A specific clinical pattern of camptocormia in Parkinson's disease.
Identifieur interne : 000E31 ( PubMed/Corpus ); précédent : 000E30; suivant : 000E32A specific clinical pattern of camptocormia in Parkinson's disease.
Auteurs : A-C Lepoutre ; D. Devos ; A. Blanchard-Dauphin ; V. Pardessus ; C-A Maurage ; D. Ferriby ; J-F Hurtevent ; A. Cotten ; A. Destée ; L. DefebvreSource :
- Journal of neurology, neurosurgery, and psychiatry [ 1468-330X ] ; 2006.
English descriptors
- KwdEn :
- MESH :
- complications : Parkinson Disease.
- etiology : Dystonia.
- pathology : Brain, Muscle, Skeletal, Spine.
- physiopathology : Dystonia.
- Aged, Cross-Sectional Studies, Female, Humans, Magnetic Resonance Imaging, Male, Middle Aged, Posture.
Abstract
Camptocormia, characterised by extreme forward flexion of the thoracolumbar spine and severe stooping in the supine position, seems to be prevalent in Parkinson's disease.
DOI: 10.1136/jnnp.2005.083998
PubMed: 16735399
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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">A specific clinical pattern of camptocormia in Parkinson's disease.</title><author><name sortKey="Lepoutre, A C" sort="Lepoutre, A C" uniqKey="Lepoutre A" first="A-C" last="Lepoutre">A-C Lepoutre</name><affiliation><nlm:affiliation>Department of Neurology, EA2683, IFR 114, Lille University Medical Centre, Lille, France.</nlm:affiliation></affiliation></author><author><name sortKey="Devos, D" sort="Devos, D" uniqKey="Devos D" first="D" last="Devos">D. Devos</name></author><author><name sortKey="Blanchard Dauphin, A" sort="Blanchard Dauphin, A" uniqKey="Blanchard Dauphin A" first="A" last="Blanchard-Dauphin">A. Blanchard-Dauphin</name></author><author><name sortKey="Pardessus, V" sort="Pardessus, V" uniqKey="Pardessus V" first="V" last="Pardessus">V. Pardessus</name></author><author><name sortKey="Maurage, C A" sort="Maurage, C A" uniqKey="Maurage C" first="C-A" last="Maurage">C-A Maurage</name></author><author><name sortKey="Ferriby, D" sort="Ferriby, D" uniqKey="Ferriby D" first="D" last="Ferriby">D. Ferriby</name></author><author><name sortKey="Hurtevent, J F" sort="Hurtevent, J F" uniqKey="Hurtevent J" first="J-F" last="Hurtevent">J-F Hurtevent</name></author><author><name sortKey="Cotten, A" sort="Cotten, A" uniqKey="Cotten A" first="A" last="Cotten">A. Cotten</name></author><author><name sortKey="Destee, A" sort="Destee, A" uniqKey="Destee A" first="A" last="Destée">A. Destée</name></author><author><name sortKey="Defebvre, L" sort="Defebvre, L" uniqKey="Defebvre L" first="L" last="Defebvre">L. Defebvre</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="2006">2006</date><idno type="RBID">pubmed:16735399</idno><idno type="pmid">16735399</idno><idno type="doi">10.1136/jnnp.2005.083998</idno><idno type="wicri:Area/PubMed/Corpus">000E31</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">000E31</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">A specific clinical pattern of camptocormia in Parkinson's disease.</title><author><name sortKey="Lepoutre, A C" sort="Lepoutre, A C" uniqKey="Lepoutre A" first="A-C" last="Lepoutre">A-C Lepoutre</name><affiliation><nlm:affiliation>Department of Neurology, EA2683, IFR 114, Lille University Medical Centre, Lille, France.</nlm:affiliation></affiliation></author><author><name sortKey="Devos, D" sort="Devos, D" uniqKey="Devos D" first="D" last="Devos">D. Devos</name></author><author><name sortKey="Blanchard Dauphin, A" sort="Blanchard Dauphin, A" uniqKey="Blanchard Dauphin A" first="A" last="Blanchard-Dauphin">A. Blanchard-Dauphin</name></author><author><name sortKey="Pardessus, V" sort="Pardessus, V" uniqKey="Pardessus V" first="V" last="Pardessus">V. Pardessus</name></author><author><name sortKey="Maurage, C A" sort="Maurage, C A" uniqKey="Maurage C" first="C-A" last="Maurage">C-A Maurage</name></author><author><name sortKey="Ferriby, D" sort="Ferriby, D" uniqKey="Ferriby D" first="D" last="Ferriby">D. Ferriby</name></author><author><name sortKey="Hurtevent, J F" sort="Hurtevent, J F" uniqKey="Hurtevent J" first="J-F" last="Hurtevent">J-F Hurtevent</name></author><author><name sortKey="Cotten, A" sort="Cotten, A" uniqKey="Cotten A" first="A" last="Cotten">A. Cotten</name></author><author><name sortKey="Destee, A" sort="Destee, A" uniqKey="Destee A" first="A" last="Destée">A. Destée</name></author><author><name sortKey="Defebvre, L" sort="Defebvre, L" uniqKey="Defebvre L" first="L" last="Defebvre">L. Defebvre</name></author></analytic><series><title level="j">Journal of neurology, neurosurgery, and psychiatry</title><idno type="eISSN">1468-330X</idno><imprint><date when="2006" type="published">2006</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Brain (pathology)</term><term>Cross-Sectional Studies</term><term>Dystonia (etiology)</term><term>Dystonia (physiopathology)</term><term>Female</term><term>Humans</term><term>Magnetic Resonance Imaging</term><term>Male</term><term>Middle Aged</term><term>Muscle, Skeletal (pathology)</term><term>Parkinson Disease (complications)</term><term>Posture</term><term>Spine (pathology)</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Brain</term><term>Muscle, Skeletal</term><term>Spine</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Cross-Sectional Studies</term><term>Female</term><term>Humans</term><term>Magnetic Resonance Imaging</term><term>Male</term><term>Middle Aged</term><term>Posture</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Camptocormia, characterised by extreme forward flexion of the thoracolumbar spine and severe stooping in the supine position, seems to be prevalent in Parkinson's disease.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">16735399</PMID><DateCreated><Year>2006</Year><Month>10</Month><Day>17</Day></DateCreated><DateCompleted><Year>2006</Year><Month>10</Month><Day>27</Day></DateCompleted><DateRevised><Year>2014</Year><Month>09</Month><Day>09</Day></DateRevised><Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1468-330X</ISSN><JournalIssue CitedMedium="Internet"><Volume>77</Volume><Issue>11</Issue><PubDate><Year>2006</Year><Month>Nov</Month></PubDate></JournalIssue><Title>Journal of neurology, neurosurgery, and psychiatry</Title><ISOAbbreviation>J. Neurol. Neurosurg. Psychiatr.</ISOAbbreviation></Journal><ArticleTitle>A specific clinical pattern of camptocormia in Parkinson's disease.</ArticleTitle><Pagination><MedlinePgn>1229-34</MedlinePgn></Pagination><Abstract><AbstractText Label="BACKGROUND" NlmCategory="BACKGROUND">Camptocormia, characterised by extreme forward flexion of the thoracolumbar spine and severe stooping in the supine position, seems to be prevalent in Parkinson's disease.</AbstractText><AbstractText Label="OBJECTIVE" NlmCategory="OBJECTIVE">The aim of this study was to identify features of parkinsonian camptocormia and to describe the main clinical characteristics of patients with Parkinson's disease who develop the condition.</AbstractText><AbstractText Label="METHODS" NlmCategory="METHODS">An extensive range of clinical, biochemical and imaging data were gathered for 23 patients with Parkinson's disease with camptocormia, notably including magnetic resonance imaging (MRI) of the brain and spine, electromyographic recordings of the paravertebral muscles and muscle biopsies.</AbstractText><AbstractText Label="RESULTS" NlmCategory="RESULTS">Camptocormia occurred in severe Parkinson's disease with axial predominance, motor fluctuations and dysautonomic symptoms. The condition was often associated with spondyloarthritic changes and pain. MRI showed paraspinal muscle signal abnormalities in five patients and fatty involution in seven patients. The seven patients had motor unit reductions on the spinal erector electromyogram. The MRI results for the girdle muscles were normal. Cranial MRI showed signal abnormalities for the basal ganglia in three patients.</AbstractText><AbstractText Label="DISCUSSION" NlmCategory="CONCLUSIONS">Various mechanisms may contribute to the development of parkinsonian camptocormia: dopaminergic depletion in Parkinson's disease induces functional changes in the organisation of the corticospinal and reticulospinal tracts, where dysfunction could contribute to axial rigidity. Furthermore, rigidity of the spinal flexion muscles could lead to under-use of the spinal extension muscles, which become progressively atrophic. Rigidity may also induce spinal deformations, leading to a neurogenic syndrome via compression of the spinal nerves.</AbstractText><AbstractText Label="CONCLUSION" NlmCategory="CONCLUSIONS">The screening and early management of camptocormia in Parkinson's disease is likely to be important for preventing axial disorders and spinal deformations.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Lepoutre</LastName><ForeName>A-C</ForeName><Initials>AC</Initials><AffiliationInfo><Affiliation>Department of Neurology, EA2683, IFR 114, Lille University Medical Centre, Lille, France.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Devos</LastName><ForeName>D</ForeName><Initials>D</Initials></Author><Author ValidYN="Y"><LastName>Blanchard-Dauphin</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Pardessus</LastName><ForeName>V</ForeName><Initials>V</Initials></Author><Author ValidYN="Y"><LastName>Maurage</LastName><ForeName>C-A</ForeName><Initials>CA</Initials></Author><Author ValidYN="Y"><LastName>Ferriby</LastName><ForeName>D</ForeName><Initials>D</Initials></Author><Author ValidYN="Y"><LastName>Hurtevent</LastName><ForeName>J-F</ForeName><Initials>JF</Initials></Author><Author ValidYN="Y"><LastName>Cotten</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Destée</LastName><ForeName>A</ForeName><Initials>A</Initials></Author><Author ValidYN="Y"><LastName>Defebvre</LastName><ForeName>L</ForeName><Initials>L</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList><ArticleDate DateType="Electronic"><Year>2006</Year><Month>05</Month><Day>30</Day></ArticleDate></Article><MedlineJournalInfo><Country>England</Country><MedlineTA>J Neurol Neurosurg Psychiatry</MedlineTA><NlmUniqueID>2985191R</NlmUniqueID><ISSNLinking>0022-3050</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><CommentsCorrectionsList><CommentsCorrections RefType="Cites"><RefSource>Epilepsy Res. 1987 Jul;1(4):254-7</RefSource><PMID Version="1">3143551</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Acta Neuropathol. 1976 Mar 30;34(3):183-97</RefSource><PMID Version="1">178142</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Neuromuscul Disord. 1993 Sep-Nov;3(5-6):579-82</RefSource><PMID Version="1">8186716</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Rev Rhum Ed Fr. 1994 Dec;61(11):868-70</RefSource><PMID Version="1">7858588</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Spine (Phila Pa 1976). 1995 May 1;20(9):1011-6</RefSource><PMID Version="1">7631230</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>J 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2003 Apr 22;60(8):1393-4</RefSource><PMID Version="1">12707457</PMID></CommentsCorrections><CommentsCorrections RefType="Cites"><RefSource>Adv Neurol. 1975;9:165-75</RefSource><PMID Version="1">1146651</PMID></CommentsCorrections><CommentsCorrections RefType="CommentIn"><RefSource>J Neurol Neurosurg Psychiatry. 2006 Nov;77(11):1205</RefSource><PMID Version="1">17043289</PMID></CommentsCorrections></CommentsCorrectionsList><MeshHeadingList><MeshHeading><DescriptorName UI="D000368" MajorTopicYN="N">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001921" MajorTopicYN="N">Brain</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D003430" MajorTopicYN="N">Cross-Sectional Studies</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D004421" MajorTopicYN="N">Dystonia</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008279" MajorTopicYN="N">Magnetic Resonance Imaging</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008875" MajorTopicYN="N">Middle Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D018482" MajorTopicYN="N">Muscle, Skeletal</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D010300" MajorTopicYN="N">Parkinson Disease</DescriptorName><QualifierName UI="Q000150" MajorTopicYN="Y">complications</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011187" MajorTopicYN="Y">Posture</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D013131" MajorTopicYN="N">Spine</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="Y">pathology</QualifierName></MeshHeading></MeshHeadingList><OtherID Source="NLM">PMC2077366</OtherID></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2006</Year><Month>6</Month><Day>1</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2006</Year><Month>10</Month><Day>28</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2006</Year><Month>6</Month><Day>1</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">16735399</ArticleId><ArticleId IdType="pii">jnnp.2005.083998</ArticleId><ArticleId IdType="doi">10.1136/jnnp.2005.083998</ArticleId><ArticleId 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