Control of oncogene expression by antisense nucleic acids
Identifieur interne : 001652 ( Istex/Corpus ); précédent : 001651; suivant : 001653Control of oncogene expression by antisense nucleic acids
Auteurs : C. HélèneSource :
- European Journal of Cancer [ 0959-8049 ] ; 1994.
English descriptors
- Teeft :
- Acad, Adjuvant tamoxifen, Animal models, Animal studies, Antigene, Antigene oligonucleotides, Antigene strategy, Antisense, Antisense inhibition, Antisense oligonucleotide, Antisense oligonucleotides, Antisense research, Base pairs, Biol chem, Biological effects, Biological response, Block translation, Breast cancer, Breast cancer patients, Cell culture experiments, Cell cultures, Cell proliferation, Cellular uptake, Circular oligonucleotides, Cleavage, Clinical trials, Complementary sequence, Control gene expression, Double helix, Early breast cancer, Endometrial cancer, Form hoogsteen, Gene, Gene expression, Gene promoter, Helene, Helix, Hoogsteen, Hoogsteen hydrogen bonds, Human bladder carcinoma, Immune response, Intercalating agents, Large number, Major groove, Malignant cells, Mrna, Mutation, Nanoparticles, Nat1, Nucl acids, Nucleic, Nucleic acid, Nucleic acids, Nude mice, Oligonucleotide, Oligonucleotide decoy, Oligonucleotides, Oligopurine sequence, Oncogene, Oncogene expression, Oncogene family, Original tumour, Polyalkylcyanoacrylate nanoparticles, Proc, Proc nat1 acad, Proc nat1 acadsci, Reactive group, Repair genes, Ribozyme, Short oligonucleotides, Synthetic antisense oligonucleotides, Tamoxifen, Target sequence, Therapeutical applications, Transcription, Transcription factor, Transcription factors, Transgenic mice, Translation machinery, Triple helix, Triple helix formation, Tumour, Tumour cells, Tumour development, Tumour growth, Viral.
Url:
DOI: 10.1016/0959-8049(93)E0352-Q
Links to Exploration step
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<head><ce:article-footnote><ce:label>☆</ce:label>
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