ParkinsonFranceV1, Ncbi, Curation, bibRecord, 000E79

Camptocormia as a clinical manifestation of polymyositis/systemic sclerosis overlap myositis associated with anti-Ku.

Identifieur interne : 000E79 ( Ncbi/Curation ); précédent : 000E78; suivant : 000E80

Camptocormia as a clinical manifestation of polymyositis/systemic sclerosis overlap myositis associated with anti-Ku.

Auteurs : Thierry Zenone [France] ; Nathalie Streichenberger ; Marie Puget

Source :

Rheumatology international [ 1437-160X ] ; 2013.

RBID : pubmed:22453528

English descriptors

KwdEn :
- Aged, Antigens, Nuclear (immunology), Autoantibodies (blood), DNA-Binding Proteins (immunology), Humans, Ku Autoantigen, Male, Muscular Atrophy, Spinal (etiology), Polymyositis (complications), Polymyositis (pathology), Scleroderma, Systemic (complications), Scleroderma, Systemic (pathology), Spinal Curvatures (etiology).
MESH :
- chemical , blood : Autoantibodies.
- chemical , immunology : Antigens, Nuclear, DNA-Binding Proteins.
- complications : Polymyositis, Scleroderma, Systemic.
- etiology : Muscular Atrophy, Spinal, Spinal Curvatures.
- pathology : Polymyositis, Scleroderma, Systemic.
- Aged, Humans, Ku Autoantigen, Male.

Abstract

Camptocormia, an abnormal truncal flexion posture that occurs while walking or standing, is usually caused by various hypokinetic movement disorders, mainly Parkinson disease. We describe the case of a man with subacute onset of camptocormia. Quadriceps muscle biopsy showed significant rhabdomyolysis, few isolated inflammatory cells and mild expression of type I MHC in few fibers, a pattern usually found in immune-mediated necrotizing myopathies. Myositis was associated with Raynaud's phenomenon, mild sclerodactyly, and anti-Ku antibodies leading to the diagnosis of polymyositis/systemic sclerosis overlap myositis. The posture showed modest improvement in response to treatment.

DOI: 10.1007/s00296-012-2412-6
PubMed: 22453528

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Le document en format XML

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<term>Male</term>
<term>Muscular Atrophy, Spinal (etiology)</term>
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<term>Polymyositis (pathology)</term>
<term>Scleroderma, Systemic (complications)</term>
<term>Scleroderma, Systemic (pathology)</term>
<term>Spinal Curvatures (etiology)</term>
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<front><div type="abstract" xml:lang="en">Camptocormia, an abnormal truncal flexion posture that occurs while walking or standing, is usually caused by various hypokinetic movement disorders, mainly Parkinson disease. We describe the case of a man with subacute onset of camptocormia. Quadriceps muscle biopsy showed significant rhabdomyolysis, few isolated inflammatory cells and mild expression of type I MHC in few fibers, a pattern usually found in immune-mediated necrotizing myopathies. Myositis was associated with Raynaud's phenomenon, mild sclerodactyly, and anti-Ku antibodies leading to the diagnosis of polymyositis/systemic sclerosis overlap myositis. The posture showed modest improvement in response to treatment.</div>
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Camptocormia as a clinical manifestation of polymyositis/systemic sclerosis overlap myositis associated with anti-Ku.

Camptocormia as a clinical manifestation of polymyositis/systemic sclerosis overlap myositis associated with anti-Ku.

Source :

English descriptors

Abstract

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Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki