The shadow of movement
Identifieur interne : 004F18 ( Main/Exploration ); précédent : 004F17; suivant : 004F19The shadow of movement
Auteurs : Pierre Rondot [France]Source :
- Journal of Neurology [ 0340-5354 ] ; 1991-12-01.
English descriptors
- KwdEn :
- Animals, Athetosis (physiopathology), Corpus Striatum (physiology), Decerebrate State (physiopathology), Dystonia (physiopathology), Electromyography, Globus Pallidus (physiology), Haplorhini, Humans, Motor Cortex (physiology), Movement (physiology), Muscle Contraction, Neurons (physiology), Parkinson Disease (physiopathology), Posture (physiology), Reflex (physiology), Reflex, Abnormal (physiology), Volition.
- MESH :
- physiology : Corpus Striatum, Globus Pallidus, Motor Cortex, Movement, Neurons, Posture, Reflex, Reflex, Abnormal.
- physiopathology : Athetosis, Decerebrate State, Dystonia, Parkinson Disease.
- Animals, Electromyography, Haplorhini, Humans, Muscle Contraction, Volition.
Abstract
Summary: Movement is preceded, accompanied and followed by reactions which give to the primary action its correct execution and ensure that the body's axis, together with the limbs, maintains the right balance. If these reactions are interfered with, incoordination of movement, lack of balance, hypertonia or dystonia may all appear. In the case of dystonia, postural mechanisms tend to become dominant and take over from the kinetic component of movement. In the upper limbs, the dystonic posture follows patterns analogous to those used by monkeys for postural purposes. Thus, while the initial mechanisms of movement represent highly sophisticated processes thoroughly adapted to living in an upright state, the reactions that go with the movement are more primitive and probably have a less helpful role.
Url:
DOI: 10.1007/BF00314646
Affiliations:
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Le document en format XML
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type="wicri:Area/Main/Exploration">004F18</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">The shadow of movement</title><author><name sortKey="Rondot, Pierre" sort="Rondot, Pierre" uniqKey="Rondot P" first="Pierre" last="Rondot">Pierre Rondot</name><affiliation wicri:level="3"><country xml:lang="fr">France</country><wicri:regionArea>Hôpital Sainte Anne, 1, Rue Cabanis, F-75014, Paris</wicri:regionArea><placeName><region type="region" nuts="2">Île-de-France</region><settlement type="city">Paris</settlement></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j">Journal of Neurology</title><title level="j" type="abbrev">J Neurol</title><idno type="ISSN">0340-5354</idno><idno type="eISSN">1432-1459</idno><imprint><publisher>Springer-Verlag</publisher><pubPlace>Berlin/Heidelberg</pubPlace><date type="published" when="1991-12-01">1991-12-01</date><biblScope unit="volume">238</biblScope><biblScope unit="issue">8</biblScope><biblScope unit="page" from="411">411</biblScope><biblScope unit="page" to="419">419</biblScope></imprint><idno type="ISSN">0340-5354</idno></series><idno type="istex">E2B66985388881DE5C142E74AE23D670D7309C0A</idno><idno type="DOI">10.1007/BF00314646</idno><idno type="ArticleID">BF00314646</idno><idno type="ArticleID">Art1</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0340-5354</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term><term>Athetosis (physiopathology)</term><term>Corpus Striatum (physiology)</term><term>Decerebrate State (physiopathology)</term><term>Dystonia (physiopathology)</term><term>Electromyography</term><term>Globus Pallidus (physiology)</term><term>Haplorhini</term><term>Humans</term><term>Motor Cortex (physiology)</term><term>Movement (physiology)</term><term>Muscle Contraction</term><term>Neurons (physiology)</term><term>Parkinson Disease (physiopathology)</term><term>Posture (physiology)</term><term>Reflex (physiology)</term><term>Reflex, Abnormal (physiology)</term><term>Volition</term></keywords><keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Corpus Striatum</term><term>Globus Pallidus</term><term>Motor Cortex</term><term>Movement</term><term>Neurons</term><term>Posture</term><term>Reflex</term><term>Reflex, Abnormal</term></keywords><keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Athetosis</term><term>Decerebrate State</term><term>Dystonia</term><term>Parkinson Disease</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Animals</term><term>Electromyography</term><term>Haplorhini</term><term>Humans</term><term>Muscle Contraction</term><term>Volition</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Summary: Movement is preceded, accompanied and followed by reactions which give to the primary action its correct execution and ensure that the body's axis, together with the limbs, maintains the right balance. If these reactions are interfered with, incoordination of movement, lack of balance, hypertonia or dystonia may all appear. In the case of dystonia, postural mechanisms tend to become dominant and take over from the kinetic component of movement. In the upper limbs, the dystonic posture follows patterns analogous to those used by monkeys for postural purposes. Thus, while the initial mechanisms of movement represent highly sophisticated processes thoroughly adapted to living in an upright state, the reactions that go with the movement are more primitive and probably have a less helpful role.</div></front></TEI><affiliations><list><country><li>France</li></country><region><li>Île-de-France</li></region><settlement><li>Paris</li></settlement></list><tree><country name="France"><region name="Île-de-France"><name sortKey="Rondot, Pierre" sort="Rondot, Pierre" uniqKey="Rondot P" first="Pierre" last="Rondot">Pierre Rondot</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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