Neuropsychiatric interpretations of postencephalitic movement disorders
Identifieur interne : 002468 ( PascalFrancis/Corpus ); précédent : 002467; suivant : 002469Neuropsychiatric interpretations of postencephalitic movement disorders
Auteurs : Christopher D. WardSource :
- Movement disorders [ 0885-3185 ] ; 2003.
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- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
This study reviews the impact of encephalitis lethargica (EL) on concepts of behaviour and movement during the 1920s and 1930s. Clinicopathological correlations were imprecise but supported the role of subcortical structures in complex patterns of motor behaviour. This possibility challenged the widely assumed hegemony of the cerebral cortex. There was a perceived link between involuntary movements and reduced impulse control and also between parkinsonism and a defect in volition. Contemporary observers interpreted postencephalitic phenomena such as oculogyria in psychodynamic as well as in neurophysiological terms. EL also gave some support to the idea that neuroses such as obsessional neurosis and hysteria might have an organic basis. These speculations recently have acquired more credibility. The large amount of literature on EL and its sequelae could perhaps make further contributions to understanding the pathology of voluntary movement and action.
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Format Inist (serveur)
NO : | PASCAL 03-0379972 INIST |
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ET : | Neuropsychiatric interpretations of postencephalitic movement disorders |
AU : | WARD (Christopher D.) |
AF : | University of Nottingham Rehabilitation Research Unit, Derby City General Hospital/Derby/Royaume-Uni (1 aut.) |
DT : | Publication en série; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2003; Vol. 18; No. 6; Pp. 623-630; Bibl. 64 ref. |
LA : | Anglais |
EA : | This study reviews the impact of encephalitis lethargica (EL) on concepts of behaviour and movement during the 1920s and 1930s. Clinicopathological correlations were imprecise but supported the role of subcortical structures in complex patterns of motor behaviour. This possibility challenged the widely assumed hegemony of the cerebral cortex. There was a perceived link between involuntary movements and reduced impulse control and also between parkinsonism and a defect in volition. Contemporary observers interpreted postencephalitic phenomena such as oculogyria in psychodynamic as well as in neurophysiological terms. EL also gave some support to the idea that neuroses such as obsessional neurosis and hysteria might have an organic basis. These speculations recently have acquired more credibility. The large amount of literature on EL and its sequelae could perhaps make further contributions to understanding the pathology of voluntary movement and action. |
CC : | 002B17A02 |
FD : | Encéphalite; Mouvement involontaire; Parkinsonisme; Trouble comportement; Histoire; Epidémiologie; Homme; Siècle 20eme; Encéphalite léthargique |
FG : | Encéphale pathologie; Système nerveux pathologie; Système nerveux central pathologie; Trouble neurologique |
ED : | Encephalitis; Involuntary movement; Parkinsonism; Behavioral disorder; History; Epidemiology; Human; Century 20th |
EG : | Cerebral disorder; Nervous system diseases; Central nervous system disease; Neurological disorder |
SD : | Encefalitis; Movimiento involuntario; Parkinson síndrome; Trastorno conducta; Historia; Epidemiología; Hombre; Siglo 20 |
LO : | INIST-20953.354000119840860020 |
ID : | 03-0379972 |
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Pascal:03-0379972Le document en format XML
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<front><div type="abstract" xml:lang="en">This study reviews the impact of encephalitis lethargica (EL) on concepts of behaviour and movement during the 1920s and 1930s. Clinicopathological correlations were imprecise but supported the role of subcortical structures in complex patterns of motor behaviour. This possibility challenged the widely assumed hegemony of the cerebral cortex. There was a perceived link between involuntary movements and reduced impulse control and also between parkinsonism and a defect in volition. Contemporary observers interpreted postencephalitic phenomena such as oculogyria in psychodynamic as well as in neurophysiological terms. EL also gave some support to the idea that neuroses such as obsessional neurosis and hysteria might have an organic basis. These speculations recently have acquired more credibility. The large amount of literature on EL and its sequelae could perhaps make further contributions to understanding the pathology of voluntary movement and action.</div>
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<ET>Neuropsychiatric interpretations of postencephalitic movement disorders</ET>
<AU>WARD (Christopher D.)</AU>
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<DT>Publication en série; Niveau analytique</DT>
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