Serveur d'exploration SRAS

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The interferon gamma gene polymorphism +874 A/T is associated with severe acute respiratory syndrome

Identifieur interne : 001487 ( Ncbi/Curation ); précédent : 001486; suivant : 001488

The interferon gamma gene polymorphism +874 A/T is associated with severe acute respiratory syndrome

Auteurs : Wai Po Chong ; Wk Eddie Ip ; Gloria Hoi Wan Tso ; Man Wai Ng ; Wilfred Hing Sang Wong ; Helen Ka Wai Law ; Raymond Wh Yung [République populaire de Chine] ; Eudora Y. Chow [République populaire de Chine] ; Kl Au [République populaire de Chine] ; Eric Yt Chan [République populaire de Chine] ; Wilina Lim [République populaire de Chine] ; Js Malik Peiris [République populaire de Chine] ; Yu Lung Lau

Source :

RBID : PMC:1468415

Abstract

Background

Cytokines play important roles in antiviral action. We examined whether polymorphisms of IFN-γ,TNF-α and IL-10 affect the susceptibility to and outcome of severe acute respiratory syndrome (SARS).

Methods

A case-control study was carried out in 476 Chinese SARS patients and 449 healthy controls. We tested the polymorphisms of IFN-γ,TNF-α and IL-10 for their associations with SARS.

Results

IFN-γ +874A allele was associated with susceptibility to SARS in a dose-dependent manner (P < 0.001). Individuals with IFN-γ +874 AA and AT genotype had a 5.19-fold (95% Confidence Interval [CI], 2.78-9.68) and 2.57-fold (95% CI, 1.35-4.88) increased risk of developing SARS respectively. The polymorphisms of IL-10 and TNF-α were not associated with SARS susceptibility.

Conclusion

IFN-γ +874A allele was shown to be a risk factor in SARS susceptibility.


Url:
DOI: 10.1186/1471-2334-6-82
PubMed: 16672072
PubMed Central: 1468415

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PMC:1468415

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Wai Po Chong
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Wk Eddie Ip
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Gloria Hoi Wan Tso
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Man Wai Ng
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Wilfred Hing Sang Wong
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Helen Ka Wai Law
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Yu Lung Lau
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</author>
<author>
<name sortKey="Peiris, Js Malik" sort="Peiris, Js Malik" uniqKey="Peiris J" first="Js Malik" last="Peiris">Js Malik Peiris</name>
<affiliation wicri:level="1">
<nlm:aff id="Aff7">
<institution-wrap>
<institution-id institution-id-type="GRID">grid.194645.b</institution-id>
<institution-id institution-id-type="ISNI">0000000121742757</institution-id>
<institution>Department of Microbiology,</institution>
<institution>The Hong Kong Jockey Club Research Centre, Pokfulam, The University of Hong Kong,</institution>
</institution-wrap>
Hong Kong SAR, China</nlm:aff>
<country xml:lang="fr">République populaire de Chine</country>
<wicri:regionArea>Hong Kong SAR</wicri:regionArea>
</affiliation>
</author>
<author>
<name sortKey="Lau, Yu Lung" sort="Lau, Yu Lung" uniqKey="Lau Y" first="Yu Lung" last="Lau">Yu Lung Lau</name>
<affiliation>
<nlm:aff id="Aff1">
<institution-wrap>
<institution-id institution-id-type="GRID">grid.194645.b</institution-id>
<institution-id institution-id-type="ISNI">0000000121742757</institution-id>
<institution>Department of Paediatrics and Adolescent Medicine, The Hong Kong Jockey Club Research Centre,</institution>
<institution>The University of Hong Kong,</institution>
</institution-wrap>
Pokfulam, Hong Kong SAR China</nlm:aff>
<wicri:noCountry code="subfield">Hong Kong SAR China</wicri:noCountry>
</affiliation>
</author>
</analytic>
<series>
<title level="j">BMC Infectious Diseases</title>
<idno type="eISSN">1471-2334</idno>
<imprint>
<date when="2006">2006</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc>
<textClass></textClass>
</profileDesc>
</teiHeader>
<front>
<div type="abstract" xml:lang="en">
<sec>
<title>Background</title>
<p>Cytokines play important roles in antiviral action. We examined whether polymorphisms of
<italic>IFN-γ</italic>
,
<italic>TNF-α</italic>
and
<italic>IL-10</italic>
affect the susceptibility to and outcome of severe acute respiratory syndrome (SARS).</p>
</sec>
<sec>
<title>Methods</title>
<p>A case-control study was carried out in 476 Chinese SARS patients and 449 healthy controls. We tested the polymorphisms of
<italic>IFN-γ</italic>
,
<italic>TNF-α</italic>
and
<italic>IL-10</italic>
for their associations with SARS.</p>
</sec>
<sec>
<title>Results</title>
<p>
<italic>IFN-γ</italic>
+874A allele was associated with susceptibility to SARS in a dose-dependent manner (
<italic>P</italic>
< 0.001). Individuals with
<italic>IFN-γ</italic>
+874 AA and AT genotype had a 5.19-fold (95% Confidence Interval [CI], 2.78-9.68) and 2.57-fold (95% CI, 1.35-4.88) increased risk of developing SARS respectively. The polymorphisms of
<italic>IL-10</italic>
and
<italic>TNF-α</italic>
were not associated with SARS susceptibility.</p>
</sec>
<sec>
<title>Conclusion</title>
<p>
<italic>IFN-γ</italic>
+874A allele was shown to be a risk factor in SARS susceptibility.</p>
</sec>
</div>
</front>
<back>
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