SidaGhanaV1, PascalFrancis, Curation, bibRecord, 000040

Higher viral load may explain the dominance of CRF02―AG in the molecular epidemiology of HIV in Ghana

Identifieur interne : 000040 ( PascalFrancis/Curation ); précédent : 000039; suivant : 000041

Higher viral load may explain the dominance of CRF02―AG in the molecular epidemiology of HIV in Ghana

Auteurs : Lucia Fischetti [Royaume-Uni] ; Ohene Opare-Sem [Ghana] ; Daniel Candotti [Royaume-Uni] ; Helen Lee [Royaume-Uni] ; Jean-Pierre Allain [Royaume-Uni]

Source :

AIDS : (London) [ 0269-9370 ] ; 2004.

RBID : Pascal:05-0065331

Descripteurs français

Pascal (Inist)
- SIDA, Réaction chaîne polymérase, Charge virale, Epidémiologie moléculaire, Ghana, Virus immunodéficience humaine, Virus HIV1, Arbre phylogénétique, Plasma sanguin, Asymptomatique, Donneur sang, Homme, Prévalence, RNA-directed DNA polymerase, Forme moléculaire, Pouvoir infectant, Reverse transcriptase.
Wicri :
- geographic : Ghana.
- topic : Homme.

English descriptors

KwdEn :
- AIDS, Asymptomatic, Blood donor, Blood plasma, Ghana, HIV-1 virus, Human, Human immunodeficiency virus, Infectivity, Molecular epidemiology, Molecular form, Phylogenetic tree, Polymerase chain reaction, Prevalence, RNA-directed DNA polymerase, Viral load.

Abstract

Phylogenetic analysis of Ghanaian plasma samples from 84 asymptomatic candidate blood donors and 150 AIDS patients revealed a 63% prevalence of HIV-1 CRF02―AG. HIV-1 viral load in both populations was quantified using real-time reverse transcriptase-polymerase chain reaction. In asymptomatic candidate blood donors infected with CRF02―AG, the viral load was significantly higher than in donors infected with other HIV-1 molecular forms. A higher viral load of CRF02―AG is compatible with higher infectivity and prevalence.

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C03	`10`	`X`	`SPA`	`@0 Asintomático @5 15`
C03	`11`	`X`	`FRE`	`@0 Donneur sang @5 17`
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C03	`11`	`X`	`SPA`	`@0 Donador sangre @5 17`
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C03	`16`	`X`	`SPA`	`@0 Poder infectante @5 22`
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C07	`09`	`X`	`FRE`	`@0 Enzyme @2 FE`
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C07	`11`	`X`	`SPA`	`@0 Inmunopatología @5 38`
C07	`12`	`X`	`FRE`	`@0 Immunodéficit @5 39`
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Pascal:05-0065331

Le document en format XML

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<term>Ghana</term>
<term>HIV-1 virus</term>
<term>Human</term>
<term>Human immunodeficiency virus</term>
<term>Infectivity</term>
<term>Molecular epidemiology</term>
<term>Molecular form</term>
<term>Phylogenetic tree</term>
<term>Polymerase chain reaction</term>
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<term>Virus immunodéficience humaine</term>
<term>Virus HIV1</term>
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<front><div type="abstract" xml:lang="en">Phylogenetic analysis of Ghanaian plasma samples from 84 asymptomatic candidate blood donors and 150 AIDS patients revealed a 63% prevalence of HIV-1 CRF02―AG. HIV-1 viral load in both populations was quantified using real-time reverse transcriptase-polymerase chain reaction. In asymptomatic candidate blood donors infected with CRF02―AG, the viral load was significantly higher than in donors infected with other HIV-1 molecular forms. A higher viral load of CRF02―AG is compatible with higher infectivity and prevalence.</div>
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<fA11 i1="01" i2="1"><s1>FISCHETTI (Lucia)</s1>
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<fC01 i1="01" l="ENG"><s0>Phylogenetic analysis of Ghanaian plasma samples from 84 asymptomatic candidate blood donors and 150 AIDS patients revealed a 63% prevalence of HIV-1 CRF02―AG. HIV-1 viral load in both populations was quantified using real-time reverse transcriptase-polymerase chain reaction. In asymptomatic candidate blood donors infected with CRF02―AG, the viral load was significantly higher than in donors infected with other HIV-1 molecular forms. A higher viral load of CRF02―AG is compatible with higher infectivity and prevalence.</s0>
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<s5>07</s5>
</fC03>
<fC03 i1="03" i2="X" l="ENG"><s0>Viral load</s0>
<s5>07</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA"><s0>Carga vírica</s0>
<s5>07</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE"><s0>Epidémiologie moléculaire</s0>
<s5>08</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG"><s0>Molecular epidemiology</s0>
<s5>08</s5>
</fC03>
<fC03 i1="04" i2="X" l="SPA"><s0>Epidemiología molecular</s0>
<s5>08</s5>
</fC03>
<fC03 i1="05" i2="X" l="FRE"><s0>Ghana</s0>
<s2>NG</s2>
<s5>09</s5>
</fC03>
<fC03 i1="05" i2="X" l="ENG"><s0>Ghana</s0>
<s2>NG</s2>
<s5>09</s5>
</fC03>
<fC03 i1="05" i2="X" l="SPA"><s0>Ghana</s0>
<s2>NG</s2>
<s5>09</s5>
</fC03>
<fC03 i1="06" i2="X" l="FRE"><s0>Virus immunodéficience humaine</s0>
<s2>NW</s2>
<s5>10</s5>
</fC03>
<fC03 i1="06" i2="X" l="ENG"><s0>Human immunodeficiency virus</s0>
<s2>NW</s2>
<s5>10</s5>
</fC03>
<fC03 i1="06" i2="X" l="SPA"><s0>Human immunodeficiency virus</s0>
<s2>NW</s2>
<s5>10</s5>
</fC03>
<fC03 i1="07" i2="X" l="FRE"><s0>Virus HIV1</s0>
<s2>NW</s2>
<s5>11</s5>
</fC03>
<fC03 i1="07" i2="X" l="ENG"><s0>HIV-1 virus</s0>
<s2>NW</s2>
<s5>11</s5>
</fC03>
<fC03 i1="07" i2="X" l="SPA"><s0>HIV-1 virus</s0>
<s2>NW</s2>
<s5>11</s5>
</fC03>
<fC03 i1="08" i2="X" l="FRE"><s0>Arbre phylogénétique</s0>
<s5>13</s5>
</fC03>
<fC03 i1="08" i2="X" l="ENG"><s0>Phylogenetic tree</s0>
<s5>13</s5>
</fC03>
<fC03 i1="08" i2="X" l="SPA"><s0>Arbol filogenético</s0>
<s5>13</s5>
</fC03>
<fC03 i1="09" i2="X" l="FRE"><s0>Plasma sanguin</s0>
<s5>14</s5>
</fC03>
<fC03 i1="09" i2="X" l="ENG"><s0>Blood plasma</s0>
<s5>14</s5>
</fC03>
<fC03 i1="09" i2="X" l="SPA"><s0>Plasma sanguíneo</s0>
<s5>14</s5>
</fC03>
<fC03 i1="10" i2="X" l="FRE"><s0>Asymptomatique</s0>
<s5>15</s5>
</fC03>
<fC03 i1="10" i2="X" l="ENG"><s0>Asymptomatic</s0>
<s5>15</s5>
</fC03>
<fC03 i1="10" i2="X" l="SPA"><s0>Asintomático</s0>
<s5>15</s5>
</fC03>
<fC03 i1="11" i2="X" l="FRE"><s0>Donneur sang</s0>
<s5>17</s5>
</fC03>
<fC03 i1="11" i2="X" l="ENG"><s0>Blood donor</s0>
<s5>17</s5>
</fC03>
<fC03 i1="11" i2="X" l="SPA"><s0>Donador sangre</s0>
<s5>17</s5>
</fC03>
<fC03 i1="12" i2="X" l="FRE"><s0>Homme</s0>
<s5>18</s5>
</fC03>
<fC03 i1="12" i2="X" l="ENG"><s0>Human</s0>
<s5>18</s5>
</fC03>
<fC03 i1="12" i2="X" l="SPA"><s0>Hombre</s0>
<s5>18</s5>
</fC03>
<fC03 i1="13" i2="X" l="FRE"><s0>Prévalence</s0>
<s5>19</s5>
</fC03>
<fC03 i1="13" i2="X" l="ENG"><s0>Prevalence</s0>
<s5>19</s5>
</fC03>
<fC03 i1="13" i2="X" l="SPA"><s0>Prevalencia</s0>
<s5>19</s5>
</fC03>
<fC03 i1="14" i2="X" l="FRE"><s0>RNA-directed DNA polymerase</s0>
<s2>FE</s2>
<s5>20</s5>
</fC03>
<fC03 i1="14" i2="X" l="ENG"><s0>RNA-directed DNA polymerase</s0>
<s2>FE</s2>
<s5>20</s5>
</fC03>
<fC03 i1="14" i2="X" l="SPA"><s0>RNA-directed DNA polymerase</s0>
<s2>FE</s2>
<s5>20</s5>
</fC03>
<fC03 i1="15" i2="X" l="FRE"><s0>Forme moléculaire</s0>
<s5>21</s5>
</fC03>
<fC03 i1="15" i2="X" l="ENG"><s0>Molecular form</s0>
<s5>21</s5>
</fC03>
<fC03 i1="15" i2="X" l="SPA"><s0>Forma molecular</s0>
<s5>21</s5>
</fC03>
<fC03 i1="16" i2="X" l="FRE"><s0>Pouvoir infectant</s0>
<s5>22</s5>
</fC03>
<fC03 i1="16" i2="X" l="ENG"><s0>Infectivity</s0>
<s5>22</s5>
</fC03>
<fC03 i1="16" i2="X" l="SPA"><s0>Poder infectante</s0>
<s5>22</s5>
</fC03>
<fC03 i1="17" i2="X" l="FRE"><s0>Reverse transcriptase</s0>
<s4>INC</s4>
<s5>86</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE"><s0>Virose</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="01" i2="X" l="ENG"><s0>Viral disease</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="01" i2="X" l="SPA"><s0>Virosis</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="02" i2="X" l="FRE"><s0>Infection</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="02" i2="X" l="ENG"><s0>Infection</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="02" i2="X" l="SPA"><s0>Infección</s0>
<s2>NM</s2>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Afrique</s0>
<s2>NG</s2>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Africa</s0>
<s2>NG</s2>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Africa</s0>
<s2>NG</s2>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Lentivirus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Lentivirus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Lentivirus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Retroviridae</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="05" i2="X" l="ENG"><s0>Retroviridae</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="05" i2="X" l="SPA"><s0>Retroviridae</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="06" i2="X" l="FRE"><s0>Virus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="06" i2="X" l="ENG"><s0>Virus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="06" i2="X" l="SPA"><s0>Virus</s0>
<s2>NW</s2>
</fC07>
<fC07 i1="07" i2="X" l="FRE"><s0>Nucleotidyltransferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="07" i2="X" l="ENG"><s0>Nucleotidyltransferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="07" i2="X" l="SPA"><s0>Nucleotidyltransferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="08" i2="X" l="FRE"><s0>Transferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="08" i2="X" l="ENG"><s0>Transferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="08" i2="X" l="SPA"><s0>Transferases</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="09" i2="X" l="FRE"><s0>Enzyme</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="09" i2="X" l="ENG"><s0>Enzyme</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="09" i2="X" l="SPA"><s0>Enzima</s0>
<s2>FE</s2>
</fC07>
<fC07 i1="10" i2="X" l="FRE"><s0>Exploration microbiologique</s0>
<s5>37</s5>
</fC07>
<fC07 i1="10" i2="X" l="ENG"><s0>Microbiological investigation</s0>
<s5>37</s5>
</fC07>
<fC07 i1="10" i2="X" l="SPA"><s0>Análisis microbiológico</s0>
<s5>37</s5>
</fC07>
<fC07 i1="11" i2="X" l="FRE"><s0>Immunopathologie</s0>
<s5>38</s5>
</fC07>
<fC07 i1="11" i2="X" l="ENG"><s0>Immunopathology</s0>
<s5>38</s5>
</fC07>
<fC07 i1="11" i2="X" l="SPA"><s0>Inmunopatología</s0>
<s5>38</s5>
</fC07>
<fC07 i1="12" i2="X" l="FRE"><s0>Immunodéficit</s0>
<s5>39</s5>
</fC07>
<fC07 i1="12" i2="X" l="ENG"><s0>Immune deficiency</s0>
<s5>39</s5>
</fC07>
<fC07 i1="12" i2="X" l="SPA"><s0>Inmunodeficiencia</s0>
<s5>39</s5>
</fC07>
<fC07 i1="13" i2="X" l="FRE"><s0>Biologie moléculaire</s0>
<s5>41</s5>
</fC07>
<fC07 i1="13" i2="X" l="ENG"><s0>Molecular biology</s0>
<s5>41</s5>
</fC07>
<fC07 i1="13" i2="X" l="SPA"><s0>Biología molecular</s0>
<s5>41</s5>
</fC07>
<fN21><s1>031</s1>
</fN21>
<fN44 i1="01"><s1>OTO</s1>
</fN44>
<fN82><s1>OTO</s1>
</fN82>
</pA>
</standard>
</inist>
</record>

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   |texte=   Higher viral load may explain the dominance of CRF02―AG in the molecular epidemiology of HIV in Ghana
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	Le SIDA au Ghana (serveur d'exploration)
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Le SIDA au Ghana (serveur d'exploration)

Higher viral load may explain the dominance of CRF02―AG in the molecular epidemiology of HIV in Ghana

Higher viral load may explain the dominance of CRF02―AG in the molecular epidemiology of HIV in Ghana

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