Reversible chorea in primary antiphospholipid syndrome.
Identifieur interne : 004E82 ( Ncbi/Curation ); précédent : 004E81; suivant : 004E83Reversible chorea in primary antiphospholipid syndrome.
Auteurs : J. Sundén-Cullberg [Suède] ; J. Tedroff ; S M AquiloniusSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 1998.
English descriptors
- KwdEn :
- MESH :
- complications : Antiphospholipid Syndrome.
- etiology : Chorea.
- metabolism : Antiphospholipid Syndrome, Brain, Chorea, Corpus Striatum.
- Acute Disease, Adult, Case-Control Studies, Female, Humans, Longitudinal Studies, Tomography, Emission-Computed.
Abstract
A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.
DOI: 10.1002/mds.870130127
PubMed: 9452340
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pubmed:9452340Le document en format XML
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<author><name sortKey="Sunden Cullberg, J" sort="Sunden Cullberg, J" uniqKey="Sunden Cullberg J" first="J" last="Sundén-Cullberg">J. Sundén-Cullberg</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, University Hospital, Uppsala, Sweden.</nlm:affiliation>
<country xml:lang="fr">Suède</country>
<wicri:regionArea>Department of Neurology, University Hospital, Uppsala</wicri:regionArea>
<wicri:noRegion>Uppsala</wicri:noRegion>
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<author><name sortKey="Tedroff, J" sort="Tedroff, J" uniqKey="Tedroff J" first="J" last="Tedroff">J. Tedroff</name>
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<author><name sortKey="Aquilonius, S M" sort="Aquilonius, S M" uniqKey="Aquilonius S" first="S M" last="Aquilonius">S M Aquilonius</name>
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<sourceDesc><biblStruct><analytic><title xml:lang="en">Reversible chorea in primary antiphospholipid syndrome.</title>
<author><name sortKey="Sunden Cullberg, J" sort="Sunden Cullberg, J" uniqKey="Sunden Cullberg J" first="J" last="Sundén-Cullberg">J. Sundén-Cullberg</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, University Hospital, Uppsala, Sweden.</nlm:affiliation>
<country xml:lang="fr">Suède</country>
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<author><name sortKey="Tedroff, J" sort="Tedroff, J" uniqKey="Tedroff J" first="J" last="Tedroff">J. Tedroff</name>
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<series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title>
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<imprint><date when="1998" type="published">1998</date>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Acute Disease</term>
<term>Adult</term>
<term>Antiphospholipid Syndrome (complications)</term>
<term>Antiphospholipid Syndrome (metabolism)</term>
<term>Brain (metabolism)</term>
<term>Case-Control Studies</term>
<term>Chorea (etiology)</term>
<term>Chorea (metabolism)</term>
<term>Corpus Striatum (metabolism)</term>
<term>Female</term>
<term>Humans</term>
<term>Longitudinal Studies</term>
<term>Tomography, Emission-Computed</term>
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<keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Antiphospholipid Syndrome</term>
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<keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Chorea</term>
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<keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Antiphospholipid Syndrome</term>
<term>Brain</term>
<term>Chorea</term>
<term>Corpus Striatum</term>
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<keywords scheme="MESH" xml:lang="en"><term>Acute Disease</term>
<term>Adult</term>
<term>Case-Control Studies</term>
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<front><div type="abstract" xml:lang="en">A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.</div>
</front>
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