Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.
Identifieur interne : 001856 ( Istex/Corpus ); précédent : 001855; suivant : 001857Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.
Auteurs : B. Brembilla-Perrot ; A. Terrier De La Chaise ; S. Briançon ; M. Takoordial ; C. Suty-Selton ; B. Thiel ; J L BruaSource :
- British Heart Journal [ 0007-0769 ] ; 1993-01.
Abstract
Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was < 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.
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DOI: 10.1136/hrt.69.1.20
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</title><author><name sortKey="Brembilla Perrot, B" sort="Brembilla Perrot, B" uniqKey="Brembilla Perrot B" first="B" last="Brembilla-Perrot">B. Brembilla-Perrot</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Terrier De La Chaise, A" sort="Terrier De La Chaise, A" uniqKey="Terrier De La Chaise A" first="A" last="Terrier De La Chaise">A. Terrier De La Chaise</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Briancon, S" sort="Briancon, S" uniqKey="Briancon S" first="S" last="Briançon">S. Briançon</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Takoordial, M" sort="Takoordial, M" uniqKey="Takoordial M" first="M" last="Takoordial">M. Takoordial</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Suty Selton, C" sort="Suty Selton, C" uniqKey="Suty Selton C" first="C" last="Suty-Selton">C. 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Brembilla-Perrot</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Terrier De La Chaise, A" sort="Terrier De La Chaise, A" uniqKey="Terrier De La Chaise A" first="A" last="Terrier De La Chaise">A. Terrier De La Chaise</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Briancon, S" sort="Briancon, S" uniqKey="Briancon S" first="S" last="Briançon">S. Briançon</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Takoordial, M" sort="Takoordial, M" uniqKey="Takoordial M" first="M" last="Takoordial">M. Takoordial</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Suty Selton, C" sort="Suty Selton, C" uniqKey="Suty Selton C" first="C" last="Suty-Selton">C. Suty-Selton</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Thiel, B" sort="Thiel, B" uniqKey="Thiel B" first="B" last="Thiel">B. Thiel</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author><author><name sortKey="Brua, J L" sort="Brua, J L" uniqKey="Brua J" first="J L" last="Brua">J L Brua</name><affiliation><mods:affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">British Heart Journal</title><title level="j" type="abbrev">Br Heart J</title><idno type="ISSN">0007-0769</idno><idno type="eISSN">1468-201X</idno><imprint><publisher>BMJ Publishing Group Ltd and British Cardiovascular Society</publisher><date type="published" when="1993-01">1993-01</date><biblScope unit="volume">69</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="20">20</biblScope></imprint><idno type="ISSN">0007-0769</idno></series><idno type="istex">2074659A4B20DA2950C01219A5254A25714D45E1</idno><idno type="DOI">10.1136/hrt.69.1.20</idno><idno type="href">heartjnl-69-20.pdf</idno><idno type="PMID">8457388</idno><idno type="local">heartjnl;69/1/20</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0007-0769</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was < 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.</div></front></TEI><istex><corpusName>bmj</corpusName><author><json:item><name>B Brembilla-Perrot</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>A Terrier de la Chaise</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>S Briançon</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>M Takoordial</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>C Suty-Selton</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>B Thiel</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item><json:item><name>J L Brua</name><affiliations><json:string>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</json:string></affiliations></json:item></author><language><json:string>eng</json:string></language><originalGenre><json:string>research-article</json:string></originalGenre><abstract>Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was > 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.</abstract><qualityIndicators><score>7.42</score><pdfVersion>1.3</pdfVersion><pdfPageSize>586.08 x 851.52 pts</pdfPageSize><refBibsNative>false</refBibsNative><keywordCount>0</keywordCount><abstractCharCount>1224</abstractCharCount><pdfWordCount>3188</pdfWordCount><pdfCharCount>21567</pdfCharCount><pdfPageCount>6</pdfPageCount><abstractWordCount>186</abstractWordCount></qualityIndicators><title>Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</title><pmid><json:string>8457388</json:string></pmid><refBibs><json:item><author><json:item><name>R Mahmud</name></json:item><json:item><name>S Denker</name></json:item><json:item><name>Mh Lehmann</name></json:item><json:item><name>P Tchou</name></json:item><json:item><name>J 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arrhythmias.</title><respStmt><resp>Références bibliographiques récupérées via GROBID</resp><name resp="ISTEX-API">ISTEX-API (INIST-CNRS)</name></respStmt></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>BMJ Publishing Group Ltd and British Cardiovascular Society</publisher><availability><p>BMJ</p></availability><date>1993-01-01</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</title><author xml:id="author-1"><persName><forename type="first">B</forename><surname>Brembilla-Perrot</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-2"><persName><forename type="first">A</forename><surname>Terrier de la Chaise</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-3"><persName><forename type="first">S</forename><surname>Briançon</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-4"><persName><forename type="first">M</forename><surname>Takoordial</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-5"><persName><forename type="first">C</forename><surname>Suty-Selton</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-6"><persName><forename type="first">B</forename><surname>Thiel</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author><author xml:id="author-7"><persName><forename type="first">J L</forename><surname>Brua</surname></persName><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation></author></analytic><monogr><title level="j">British Heart Journal</title><title level="j" type="abbrev">Br Heart J</title><idno type="pISSN">0007-0769</idno><idno type="eISSN">1468-201X</idno><imprint><publisher>BMJ Publishing Group Ltd and British Cardiovascular Society</publisher><date type="published" when="1993-01"></date><biblScope unit="volume">69</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="20">20</biblScope></imprint></monogr><idno type="istex">2074659A4B20DA2950C01219A5254A25714D45E1</idno><idno type="DOI">10.1136/hrt.69.1.20</idno><idno type="href">heartjnl-69-20.pdf</idno><idno type="PMID">8457388</idno><idno type="local">heartjnl;69/1/20</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1993-01-01</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was < 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.</p></abstract></profileDesc><revisionDesc><change when="1993-01-01">Created</change><change when="1993-01">Published</change><change xml:id="refBibs-istex" who="#ISTEX-API" when="2017-01-19">References added</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/2074659A4B20DA2950C01219A5254A25714D45E1/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="corpus bmj" wicri:toSee="no header"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="no"</istex:xmlDeclaration><istex:docType PUBLIC="-//NLM//DTD Journal Archiving and Interchange DTD v2.3 20070202//EN" URI="archivearticle.dtd" name="istex:docType"></istex:docType><istex:document><article article-type="research-article" xml:lang="EN"><front><journal-meta><journal-id journal-id-type="hwp">heartjnl</journal-id><journal-id journal-id-type="nlm-ta">Heart</journal-id><journal-title>British Heart Journal</journal-title><abbrev-journal-title abbrev-type="publisher">Br Heart J</abbrev-journal-title><issn pub-type="ppub">0007-0769</issn><issn pub-type="epub">1468-201X</issn><publisher><publisher-name>BMJ Publishing Group Ltd and British Cardiovascular Society</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">8457388</article-id><article-id pub-id-type="other">heartjnl;69/1/20</article-id><article-id pub-id-type="doi">10.1136/hrt.69.1.20</article-id><article-id pub-id-type="other">20</article-id><article-id pub-id-type="other">hrt.69.1.20</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group></article-categories><title-group><article-title>Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</article-title></title-group><contrib-group><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Brembilla-Perrot</surname><given-names>B</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Terrier de la Chaise</surname><given-names>A</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Briançon</surname><given-names>S</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Takoordial</surname><given-names>M</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Suty-Selton</surname><given-names>C</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Thiel</surname><given-names>B</given-names></name></contrib><contrib contrib-type="author" xlink:type="simple"><name name-style="western"><surname>Brua</surname><given-names>J L</given-names></name></contrib></contrib-group><aff>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</aff><pub-date pub-type="ppub"><month>1</month><year>1993</year></pub-date><pub-date pub-type="epub"><day>1</day><month>1</month><year>1993</year></pub-date><volume>69</volume><volume-id pub-id-type="other">69</volume-id><volume-id pub-id-type="other">69</volume-id><issue>1</issue><issue-id pub-id-type="other">heartjnl;69/1</issue-id><issue-id pub-id-type="other">1</issue-id><issue-id pub-id-type="other">69/1</issue-id><fpage>20</fpage><self-uri content-type="pdf" xlink:role="full-text" xlink:href="heartjnl-69-20.pdf"></self-uri><abstract><p>Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was < 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.</p></abstract></article-meta></front></article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Clinical significance of rapid ventricular tachycardia (> 270 beats per minute) provoked at programmed stimulation in patients without confirmed rapid ventricular arrhythmias.</title></titleInfo><name type="personal"><namePart type="given">B</namePart><namePart type="family">Brembilla-Perrot</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A</namePart><namePart type="family">Terrier de la Chaise</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">S</namePart><namePart type="family">Briançon</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M</namePart><namePart type="family">Takoordial</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">C</namePart><namePart type="family">Suty-Selton</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">B</namePart><namePart type="family">Thiel</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">J L</namePart><namePart type="family">Brua</namePart><affiliation>Department of Cardiology, Brabois University Hospital, Vandoeuvre Les Nancy, France.</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="research-article"></genre><originInfo><publisher>BMJ Publishing Group Ltd and British Cardiovascular Society</publisher><dateIssued encoding="w3cdtf">1993-01</dateIssued><dateCreated encoding="w3cdtf">1993-01-01</dateCreated><copyrightDate encoding="w3cdtf">1993</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Rapid uniform ventricular tachycardia (VT) (> 270 beats/min) or ventricular flutter induced during electrophysiological studies is thought not to be clinically significant in patients without cardiac arrest or documented rapid VT. The purpose of the study was to follow up 73 patients with inducible ventricular flutter but without confirmed rapid spontaneous VT. A long follow up (mean 3.5 years) identified two groups of patients. The first group had an excellent outcome and was characterised by a normal 24 hour Holter monitoring. In the second group, however, the risk of cardiac mortality was high (35%) and spontaneous VT was < 270 beats/min (26%) and was characterised by couplets or salvos of extrasystoles on Holter monitoring. In this group the history of syncope and decreased left ejection fraction increased the risk of mortality and VT. The presence of late potentials increased the risk of spontaneous VT. Electrophysiologically guided antiarrhythmic therapy reduced the risk of VT. Ventricular flutter was a non-specific finding in patients with normal Holter monitoring. In contrast, in patients with salvos of extrasystoles, ventricular flutter was associated with a high risk of cardiac mortality and VT.</abstract><relatedItem type="host"><titleInfo><title>British Heart Journal</title></titleInfo><titleInfo type="abbreviated"><title>Br Heart J</title></titleInfo><genre type="journal">journal</genre><identifier type="ISSN">0007-0769</identifier><identifier type="eISSN">1468-201X</identifier><identifier type="PublisherID-hwp">heartjnl</identifier><identifier type="PublisherID-nlm-ta">Heart</identifier><part><date>1993</date><detail type="volume"><caption>vol.</caption><number>69</number></detail><detail type="issue"><caption>no.</caption><number>1</number></detail><extent unit="pages"><start>20</start></extent></part></relatedItem><identifier type="istex">2074659A4B20DA2950C01219A5254A25714D45E1</identifier><identifier type="DOI">10.1136/hrt.69.1.20</identifier><identifier type="href">heartjnl-69-20.pdf</identifier><identifier type="PMID">8457388</identifier><identifier type="local">heartjnl;69/1/20</identifier><recordInfo><recordContentSource>BMJ</recordContentSource></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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