Reversible chorea in primary antiphospholipid syndrome.
Identifieur interne : 004342 ( PubMed/Checkpoint ); précédent : 004341; suivant : 004343Reversible chorea in primary antiphospholipid syndrome.
Auteurs : J. Sundén-Cullberg [Suède] ; J. Tedroff ; S M AquiloniusSource :
- Movement disorders : official journal of the Movement Disorder Society [ 0885-3185 ] ; 1998.
English descriptors
- KwdEn :
- MESH :
- complications : Antiphospholipid Syndrome.
- etiology : Chorea.
- metabolism : Antiphospholipid Syndrome, Brain, Chorea, Corpus Striatum.
- Acute Disease, Adult, Case-Control Studies, Female, Humans, Longitudinal Studies, Tomography, Emission-Computed.
Abstract
A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.
DOI: 10.1002/mds.870130127
PubMed: 9452340
Affiliations:
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Tedroff</name></author><author><name sortKey="Aquilonius, S M" sort="Aquilonius, S M" uniqKey="Aquilonius S" first="S M" last="Aquilonius">S M Aquilonius</name></author></titleStmt><publicationStmt><idno type="wicri:source">PubMed</idno><date when="1998">1998</date><idno type="RBID">pubmed:9452340</idno><idno type="pmid">9452340</idno><idno type="doi">10.1002/mds.870130127</idno><idno type="wicri:Area/PubMed/Corpus">004503</idno><idno type="wicri:Area/PubMed/Curation">004503</idno><idno type="wicri:Area/PubMed/Checkpoint">004342</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en">Reversible chorea in primary antiphospholipid syndrome.</title><author><name sortKey="Sunden Cullberg, J" sort="Sunden Cullberg, J" uniqKey="Sunden Cullberg J" first="J" last="Sundén-Cullberg">J. Sundén-Cullberg</name><affiliation wicri:level="1"><nlm:affiliation>Department of Neurology, University Hospital, Uppsala, Sweden.</nlm:affiliation><country xml:lang="fr">Suède</country><wicri:regionArea>Department of Neurology, University Hospital, Uppsala</wicri:regionArea><wicri:noRegion>Uppsala</wicri:noRegion></affiliation></author><author><name sortKey="Tedroff, J" sort="Tedroff, J" uniqKey="Tedroff J" first="J" last="Tedroff">J. Tedroff</name></author><author><name sortKey="Aquilonius, S M" sort="Aquilonius, S M" uniqKey="Aquilonius S" first="S M" last="Aquilonius">S M Aquilonius</name></author></analytic><series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title><idno type="ISSN">0885-3185</idno><imprint><date when="1998" type="published">1998</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Acute Disease</term><term>Adult</term><term>Antiphospholipid Syndrome (complications)</term><term>Antiphospholipid Syndrome (metabolism)</term><term>Brain (metabolism)</term><term>Case-Control Studies</term><term>Chorea (etiology)</term><term>Chorea (metabolism)</term><term>Corpus Striatum (metabolism)</term><term>Female</term><term>Humans</term><term>Longitudinal Studies</term><term>Tomography, Emission-Computed</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Antiphospholipid Syndrome</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Chorea</term></keywords><keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Antiphospholipid Syndrome</term><term>Brain</term><term>Chorea</term><term>Corpus Striatum</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Acute Disease</term><term>Adult</term><term>Case-Control Studies</term><term>Female</term><term>Humans</term><term>Longitudinal Studies</term><term>Tomography, Emission-Computed</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.</div></front></TEI><pubmed><MedlineCitation Owner="NLM" Status="MEDLINE"><PMID Version="1">9452340</PMID><DateCreated><Year>1998</Year><Month>03</Month><Day>26</Day></DateCreated><DateCompleted><Year>1998</Year><Month>03</Month><Day>26</Day></DateCompleted><DateRevised><Year>2004</Year><Month>11</Month><Day>17</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0885-3185</ISSN><JournalIssue CitedMedium="Print"><Volume>13</Volume><Issue>1</Issue><PubDate><Year>1998</Year><Month>Jan</Month></PubDate></JournalIssue><Title>Movement disorders : official journal of the Movement Disorder Society</Title><ISOAbbreviation>Mov. Disord.</ISOAbbreviation></Journal><ArticleTitle>Reversible chorea in primary antiphospholipid syndrome.</ArticleTitle><Pagination><MedlinePgn>147-9</MedlinePgn></Pagination><Abstract><AbstractText>A 20-year-old woman with acute chorea induced by primary antiphospholipid syndrome was studied by using fluorodeoxyglucose and positron emission tomography (PET). PET sessions were conducted during an episode of severe chorea and after recovery. The symptoms predominantly affected the right side of her face and body, and PET demonstrated a corresponding increase in lentiform and caudate nucleus metabolism prevailing on the left side. After recovery, PET showed normal values in the regions previously studied. This study adds further evidence to support the theory that acute choreas are somehow the result of striatal hypermetabolism.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Sundén-Cullberg</LastName><ForeName>J</ForeName><Initials>J</Initials><AffiliationInfo><Affiliation>Department of Neurology, University Hospital, Uppsala, Sweden.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Tedroff</LastName><ForeName>J</ForeName><Initials>J</Initials></Author><Author ValidYN="Y"><LastName>Aquilonius</LastName><ForeName>S M</ForeName><Initials>SM</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D002363">Case Reports</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>UNITED STATES</Country><MedlineTA>Mov Disord</MedlineTA><NlmUniqueID>8610688</NlmUniqueID><ISSNLinking>0885-3185</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName MajorTopicYN="N" UI="D000208">Acute Disease</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D000328">Adult</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D016736">Antiphospholipid Syndrome</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000150">complications</QualifierName><QualifierName MajorTopicYN="N" UI="Q000378">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D001921">Brain</DescriptorName><QualifierName MajorTopicYN="N" UI="Q000378">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D016022">Case-Control Studies</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D002819">Chorea</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000209">etiology</QualifierName><QualifierName MajorTopicYN="N" UI="Q000378">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D003342">Corpus Striatum</DescriptorName><QualifierName MajorTopicYN="Y" UI="Q000378">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D005260">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D006801">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D008137">Longitudinal Studies</DescriptorName></MeshHeading><MeshHeading><DescriptorName MajorTopicYN="N" UI="D014055">Tomography, Emission-Computed</DescriptorName></MeshHeading></MeshHeadingList><NumberOfReferences>23</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1998</Year><Month>2</Month><Day>6</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1998</Year><Month>2</Month><Day>6</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1998</Year><Month>2</Month><Day>6</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">9452340</ArticleId><ArticleId IdType="doi">10.1002/mds.870130127</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Suède</li></country></list><tree><noCountry><name sortKey="Aquilonius, S M" sort="Aquilonius, S M" uniqKey="Aquilonius S" first="S M" last="Aquilonius">S M Aquilonius</name><name sortKey="Tedroff, J" sort="Tedroff, J" uniqKey="Tedroff J" first="J" last="Tedroff">J. Tedroff</name></noCountry><country name="Suède"><noRegion><name sortKey="Sunden Cullberg, J" sort="Sunden Cullberg, J" uniqKey="Sunden Cullberg J" first="J" last="Sundén-Cullberg">J. Sundén-Cullberg</name></noRegion></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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