SARS Virus (genetics), SARS Virus (metabolism), SARS Virus (pathogenicity), Virulence.
MESH :
- chemical , genetics : Chemokines.
- chemical , metabolism : Chemokines.
- genetics : Gene Expression Regulation, Viral, Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- immunology : Respiratory Tract Infections.
- metabolism : Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- pathogenicity : Influenza A Virus, H1N1 Subtype, Influenza A Virus, H5N1 Subtype, SARS Virus.
- virology : Respiratory Tract Infections.
- Cluster Analysis, Gene Expression Profiling, Humans, Virulence.

Abstract

During respiratory viral infections host injury occurs due in part to inappropriate host responses. In this study we sought to uncover the host transcriptional responses underlying differences between high- and low-pathogenic infections.

DOI: 10.1186/1752-0509-5-202
PubMed: 22189154

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Le document en format XML

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<term>Infections de l'appareil respiratoire (virologie)</term>
<term>Régulation de l'expression des gènes viraux (génétique)</term>
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<front><div type="abstract" xml:lang="en">During respiratory viral infections host injury occurs due in part to inappropriate host responses. In this study we sought to uncover the host transcriptional responses underlying differences between high- and low-pathogenic infections.</div>
</front>
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<DateCompleted><Year>2012</Year>
<Month>06</Month>
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<Title>BMC systems biology</Title>
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<ArticleTitle>A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.</ArticleTitle>
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<Abstract><AbstractText Label="BACKGROUND" NlmCategory="BACKGROUND">During respiratory viral infections host injury occurs due in part to inappropriate host responses. In this study we sought to uncover the host transcriptional responses underlying differences between high- and low-pathogenic infections.</AbstractText>
<AbstractText Label="RESULTS" NlmCategory="RESULTS">From a compendium of 12 studies that included responses to influenza A subtype H5N1, reconstructed 1918 influenza A virus, and SARS coronavirus, we used meta-analysis to derive multiple gene expression signatures. We compared these signatures by their capacity to segregate biological conditions by pathogenicity and predict pathogenicity in a test data set. The highest-performing signature was expressed as a continuum in low-, medium-, and high-pathogenicity samples, suggesting a direct, analog relationship between expression and pathogenicity. This signature comprised 57 genes including a subnetwork of chemokines, implicating dysregulated cell recruitment in injury.</AbstractText>
<AbstractText Label="CONCLUSIONS" NlmCategory="CONCLUSIONS">Highly pathogenic viruses elicit expression of many of the same key genes as lower pathogenic viruses but to a higher degree. This increased degree of expression may result in the uncontrolled co-localization of inflammatory cell types and lead to irreversible host damage.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Chang</LastName>
<ForeName>Stewart T</ForeName>
<Initials>ST</Initials>
<AffiliationInfo><Affiliation>Department of Microbiology, University of Washington, Seattle WA, USA.</Affiliation>
</AffiliationInfo>
</Author>
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<ForeName>Nicolas</ForeName>
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<Author ValidYN="Y"><LastName>Ghosh</LastName>
<ForeName>Debashis</ForeName>
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<ForeName>Arndt</ForeName>
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<Author ValidYN="Y"><LastName>Katze</LastName>
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<Agency>NIAID NIH HHS</Agency>
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A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.

A chemokine gene expression signature derived from meta-analysis predicts the pathogenicity of viral respiratory infections.

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