Atypical Parkinsonism combining α-synuclein inclusions and polyglucosan body disease
Identifieur interne : 001F60 ( PascalFrancis/Corpus ); précédent : 001F59; suivant : 001F61Atypical Parkinsonism combining α-synuclein inclusions and polyglucosan body disease
Auteurs : Elsa Krim ; Anne Vital ; Frederic Macia ; Farid Yekhlef ; Francois TisonSource :
- Movement disorders [ 0885-3185 ] ; 2005.
Descripteurs français
- Pascal (Inist)
English descriptors
Abstract
Adult polyglucosan body disease (APGBD) is a rare disorder affecting the central and peripheral nervous systems and in which parkinsonism is unusual. A 71-year-old man presented levodopa-unresponsive parkinsonism with urinary incontinence and recurrent syncopes of 6 years standing masquerading as atypical parkinsonism of the multiple system atrophy (MSA-P) type. Brain histopathology demonstrated massive accumulation of polyglucosan bodies particularly in the putamen. In addition, there were dense α-synuclein-positive cytoplasmic oligodendroglial inclusions in the pons and in the middle cerebellar peduncle. These inclusions may be either due to the chance association of MSA-P with APGBD, or pathologically related to APGBD.
Notice en format standard (ISO 2709)
Pour connaître la documentation sur le format Inist Standard.
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Format Inist (serveur)
NO : | PASCAL 05-0151000 INIST |
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ET : | Atypical Parkinsonism combining α-synuclein inclusions and polyglucosan body disease |
AU : | KRIM (Elsa); VITAL (Anne); MACIA (Frederic); YEKHLEF (Farid); TISON (Francois) |
AF : | Service de Neurologie, Centre Hospitalier Universitaire de Bordeaux/France (1 aut., 3 aut., 4 aut., 5 aut.); Service d'Anatomopathologie, Centre Hospitalier Universitaire de Bordeaux/France (2 aut.) |
DT : | Publication en série; Courte communication, note brève; Niveau analytique |
SO : | Movement disorders; ISSN 0885-3185; Etats-Unis; Da. 2005; Vol. 20; No. 2; Pp. 200-204; Bibl. 36 ref. |
LA : | Anglais |
EA : | Adult polyglucosan body disease (APGBD) is a rare disorder affecting the central and peripheral nervous systems and in which parkinsonism is unusual. A 71-year-old man presented levodopa-unresponsive parkinsonism with urinary incontinence and recurrent syncopes of 6 years standing masquerading as atypical parkinsonism of the multiple system atrophy (MSA-P) type. Brain histopathology demonstrated massive accumulation of polyglucosan bodies particularly in the putamen. In addition, there were dense α-synuclein-positive cytoplasmic oligodendroglial inclusions in the pons and in the middle cerebellar peduncle. These inclusions may be either due to the chance association of MSA-P with APGBD, or pathologically related to APGBD. |
CC : | 002B17; 002B17G; 002B17A03 |
FD : | Système nerveux pathologie; Corps inclusion; Parkinsonisme |
ED : | Nervous system diseases; Inclusion body; Parkinsonism |
SD : | Sistema nervioso patología; Cuerpo inclusión; Parkinson síndrome |
LO : | INIST-20953.354000126378730090 |
ID : | 05-0151000 |
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Pascal:05-0151000Le document en format XML
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