Abnormal response to cortical activation in early stages of Huntington disease.
Identifieur interne : 003639 ( Ncbi/Checkpoint ); précédent : 003638; suivant : 003640Abnormal response to cortical activation in early stages of Huntington disease.
Auteurs : Fanny Mochel [France] ; Tra-My N'Guyen ; Dinesh Deelchand ; Daisy Rinaldi ; Romain Valabregue ; Claire Wary ; Pierre G. Carlier ; Alexandra Durr ; Pierre-Gilles HenrySource :
- Movement disorders : official journal of the Movement Disorder Society [ 1531-8257 ] ; 2012.
English descriptors
- KwdEn :
- Adenosine Triphosphate (metabolism), Case-Control Studies, Cerebral Cortex (metabolism), Cerebral Cortex (physiopathology), Cerebral Cortex (radionuclide imaging), Female, Humans, Huntington Disease (pathology), Huntington Disease (radionuclide imaging), Linear Models, Magnetic Resonance Spectroscopy, Male, Phosphates (metabolism), Phosphocreatine (metabolism), Phosphorus Isotopes (metabolism).
- MESH :
- chemical , metabolism : Adenosine Triphosphate, Phosphates, Phosphocreatine, Phosphorus Isotopes.
- metabolism : Cerebral Cortex.
- pathology : Huntington Disease.
- physiopathology : Cerebral Cortex.
- radionuclide imaging : Cerebral Cortex, Huntington Disease.
- Case-Control Studies, Female, Humans, Linear Models, Magnetic Resonance Spectroscopy, Male.
Abstract
We wished to identify noninvasive in vivo biomarkers of brain energy deficit in Huntington disease.
DOI: 10.1002/mds.25009
PubMed: 22517114
Affiliations:
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pubmed:22517114Le document en format XML
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<series><title level="j">Movement disorders : official journal of the Movement Disorder Society</title>
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<term>Huntington Disease (radionuclide imaging)</term>
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<term>Phosphorus Isotopes</term>
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<front><div type="abstract" xml:lang="en">We wished to identify noninvasive in vivo biomarkers of brain energy deficit in Huntington disease.</div>
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