Chronic subthalamic nucleus stimulation and striatal D2 dopamine receptors in Parkinson's disease--A [(11)C]-raclopride PET study.
Identifieur interne : 000389 ( Ncbi/Curation ); précédent : 000388; suivant : 000390Chronic subthalamic nucleus stimulation and striatal D2 dopamine receptors in Parkinson's disease--A [(11)C]-raclopride PET study.
Auteurs : S. Thobois [France] ; V. Fraix ; M. Savasta ; N. Costes ; P. Pollak ; P. Mertens ; A. Koudsie ; D. Le Bars ; A L Benabid ; E. BroussolleSource :
- Journal of neurology [ 0340-5354 ] ; 2003.
English descriptors
- KwdEn :
- Adult, Aged, Carbon Radioisotopes, Dopamine (secretion), Dopamine Antagonists, Electric Stimulation Therapy, Female, Humans, Male, Middle Aged, Parkinson Disease (diagnostic imaging), Parkinson Disease (therapy), Raclopride, Receptors, Dopamine D2 (physiology), Subthalamic Nucleus (physiology), Tomography, Emission-Computed.
- MESH :
- chemical , physiology : Receptors, Dopamine D2.
- chemical , secretion : Dopamine.
- chemical : Carbon Radioisotopes, Dopamine Antagonists, Raclopride.
- diagnostic imaging : Parkinson Disease.
- physiology : Subthalamic Nucleus.
- therapy : Parkinson Disease.
- Adult, Aged, Electric Stimulation Therapy, Female, Humans, Male, Middle Aged, Tomography, Emission-Computed.
Abstract
Subthalamic nucleus (STN) stimulation mechanism of action remains a matter for debate. In animals, an increased striatal dopamine (DA) release due to STN stimulation has been reported.
DOI: 10.1007/s00415-003-0188-z
PubMed: 14586606
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pubmed:14586606Le document en format XML
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In animals, an increased striatal dopamine (DA) release due to STN stimulation has been reported.</div></front></TEI></record>Pour manipuler ce document sous Unix (Dilib)
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