Smoking‐responsive juvenile‐onset Parkinsonism
Identifieur interne : 003A90 ( Main/Merge ); précédent : 003A89; suivant : 003A91Smoking‐responsive juvenile‐onset Parkinsonism
Auteurs : Hasmet Ayhan Hanagasi [Turquie] ; Andrew Lees (neurologue) [Royaume-Uni] ; Janel O. Johnson [Royaume-Uni, États-Unis] ; Andrew Singleton [États-Unis] ; Murat Emre [Turquie]Source :
- Movement Disorders [ 0885-3185 ] ; 2007-01.
English descriptors
- KwdEn :
- Adult, Humans, Male, Nicotine (therapeutic use), Nicotinic Agonists (therapeutic use), Parkinsonian Disorders (drug therapy), Parkinsonian Disorders (radionuclide imaging), Smoking, Tomography, Emission-Computed, Single-Photon, Tropanes (pharmacokinetics), juvenile‐onset Parkinsonism, nicotine, sensitivity.
- MESH :
- chemical , pharmacokinetics : Tropanes.
- chemical , therapeutic use : Nicotine, Nicotinic Agonists.
- drug therapy : Parkinsonian Disorders.
- radionuclide imaging : Parkinsonian Disorders.
- Adult, Humans, Male, Smoking, Tomography, Emission-Computed, Single-Photon.
Abstract
We describe a patient with juvenile levodopa‐responsive Parkinsonism who reported a dramatic response to cigarette smoking with transient but marked improvement of motor symptoms associated with oculogyric crises and psychotic behavior. His beta‐CIT single‐photon emission computed tomography scan showed a complete absence of presynaptic dopaminergic nerve terminals. © 2006 Movement Disorder Society
Url:
DOI: 10.1002/mds.21177
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<front><div type="abstract" xml:lang="en">We describe a patient with juvenile levodopa‐responsive Parkinsonism who reported a dramatic response to cigarette smoking with transient but marked improvement of motor symptoms associated with oculogyric crises and psychotic behavior. His beta‐CIT single‐photon emission computed tomography scan showed a complete absence of presynaptic dopaminergic nerve terminals. © 2006 Movement Disorder Society</div>
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<front><div type="abstract" xml:lang="en">We describe a patient with juvenile levodopa‐responsive Parkinsonism who reported a dramatic response to cigarette smoking with transient but marked improvement of motor symptoms associated with oculogyric crises and psychotic behavior. His beta‐CIT single‐photon emission computed tomography scan showed a complete absence of presynaptic dopaminergic nerve terminals. © 2006 Movement Disorder Society</div>
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<front><div type="abstract" xml:lang="en">We describe a patient with juvenile levodopa-responsive Parkinsonism who reported a dramatic response to cigarette smoking with transient but marked improvement of motor symptoms associated with oculogyric crises and psychotic behavior. His beta-CIT single-photon emission computed tomography scan showed a complete absence of presynaptic dopaminergic nerve terminals.</div>
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