Atypical Parkinsonism combining α-synuclein inclusions and polyglucosan body disease
Identifieur interne : 001F26 ( PascalFrancis/Checkpoint ); précédent : 001F25; suivant : 001F27Atypical Parkinsonism combining α-synuclein inclusions and polyglucosan body disease
Auteurs : Elsa Krim [France] ; Anne Vital [France] ; Frederic Macia [France] ; Farid Yekhlef [France] ; Francois Tison [France]Source :
- Movement disorders [ 0885-3185 ] ; 2005.
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- Pascal (Inist)
English descriptors
Abstract
Adult polyglucosan body disease (APGBD) is a rare disorder affecting the central and peripheral nervous systems and in which parkinsonism is unusual. A 71-year-old man presented levodopa-unresponsive parkinsonism with urinary incontinence and recurrent syncopes of 6 years standing masquerading as atypical parkinsonism of the multiple system atrophy (MSA-P) type. Brain histopathology demonstrated massive accumulation of polyglucosan bodies particularly in the putamen. In addition, there were dense α-synuclein-positive cytoplasmic oligodendroglial inclusions in the pons and in the middle cerebellar peduncle. These inclusions may be either due to the chance association of MSA-P with APGBD, or pathologically related to APGBD.
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Pascal:05-0151000Le document en format XML
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