MovDisordV3, PascalFrancis, Corpus, bibRecord, 001364

The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy

Identifieur interne : 001364 ( PascalFrancis/Corpus ); précédent : 001363; suivant : 001365

The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy

Auteurs : Bart P. C. Van De Warrenburg ; Caria Cordivari ; Aisling M. Ryan ; Rahul Phadke ; Janice L. Holton ; Kailash P. Bhatia ; Mike G. Hanna ; Niall P. Quinn

Source :

Movement disorders [ 0885-3185 ] ; 2007.

RBID : Pascal:08-0147019

Descripteurs français

Pascal (Inist)
- Maladie de Parkinson, Atrophie multisystématisée, Myopathie, Pathologie du système nerveux, Tête.

English descriptors

KwdEn :
- Head, Multiple system atrophy, Myopathy, Nervous system diseases, Parkinson disease.

Abstract

We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

A01	`01`	`1`		`@0 0885-3185`
A03		`1`		`@0 Mov. disord.`
A05				`@2 22`
A06				`@2 16`
A08	`01`	`1`	`ENG`	`@1 The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy`
A11	`01`	`1`		`@1 VAN DE WARRENBURG (Bart P. C.)`
A11	`02`	`1`		`@1 CORDIVARI (Caria)`
A11	`03`	`1`		`@1 RYAN (Aisling M.)`
A11	`04`	`1`		`@1 PHADKE (Rahul)`
A11	`05`	`1`		`@1 HOLTON (Janice L.)`
A11	`06`	`1`		`@1 BHATIA (Kailash P.)`
A11	`07`	`1`		`@1 HANNA (Mike G.)`
A11	`08`	`1`		`@1 QUINN (Niall P.)`
A14	`01`			`@1 Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology @2 London @3 GBR @Z 1 aut. @Z 6 aut. @Z 8 aut.`
A14	`02`			`@1 Department of Neurology, Radboud University Nijmegen Medical Centre @2 Nijmegen @3 NLD @Z 1 aut.`
A14	`03`			`@1 Department of Neurophysiology, National Hospital of Neurology and Neurosurgery @2 London @3 GBR @Z 2 aut.`
A14	`04`			`@1 Centre for Neuromuscular Disease, National Hospital of Neurology and Neurosurgery @2 London @3 GBR @Z 3 aut.`
A14	`05`			`@1 Division of Neuropathology, Institute of Neurology @2 London @3 GBR @Z 4 aut. @Z 5 aut.`
A14	`06`			`@1 Department of Molecular Neuroscience, Institute of Neurology @2 London @3 GBR @Z 7 aut.`
A20				`@1 2325-2331`
A21				`@1 2007`
A23	`01`			`@0 ENG`
A43	`01`			`@1 INIST @2 20953 @5 354000162715700040`
A44				`@0 0000 @1 © 2008 INIST-CNRS. All rights reserved.`
A45				`@0 13 ref.`
A47	`01`	`1`		`@0 08-0147019`
A60				`@1 P`
A61				`@0 A`
A64	`01`	`1`		`@0 Movement disorders`
A66	`01`			`@0 USA`
C01	`01`		`ENG`	@0 We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.
C02	`01`	`X`		`@0 002B17`
C03	`01`	`X`	`FRE`	`@0 Maladie de Parkinson @2 NM @5 01`
C03	`01`	`X`	`ENG`	`@0 Parkinson disease @2 NM @5 01`
C03	`01`	`X`	`SPA`	`@0 Parkinson enfermedad @2 NM @5 01`
C03	`02`	`X`	`FRE`	`@0 Atrophie multisystématisée @2 NM @5 02`
C03	`02`	`X`	`ENG`	`@0 Multiple system atrophy @2 NM @5 02`
C03	`02`	`X`	`SPA`	`@0 Atrofia multisistematizada @2 NM @5 02`
C03	`03`	`X`	`FRE`	`@0 Myopathie @5 03`
C03	`03`	`X`	`ENG`	`@0 Myopathy @5 03`
C03	`03`	`X`	`SPA`	`@0 Miopatía @5 03`
C03	`04`	`X`	`FRE`	`@0 Pathologie du système nerveux @5 04`
C03	`04`	`X`	`ENG`	`@0 Nervous system diseases @5 04`
C03	`04`	`X`	`SPA`	`@0 Sistema nervioso patología @5 04`
C03	`05`	`X`	`FRE`	`@0 Tête @5 09`
C03	`05`	`X`	`ENG`	`@0 Head @5 09`
C03	`05`	`X`	`SPA`	`@0 Cabeza @5 09`
C07	`01`	`X`	`FRE`	`@0 Pathologie de l'encéphale @5 37`
C07	`01`	`X`	`ENG`	`@0 Cerebral disorder @5 37`
C07	`01`	`X`	`SPA`	`@0 Encéfalo patología @5 37`
C07	`02`	`X`	`FRE`	`@0 Syndrome extrapyramidal @5 38`
C07	`02`	`X`	`ENG`	`@0 Extrapyramidal syndrome @5 38`
C07	`02`	`X`	`SPA`	`@0 Extrapiramidal síndrome @5 38`
C07	`03`	`X`	`FRE`	`@0 Maladie dégénérative @5 39`
C07	`03`	`X`	`ENG`	`@0 Degenerative disease @5 39`
C07	`03`	`X`	`SPA`	`@0 Enfermedad degenerativa @5 39`
C07	`04`	`X`	`FRE`	`@0 Pathologie du système nerveux central @5 40`
C07	`04`	`X`	`ENG`	`@0 Central nervous system disease @5 40`
C07	`04`	`X`	`SPA`	`@0 Sistema nervosio central patología @5 40`
N21				`@1 091`
N44	`01`			`@1 OTO`
N82				`@1 OTO`

Format Inist (serveur)

NO :	PASCAL 08-0147019 INIST
ET :	The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy
AU :	VAN DE WARRENBURG (Bart P. C.); CORDIVARI (Caria); RYAN (Aisling M.); PHADKE (Rahul); HOLTON (Janice L.); BHATIA (Kailash P.); HANNA (Mike G.); QUINN (Niall P.)
AF :	Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology/London/Royaume-Uni (1 aut., 6 aut., 8 aut.); Department of Neurology, Radboud University Nijmegen Medical Centre/Nijmegen/Pays-Bas (1 aut.); Department of Neurophysiology, National Hospital of Neurology and Neurosurgery/London/Royaume-Uni (2 aut.); Centre for Neuromuscular Disease, National Hospital of Neurology and Neurosurgery/London/Royaume-Uni (3 aut.); Division of Neuropathology, Institute of Neurology/London/Royaume-Uni (4 aut., 5 aut.); Department of Molecular Neuroscience, Institute of Neurology/London/Royaume-Uni (7 aut.)
DT :	Publication en série; Niveau analytique
SO :	Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2007; Vol. 22; No. 16; Pp. 2325-2331; Bibl. 13 ref.
LA :	Anglais
EA :	We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.
CC :	002B17
FD :	Maladie de Parkinson; Atrophie multisystématisée; Myopathie; Pathologie du système nerveux; Tête
FG :	Pathologie de l'encéphale; Syndrome extrapyramidal; Maladie dégénérative; Pathologie du système nerveux central
ED :	Parkinson disease; Multiple system atrophy; Myopathy; Nervous system diseases; Head
EG :	Cerebral disorder; Extrapyramidal syndrome; Degenerative disease; Central nervous system disease
SD :	Parkinson enfermedad; Atrofia multisistematizada; Miopatía; Sistema nervioso patología; Cabeza
LO :	INIST-20953.354000162715700040
ID :	08-0147019

Links to Exploration step

Pascal:08-0147019

Le document en format XML

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<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy</title>
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<front><div type="abstract" xml:lang="en">We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.</div>
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<fA11 i1="02" i2="1"><s1>CORDIVARI (Caria)</s1>
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<fA14 i1="01"><s1>Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology</s1>
<s2>London</s2>
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<sZ>1 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>8 aut.</sZ>
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<fA14 i1="02"><s1>Department of Neurology, Radboud University Nijmegen Medical Centre</s1>
<s2>Nijmegen</s2>
<s3>NLD</s3>
<sZ>1 aut.</sZ>
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<fA14 i1="03"><s1>Department of Neurophysiology, National Hospital of Neurology and Neurosurgery</s1>
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<fA14 i1="04"><s1>Centre for Neuromuscular Disease, National Hospital of Neurology and Neurosurgery</s1>
<s2>London</s2>
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<fA14 i1="05"><s1>Division of Neuropathology, Institute of Neurology</s1>
<s2>London</s2>
<s3>GBR</s3>
<sZ>4 aut.</sZ>
<sZ>5 aut.</sZ>
</fA14>
<fA14 i1="06"><s1>Department of Molecular Neuroscience, Institute of Neurology</s1>
<s2>London</s2>
<s3>GBR</s3>
<sZ>7 aut.</sZ>
</fA14>
<fA20><s1>2325-2331</s1>
</fA20>
<fA21><s1>2007</s1>
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<fC01 i1="01" l="ENG"><s0>We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.</s0>
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<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="SPA"><s0>Atrofia multisistematizada</s0>
<s2>NM</s2>
<s5>02</s5>
</fC03>
<fC03 i1="03" i2="X" l="FRE"><s0>Myopathie</s0>
<s5>03</s5>
</fC03>
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<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA"><s0>Miopatía</s0>
<s5>03</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE"><s0>Pathologie du   système nerveux</s0>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG"><s0>Nervous system diseases</s0>
<s5>04</s5>
</fC03>
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<s5>04</s5>
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<s5>09</s5>
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<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG"><s0>Cerebral disorder</s0>
<s5>37</s5>
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<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE"><s0>Syndrome extrapyramidal</s0>
<s5>38</s5>
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<s5>38</s5>
</fC07>
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<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Maladie dégénérative</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Degenerative disease</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Enfermedad degenerativa</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Pathologie du   système nerveux central</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Central nervous system disease</s0>
<s5>40</s5>
</fC07>
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<s5>40</s5>
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<ET>The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy</ET>
<AU>VAN DE WARRENBURG (Bart P. C.); CORDIVARI (Caria); RYAN (Aisling M.); PHADKE (Rahul); HOLTON (Janice L.); BHATIA (Kailash P.); HANNA (Mike G.); QUINN (Niall P.)</AU>
<AF>Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology/London/Royaume-Uni (1 aut., 6 aut., 8 aut.); Department of Neurology, Radboud University Nijmegen Medical Centre/Nijmegen/Pays-Bas (1 aut.); Department of Neurophysiology, National Hospital of Neurology and Neurosurgery/London/Royaume-Uni (2 aut.); Centre for Neuromuscular Disease, National Hospital of Neurology and Neurosurgery/London/Royaume-Uni (3 aut.); Division of Neuropathology, Institute of Neurology/London/Royaume-Uni (4 aut., 5 aut.); Department of Molecular Neuroscience, Institute of Neurology/London/Royaume-Uni (7 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2007; Vol. 22; No. 16; Pp. 2325-2331; Bibl. 13 ref.</SO>
<LA>Anglais</LA>
<EA>We sought to explore the phenomenon of disproportionate antecollis in multiple system atrophy (MSA) and Parkinson's disease (PD). The etiology is much debated and the main issue is whether it represents a primary myopathy or is secondary to the underlying motor disorder. The clinical, electrophysiological, and biopsy data of MSA or PD patients with antecollis were reviewed. We reviewed 16 patients (7 MSA and 9 PD) who developed antecollis during the course of their disease. The interval between onset of motor symptoms and of antecollis was shorter in the MSA group (4.6 ± 1.7 years vs. 10.5 ± 7.0 years). In 6 patients, the antecollis developed subacutely, and in 2 the abnormal neck flexion was initially an off-period phenomenon. Two additional patients also showed some dopa-responsiveness. Clinically, the antecollis was characterized by a forward flexion and anterior shift of the neck, with prominent cervical paraspinal and levator scapulae muscles, usually without weakness of residual neck extension. Electromyography of cervical paraspinal muscles showed mixed myopathic, normal, and neurogenic units, without early recruitment. Cervical paraspinal muscle biopsy in 2 patients disclosed fibrosis and nonspecific myopathic changes. We suggest that, in the context of MSA or PD, the initiating event in antecollis could be a disproportionately increased tone in anterior neck muscles that leads to secondary fibrotic and myopathic changes. However, a primary but yet unexplained neck extensor myopathy still remains the alternative possibility and longitudinal studies are necessary to settle this issue.</EA>
<CC>002B17</CC>
<FD>Maladie de Parkinson; Atrophie multisystématisée; Myopathie; Pathologie du   système nerveux; Tête</FD>
<FG>Pathologie de l'encéphale; Syndrome extrapyramidal; Maladie dégénérative; Pathologie du   système nerveux central</FG>
<ED>Parkinson disease; Multiple system atrophy; Myopathy; Nervous system diseases; Head</ED>
<EG>Cerebral disorder; Extrapyramidal syndrome; Degenerative disease; Central nervous system disease</EG>
<SD>Parkinson enfermedad; Atrofia multisistematizada; Miopatía; Sistema nervioso patología; Cabeza</SD>
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	Movement Disorders (revue)
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Movement Disorders (revue)

The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy

The Phenomenon of Disproportionate Antecollis in Parkinson's Disease and Multiple System Atrophy

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