Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis
Identifieur interne : 002584 ( PascalFrancis/Corpus ); précédent : 002583; suivant : 002585Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis
Auteurs : Michael H. Barnett ; Paul R. Jarman ; Simon J. R. Heales ; Kailash P. BhatiaSource :
- Movement disorders [ 0885-3185 ] ; 2002.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise-induced dystonia. A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders.
Notice en format standard (ISO 2709)
Pour connaître la documentation sur le format Inist Standard.
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Format Inist (serveur)
NO : | PASCAL 03-0038728 INIST |
---|---|
ET : | Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis |
AU : | BARNETT (Michael H.); JARMAN (Paul R.); HEALES (Simon J. R.); BHATIA (Kailash P.) |
AF : | National Hospital for Neurology and Neurosurgery, Institute of Neurology, Queen Square/London/Royaume-Uni (1 aut., 2 aut., 4 aut.); Department of Clinical Biochemistry, Neurometabolic Unit, National Hospital, Queen Square/London/Royaume-Uni (3 aut.) |
DT : | Publication en série; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2002; Vol. 17; No. 6; Pp. 1386-1387; Bibl. 6 ref. |
LA : | Anglais |
EA : | Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise-induced dystonia. A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders. |
CC : | 002B17A01 |
FD : | Dystonie; Déclenchement; Exercice physique; Liquide céphalorachidien; Etude cas; Pathogénie; Homme; Enregistrement vidéo; Acide homovanillique; Acide hydroxyindole-acétique |
FG : | Muscle strié pathologie; Système nerveux pathologie; Trouble neurologique; Mouvement involontaire; Extrapyramidal syndrome |
ED : | Dystonia; Triggering; Physical exercise; Cerebrospinal fluid; Case study; Pathogenesis; Human; Video recording |
EG : | Striated muscle disease; Nervous system diseases; Neurological disorder; Involuntary movement; Extrapyramidal syndrome |
SD : | Distonía; Inducción; Ejercicio físico; Líquido cefalorraquídeo; Estudio caso; Patogenia; Hombre; Registro vídeo |
LO : | INIST-20953.354000105559450420 |
ID : | 03-0038728 |
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Pascal:03-0038728Le document en format XML
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<front><div type="abstract" xml:lang="en">Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise-induced dystonia. A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders.</div>
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<ET>Further case of paroxysmal exercise-induced dystonia and some insights into pathogenesis</ET>
<AU>BARNETT (Michael H.); JARMAN (Paul R.); HEALES (Simon J. R.); BHATIA (Kailash P.)</AU>
<AF>National Hospital for Neurology and Neurosurgery, Institute of Neurology, Queen Square/London/Royaume-Uni (1 aut., 2 aut., 4 aut.); Department of Clinical Biochemistry, Neurometabolic Unit, National Hospital, Queen Square/London/Royaume-Uni (3 aut.)</AF>
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<LA>Anglais</LA>
<EA>Cerebrospinal fluid (CSF) analysis of pterin and monamine metabolites was performed before and after an attack in a patient with paroxysmal exercise-induced dystonia. A twofold increase in CSF homovanillic acid and 5-hydroxyindoleacetic acid after an attack was measured. This finding lends support to the hypothesis that increased dopaminergic transmission contributes to the clinical features of the hyperkinetic movement disorders.</EA>
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