Parkinson's disease with camptocormia.
Identifieur interne : 000E30 ( PubMed/Corpus ); précédent : 000E29; suivant : 000E31Parkinson's disease with camptocormia.
Auteurs : F. Bloch ; J L Houeto ; S. Tezenas Du Montcel ; F. Bonneville ; F. Etchepare ; M L Welter ; S. Rivaud-Pechoux ; V. Hahn-Barma ; T. Maisonobe ; C. Behar ; J Y Lazennec ; E. Kurys ; I. Arnulf ; A M Bonnet ; Y. AgidSource :
- Journal of neurology, neurosurgery, and psychiatry [ 1468-330X ] ; 2006.
English descriptors
- KwdEn :
- Aged, Basal Ganglia (physiopathology), Dystonia (etiology), Dystonia (physiopathology), Electromyography, Electrophysiology, Female, Humans, Male, Middle Aged, Muscle, Skeletal (pathology), Parkinson Disease (complications), Parkinson Disease (physiopathology), Posture, Prospective Studies, Walking.
- MESH :
- complications : Parkinson Disease.
- etiology : Dystonia.
- pathology : Muscle, Skeletal.
- physiopathology : Basal Ganglia, Dystonia, Parkinson Disease.
- Aged, Electromyography, Electrophysiology, Female, Humans, Male, Middle Aged, Posture, Prospective Studies, Walking.
Abstract
Camptocormia is defined as an abnormal flexion of the trunk that appears when standing or walking and disappears in the supine position. The origin of the disorder is unknown, but it is usually attributed either to a primary or a secondary paravertebral muscle myopathy or a motor neurone disorder. Camptocormia is also observed in a minority of patients with parkinsonism.
DOI: 10.1136/jnnp.2006.087908
PubMed: 16754693
Links to Exploration step
pubmed:16754693Le document en format XML
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Etchepare</name></author><author><name sortKey="Welter, M L" sort="Welter, M L" uniqKey="Welter M" first="M L" last="Welter">M L Welter</name></author><author><name sortKey="Rivaud Pechoux, S" sort="Rivaud Pechoux, S" uniqKey="Rivaud Pechoux S" first="S" last="Rivaud-Pechoux">S. Rivaud-Pechoux</name></author><author><name sortKey="Hahn Barma, V" sort="Hahn Barma, V" uniqKey="Hahn Barma V" first="V" last="Hahn-Barma">V. Hahn-Barma</name></author><author><name sortKey="Maisonobe, T" sort="Maisonobe, T" uniqKey="Maisonobe T" first="T" last="Maisonobe">T. Maisonobe</name></author><author><name sortKey="Behar, C" sort="Behar, C" uniqKey="Behar C" first="C" last="Behar">C. Behar</name></author><author><name sortKey="Lazennec, J Y" sort="Lazennec, J Y" uniqKey="Lazennec J" first="J Y" last="Lazennec">J Y Lazennec</name></author><author><name sortKey="Kurys, E" sort="Kurys, E" uniqKey="Kurys E" first="E" last="Kurys">E. Kurys</name></author><author><name sortKey="Arnulf, I" sort="Arnulf, I" uniqKey="Arnulf I" first="I" last="Arnulf">I. Arnulf</name></author><author><name sortKey="Bonnet, A M" sort="Bonnet, A M" uniqKey="Bonnet A" first="A M" last="Bonnet">A M Bonnet</name></author><author><name sortKey="Agid, Y" sort="Agid, Y" uniqKey="Agid Y" first="Y" last="Agid">Y. Agid</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="2006">2006</date><idno type="RBID">pubmed:16754693</idno><idno type="pmid">16754693</idno><idno type="doi">10.1136/jnnp.2006.087908</idno><idno type="wicri:Area/PubMed/Corpus">000E30</idno><idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">000E30</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Parkinson's disease with camptocormia.</title><author><name sortKey="Bloch, F" sort="Bloch, F" uniqKey="Bloch F" first="F" last="Bloch">F. Bloch</name><affiliation><nlm:affiliation>Centre d'Investigation Clinique-Fédération des Maladies du Système Nerveux, Groupe-Hospitalier Pitié-Salpêtrière, Paris, France.</nlm:affiliation></affiliation></author><author><name sortKey="Houeto, J L" sort="Houeto, J L" uniqKey="Houeto J" first="J L" last="Houeto">J L Houeto</name></author><author><name sortKey="Tezenas Du Montcel, S" sort="Tezenas Du Montcel, S" uniqKey="Tezenas Du Montcel S" first="S" last="Tezenas Du Montcel">S. Tezenas Du Montcel</name></author><author><name sortKey="Bonneville, F" sort="Bonneville, F" uniqKey="Bonneville F" first="F" last="Bonneville">F. Bonneville</name></author><author><name sortKey="Etchepare, F" sort="Etchepare, F" uniqKey="Etchepare F" first="F" last="Etchepare">F. Etchepare</name></author><author><name sortKey="Welter, M L" sort="Welter, M L" uniqKey="Welter M" first="M L" last="Welter">M L Welter</name></author><author><name sortKey="Rivaud Pechoux, S" sort="Rivaud Pechoux, S" uniqKey="Rivaud Pechoux S" first="S" last="Rivaud-Pechoux">S. Rivaud-Pechoux</name></author><author><name sortKey="Hahn Barma, V" sort="Hahn Barma, V" uniqKey="Hahn Barma V" first="V" last="Hahn-Barma">V. Hahn-Barma</name></author><author><name sortKey="Maisonobe, T" sort="Maisonobe, T" uniqKey="Maisonobe T" first="T" last="Maisonobe">T. Maisonobe</name></author><author><name sortKey="Behar, C" sort="Behar, C" uniqKey="Behar C" first="C" last="Behar">C. Behar</name></author><author><name sortKey="Lazennec, J Y" sort="Lazennec, J Y" uniqKey="Lazennec J" first="J Y" last="Lazennec">J Y Lazennec</name></author><author><name sortKey="Kurys, E" sort="Kurys, E" uniqKey="Kurys E" first="E" last="Kurys">E. Kurys</name></author><author><name sortKey="Arnulf, I" sort="Arnulf, I" uniqKey="Arnulf I" first="I" last="Arnulf">I. Arnulf</name></author><author><name sortKey="Bonnet, A M" sort="Bonnet, A M" uniqKey="Bonnet A" first="A M" last="Bonnet">A M Bonnet</name></author><author><name sortKey="Agid, Y" sort="Agid, Y" uniqKey="Agid Y" first="Y" last="Agid">Y. Agid</name></author></analytic><series><title level="j">Journal of neurology, neurosurgery, and psychiatry</title><idno type="eISSN">1468-330X</idno><imprint><date when="2006" type="published">2006</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Aged</term><term>Basal Ganglia (physiopathology)</term><term>Dystonia (etiology)</term><term>Dystonia (physiopathology)</term><term>Electromyography</term><term>Electrophysiology</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Muscle, Skeletal (pathology)</term><term>Parkinson Disease (complications)</term><term>Parkinson Disease (physiopathology)</term><term>Posture</term><term>Prospective Studies</term><term>Walking</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Parkinson Disease</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Dystonia</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Muscle, Skeletal</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Basal Ganglia</term><term>Dystonia</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Aged</term><term>Electromyography</term><term>Electrophysiology</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Posture</term><term>Prospective Studies</term><term>Walking</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Camptocormia is defined as an abnormal flexion of the trunk that appears when standing or walking and disappears in the supine position. The origin of the disorder is unknown, but it is usually attributed either to a primary or a secondary paravertebral muscle myopathy or a motor neurone disorder. Camptocormia is also observed in a minority of patients with parkinsonism.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">16754693</PMID><DateCreated><Year>2006</Year><Month>10</Month><Day>17</Day></DateCreated><DateCompleted><Year>2006</Year><Month>10</Month><Day>27</Day></DateCompleted><DateRevised><Year>2014</Year><Month>09</Month><Day>09</Day></DateRevised><Article PubModel="Print-Electronic"><Journal><ISSN IssnType="Electronic">1468-330X</ISSN><JournalIssue CitedMedium="Internet"><Volume>77</Volume><Issue>11</Issue><PubDate><Year>2006</Year><Month>Nov</Month></PubDate></JournalIssue><Title>Journal of neurology, neurosurgery, and psychiatry</Title><ISOAbbreviation>J. Neurol. Neurosurg. Psychiatr.</ISOAbbreviation></Journal><ArticleTitle>Parkinson's disease with camptocormia.</ArticleTitle><Pagination><MedlinePgn>1223-8</MedlinePgn></Pagination><Abstract><AbstractText Label="BACKGROUND" NlmCategory="BACKGROUND">Camptocormia is defined as an abnormal flexion of the trunk that appears when standing or walking and disappears in the supine position. The origin of the disorder is unknown, but it is usually attributed either to a primary or a secondary paravertebral muscle myopathy or a motor neurone disorder. Camptocormia is also observed in a minority of patients with parkinsonism.</AbstractText><AbstractText Label="OBJECTIVE" NlmCategory="OBJECTIVE">To characterise the clinical and electrophysiological features of camptocormia and parkinsonian symptoms in patients with Parkinson's disease and camptocormia compared with patients with Parkinson's disease without camptocormia.</AbstractText><AbstractText Label="METHODS" NlmCategory="METHODS">Patients with parkinsonism and camptocormia (excluding patients with multiple system atrophy) prospectively underwent a multidisciplinary clinical (neurological, neuropsychological, psychological, rheumatological) and neurophysiological (electromyogram, ocular movement recording) examination and were compared with age-matched patients with Parkinson's disease without camptocormia.</AbstractText><AbstractText Label="RESULTS" NlmCategory="RESULTS">The camptocormia developed after 8.5 (SD 5.3) years of parkinsonism, responded poorly to levodopa treatment (20%) and displayed features consistent with axial dystonia. Patients with camptocormia were characterised by prominent levodopa-unresponsive axial symptoms (ie, axial rigidity, gait disorder and postural instability), along with a tendency for greater error in the antisaccade paradigm.</AbstractText><AbstractText Label="CONCLUSION" NlmCategory="CONCLUSIONS">We suggest that (1) the salient features of parkinsonism observed in patients with camptocormia are likely to represent a specific form of Parkinson's disease and camptocormia is an axial dystonia and (2) both camptocormia and parkinsonism in these patients might result from additional, non-dopaminergic neuronal dysfunction in the basal ganglia.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Bloch</LastName><ForeName>F</ForeName><Initials>F</Initials><AffiliationInfo><Affiliation>Centre d'Investigation Clinique-Fédération des Maladies du Système Nerveux, Groupe-Hospitalier Pitié-Salpêtrière, Paris, France.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Houeto</LastName><ForeName>J L</ForeName><Initials>JL</Initials></Author><Author ValidYN="Y"><LastName>Tezenas du Montcel</LastName><ForeName>S</ForeName><Initials>S</Initials></Author><Author ValidYN="Y"><LastName>Bonneville</LastName><ForeName>F</ForeName><Initials>F</Initials></Author><Author ValidYN="Y"><LastName>Etchepare</LastName><ForeName>F</ForeName><Initials>F</Initials></Author><Author ValidYN="Y"><LastName>Welter</LastName><ForeName>M L</ForeName><Initials>ML</Initials></Author><Author ValidYN="Y"><LastName>Rivaud-Pechoux</LastName><ForeName>S</ForeName><Initials>S</Initials></Author><Author ValidYN="Y"><LastName>Hahn-Barma</LastName><ForeName>V</ForeName><Initials>V</Initials></Author><Author ValidYN="Y"><LastName>Maisonobe</LastName><ForeName>T</ForeName><Initials>T</Initials></Author><Author ValidYN="Y"><LastName>Behar</LastName><ForeName>C</ForeName><Initials>C</Initials></Author><Author ValidYN="Y"><LastName>Lazennec</LastName><ForeName>J Y</ForeName><Initials>JY</Initials></Author><Author ValidYN="Y"><LastName>Kurys</LastName><ForeName>E</ForeName><Initials>E</Initials></Author><Author ValidYN="Y"><LastName>Arnulf</LastName><ForeName>I</ForeName><Initials>I</Initials></Author><Author ValidYN="Y"><LastName>Bonnet</LastName><ForeName>A M</ForeName><Initials>AM</Initials></Author><Author ValidYN="Y"><LastName>Agid</LastName><ForeName>Y</ForeName><Initials>Y</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D003160">Comparative Study</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType></PublicationTypeList><ArticleDate DateType="Electronic"><Year>2006</Year><Month>06</Month><Day>05</Day></ArticleDate></Article><MedlineJournalInfo><Country>England</Country><MedlineTA>J Neurol Neurosurg 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Nov;77(11):1205</RefSource><PMID Version="1">17043289</PMID></CommentsCorrections></CommentsCorrectionsList><MeshHeadingList><MeshHeading><DescriptorName UI="D000368" MajorTopicYN="N">Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001479" MajorTopicYN="N">Basal Ganglia</DescriptorName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004421" MajorTopicYN="N">Dystonia</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004576" MajorTopicYN="N">Electromyography</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D004594" MajorTopicYN="N">Electrophysiology</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008875" MajorTopicYN="N">Middle Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D018482" MajorTopicYN="N">Muscle, Skeletal</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D010300" MajorTopicYN="N">Parkinson Disease</DescriptorName><QualifierName UI="Q000150" MajorTopicYN="Y">complications</QualifierName><QualifierName UI="Q000503" MajorTopicYN="Y">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011187" MajorTopicYN="Y">Posture</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D011446" MajorTopicYN="N">Prospective Studies</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D016138" MajorTopicYN="N">Walking</DescriptorName></MeshHeading></MeshHeadingList><OtherID Source="NLM">PMC2077378</OtherID></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2006</Year><Month>6</Month><Day>7</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2006</Year><Month>10</Month><Day>28</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2006</Year><Month>6</Month><Day>7</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">16754693</ArticleId><ArticleId IdType="pii">jnnp.2006.087908</ArticleId><ArticleId IdType="doi">10.1136/jnnp.2006.087908</ArticleId><ArticleId IdType="pmc">PMC2077378</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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