MovDisordV3, PascalFrancis, Corpus, bibRecord, 002467

Role of the somatosensory system in primary dystonia

Identifieur interne : 002467 ( PascalFrancis/Corpus ); précédent : 002466; suivant : 002468

Role of the somatosensory system in primary dystonia

Auteurs : Michele Tinazzi ; Tiziana Rosso ; Antonio Fiaschi

Source :

Movement disorders [ 0885-3185 ] ; 2003.

RBID : Pascal:03-0379973

Descripteurs français

Pascal (Inist)
- Dystonie, Primaire, Potentiel évoqué somatosensoriel, Cortex somatosensoriel, Noyau gris central, Article synthèse, Physiopathologie, Homme.

English descriptors

KwdEn :
- Basal ganglion, Dystonia, Human, Pathophysiology, Primary, Review, Somatosensory cortex, Somatosensory evoked potential.

Abstract

The pathophysiology of dystonia is still not fully understood, but it is widely held that a dysfunction of the corticostriatal-thalamocortical motor circuits plays a major role in the pathophysiology of this syndrome. Although the most dramatic symptoms in dystonia seem to be motor in nature, marked somatosensory perceptual deficits are also present in this disease. In addition, several lines of evidence, including neurophysiological, neuroimaging and experimental findings, suggest that both motor and somatosensory functions may be defective in dystonia. Consequently, abnormal processing of the somatosensory input in the central nervous system may lead to inefficient sensorimotor integration, thus contributing substantially to the generation of dystonic movements. Whether somatosensory abnormalities are capable of triggering dystonia is an issue warranting further study. Although it seems unlikely that abnormal somatosensory input is the only drive to dystonia, it might be more correlated to the development of focal hand than generalized dystonia because local somesthetic factors are more selectively involved in the former than in the latter where, instead it seems to be a widespread deficit in processing sensory stimuli of different modality. Because basal ganglia and motor areas are heavily connected not only with somatosensory areas, but also with visual and acoustic areas, it is possible that abnormalities of other sensory modalities, such as visual and acoustic, may also be implicated in the pathophysiology of more severe forms of primary dystonia. Further studies have to be addressed to the assessment of the role of sensory modalities and their interaction on the pathophysiology of different forms of primary dystonia.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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Format Inist (serveur)

NO :	PASCAL 03-0379973 INIST
ET :	Role of the somatosensory system in primary dystonia
AU :	TINAZZI (Michele); ROSSO (Tiziana); FIASCHI (Antonio)
AF :	Dipartimento di Scienze Neurologiche e delta Visione, Sezione di Neurologia Riabilitativa/Verona/Italie (1 aut., 2 aut., 3 aut.)
DT :	Publication en série; Niveau analytique
SO :	Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2003; Vol. 18; No. 6; Pp. 605-622; Bibl. 116 ref.
LA :	Anglais
EA :	The pathophysiology of dystonia is still not fully understood, but it is widely held that a dysfunction of the corticostriatal-thalamocortical motor circuits plays a major role in the pathophysiology of this syndrome. Although the most dramatic symptoms in dystonia seem to be motor in nature, marked somatosensory perceptual deficits are also present in this disease. In addition, several lines of evidence, including neurophysiological, neuroimaging and experimental findings, suggest that both motor and somatosensory functions may be defective in dystonia. Consequently, abnormal processing of the somatosensory input in the central nervous system may lead to inefficient sensorimotor integration, thus contributing substantially to the generation of dystonic movements. Whether somatosensory abnormalities are capable of triggering dystonia is an issue warranting further study. Although it seems unlikely that abnormal somatosensory input is the only drive to dystonia, it might be more correlated to the development of focal hand than generalized dystonia because local somesthetic factors are more selectively involved in the former than in the latter where, instead it seems to be a widespread deficit in processing sensory stimuli of different modality. Because basal ganglia and motor areas are heavily connected not only with somatosensory areas, but also with visual and acoustic areas, it is possible that abnormalities of other sensory modalities, such as visual and acoustic, may also be implicated in the pathophysiology of more severe forms of primary dystonia. Further studies have to be addressed to the assessment of the role of sensory modalities and their interaction on the pathophysiology of different forms of primary dystonia.
CC :	002B17A01
FD :	Dystonie; Primaire; Potentiel évoqué somatosensoriel; Cortex somatosensoriel; Noyau gris central; Article synthèse; Physiopathologie; Homme
FG :	Muscle strié pathologie; Système nerveux pathologie; Trouble neurologique; Mouvement involontaire; Extrapyramidal syndrome; Electrodiagnostic; Encéphale
ED :	Dystonia; Primary; Somatosensory evoked potential; Somatosensory cortex; Basal ganglion; Review; Pathophysiology; Human
EG :	Striated muscle disease; Nervous system diseases; Neurological disorder; Involuntary movement; Extrapyramidal syndrome; Electrodiagnosis; Brain (vertebrata)
SD :	Distonía; Primario; Potencial evocado somatosensorial; Corteza somatosensorial; Núcleo basal; Artículo síntesis; Fisiopatología; Hombre
LO :	INIST-20953.354000119840860010
ID :	03-0379973

Links to Exploration step

Pascal:03-0379973

Le document en format XML

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<server><NO>PASCAL 03-0379973 INIST</NO>
<ET>Role of the somatosensory system in primary dystonia</ET>
<AU>TINAZZI (Michele); ROSSO (Tiziana); FIASCHI (Antonio)</AU>
<AF>Dipartimento di Scienze Neurologiche e delta Visione, Sezione di Neurologia Riabilitativa/Verona/Italie (1 aut., 2 aut., 3 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2003; Vol. 18; No. 6; Pp. 605-622; Bibl. 116 ref.</SO>
<LA>Anglais</LA>
<EA>The pathophysiology of dystonia is still not fully understood, but it is widely held that a dysfunction of the corticostriatal-thalamocortical motor circuits plays a major role in the pathophysiology of this syndrome. Although the most dramatic symptoms in dystonia seem to be motor in nature, marked somatosensory perceptual deficits are also present in this disease. In addition, several lines of evidence, including neurophysiological, neuroimaging and experimental findings, suggest that both motor and somatosensory functions may be defective in dystonia. Consequently, abnormal processing of the somatosensory input in the central nervous system may lead to inefficient sensorimotor integration, thus contributing substantially to the generation of dystonic movements. Whether somatosensory abnormalities are capable of triggering dystonia is an issue warranting further study. Although it seems unlikely that abnormal somatosensory input is the only drive to dystonia, it might be more correlated to the development of focal hand than generalized dystonia because local somesthetic factors are more selectively involved in the former than in the latter where, instead it seems to be a widespread deficit in processing sensory stimuli of different modality. Because basal ganglia and motor areas are heavily connected not only with somatosensory areas, but also with visual and acoustic areas, it is possible that abnormalities of other sensory modalities, such as visual and acoustic, may also be implicated in the pathophysiology of more severe forms of primary dystonia. Further studies have to be addressed to the assessment of the role of sensory modalities and their interaction on the pathophysiology of different forms of primary dystonia.</EA>
<CC>002B17A01</CC>
<FD>Dystonie; Primaire; Potentiel évoqué somatosensoriel; Cortex somatosensoriel; Noyau gris central; Article synthèse; Physiopathologie; Homme</FD>
<FG>Muscle strié pathologie; Système nerveux pathologie; Trouble neurologique; Mouvement involontaire; Extrapyramidal syndrome; Electrodiagnostic; Encéphale</FG>
<ED>Dystonia; Primary; Somatosensory evoked potential; Somatosensory cortex; Basal ganglion; Review; Pathophysiology; Human</ED>
<EG>Striated muscle disease; Nervous system diseases; Neurological disorder; Involuntary movement; Extrapyramidal syndrome; Electrodiagnosis; Brain (vertebrata)</EG>
<SD>Distonía; Primario; Potencial evocado somatosensorial; Corteza somatosensorial; Núcleo basal; Artículo síntesis; Fisiopatología; Hombre</SD>
<LO>INIST-20953.354000119840860010</LO>
<ID>03-0379973</ID>
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	Movement Disorders (revue)
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Movement Disorders (revue)

Role of the somatosensory system in primary dystonia

Role of the somatosensory system in primary dystonia

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