Decreased striatal dopamine transporter binding in a patient with extrapontine myelinolysis
Identifieur interne : 005D61 ( Main/Merge ); précédent : 005D60; suivant : 005D62Decreased striatal dopamine transporter binding in a patient with extrapontine myelinolysis
Auteurs : Joong-Seok Kim [Corée du Sud] ; Kwang-Soo Lee [Corée du Sud] ; Si-Ryung Han [Corée du Sud] ; Yong-An Chung [Corée du Sud]Source :
- Movement Disorders [ 0885-3185 ] ; 2003-03.
English descriptors
- KwdEn :
- Antiparkinson Agents (therapeutic use), Dopamine Plasma Membrane Transport Proteins, Female, Humans, Hyponatremia (complications), Hyponatremia (therapy), IPT SPECT, Isotonic Solutions (therapeutic use), Levodopa (therapeutic use), Membrane Glycoproteins, Membrane Transport Proteins (metabolism), Middle Aged, Myelinolysis, Central Pontine (etiology), Myelinolysis, Central Pontine (metabolism), Myelinolysis, Central Pontine (radionuclide imaging), Nerve Tissue Proteins, Parkinsonian Disorders (drug therapy), Parkinsonian Disorders (etiology), Parkinsonian Disorders (radionuclide imaging), Protein Binding, Radiopharmaceuticals (diagnostic use), Tomography, Emission-Computed, Single-Photon, Treatment Outcome, Tropanes (diagnostic use), extrapontine myelinolysis, parkinsonism.
- MESH :
- chemical , diagnostic use : Radiopharmaceuticals, Tropanes.
- chemical , metabolism : Membrane Transport Proteins.
- chemical , therapeutic use : Antiparkinson Agents, Isotonic Solutions, Levodopa.
- chemical : Dopamine Plasma Membrane Transport Proteins, Membrane Glycoproteins, Nerve Tissue Proteins.
- complications : Hyponatremia.
- drug therapy : Parkinsonian Disorders.
- etiology : Myelinolysis, Central Pontine, Parkinsonian Disorders.
- metabolism : Myelinolysis, Central Pontine.
- radionuclide imaging : Myelinolysis, Central Pontine, Parkinsonian Disorders.
- therapy : Hyponatremia.
- Female, Humans, Middle Aged, Protein Binding, Tomography, Emission-Computed, Single-Photon, Treatment Outcome.
Abstract
We describe the case of a 61‐year‐old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [123I]N‐(3‐iodopropen‐2‐yl)‐2β‐carbomethoxy‐3β‐(4‐chlorophenyl) tropane and single photon emission computed tomography ([123I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism.
Url:
DOI: 10.1002/mds.10331
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<front><div type="abstract" xml:lang="en">We describe the case of a 61‐year‐old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [123I]N‐(3‐iodopropen‐2‐yl)‐2β‐carbomethoxy‐3β‐(4‐chlorophenyl) tropane and single photon emission computed tomography ([123I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism.</div>
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<front><div type="abstract" xml:lang="en">We describe the case of a 61‐year‐old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [123I]N‐(3‐iodopropen‐2‐yl)‐2β‐carbomethoxy‐3β‐(4‐chlorophenyl) tropane and single photon emission computed tomography ([123I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism.</div>
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<front><div type="abstract" xml:lang="en">We describe the case of a 61-year-old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [(123)I]N-(3-iodopropen-2-yl)-2beta-carbomethoxy-3beta-(4-chlorophenyl) tropane and single photon emission computed tomography ([(123)I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism.</div>
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