[The basic patterns in the development of lymph circulatory failure in the heart].
Identifieur interne : 005D40 ( PubMed/Corpus ); précédent : 005D39; suivant : 005D41[The basic patterns in the development of lymph circulatory failure in the heart].
Auteurs : A S Gavrish ; S A KravetsSource :
- Fiziologicheskii zhurnal [ 0201-8489 ]
English descriptors
- KwdEn :
- Adaptation, Physiological (physiology), Animals, Arteriosclerosis (etiology), Arteriosclerosis (physiopathology), Cholesterol, Dietary (administration & dosage), Chronic Disease, Coronary Disease (chemically induced), Coronary Disease (physiopathology), Heart (physiopathology), Hypercholesterolemia (etiology), Hypercholesterolemia (physiopathology), Lymph (physiology), Lymphatic System (physiopathology), Lymphedema (etiology), Lymphedema (physiopathology), Physical Exertion (physiology), Rabbits, Recurrence, Vasopressins.
- MESH :
- chemical , administration & dosage : Cholesterol, Dietary.
- chemically induced : Coronary Disease.
- etiology : Arteriosclerosis, Hypercholesterolemia, Lymphedema.
- physiology : Adaptation, Physiological, Lymph, Physical Exertion.
- physiopathology : Arteriosclerosis, Coronary Disease, Heart, Hypercholesterolemia, Lymphatic System, Lymphedema.
- Animals, Chronic Disease, Rabbits, Recurrence, Vasopressins.
Abstract
63 adult rabbits were used to stimulate single physical exercise, acute and relapsing coronary insufficiency, alimentary atherosclerosis and to study adaptive and pathologic changes in the lymphatic channel (LC) of the heart. LC of the heart in animals with alimentary atherosclerosis after cholesterol exclusion from their diet was studied to determine reversibility of these changes. LC of the heart was injected by differently coloured masses and was investigated by the stereoangioscopic method on clarified preparations, on semithin sections and by the electron-microscopic method. Changes in different elements of a lymphatic net were of the stereotype character, as a whole. Morphofunctional reactions determining the development of the chronic insufficiency of the lymph flow in the heart form as successively following phases which change both due to exposition of the pathological process-initiated effect and due to certain endogenic factors arising during arrangement of LC of the heart.
PubMed: 2226941
Links to Exploration step
pubmed:2226941Le document en format XML
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LC of the heart in animals with alimentary atherosclerosis after cholesterol exclusion from their diet was studied to determine reversibility of these changes. LC of the heart was injected by differently coloured masses and was investigated by the stereoangioscopic method on clarified preparations, on semithin sections and by the electron-microscopic method. Changes in different elements of a lymphatic net were of the stereotype character, as a whole. Morphofunctional reactions determining the development of the chronic insufficiency of the lymph flow in the heart form as successively following phases which change both due to exposition of the pathological process-initiated effect and due to certain endogenic factors arising during arrangement of LC of the heart.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">2226941</PMID><DateCreated><Year>1990</Year><Month>12</Month><Day>27</Day></DateCreated><DateCompleted><Year>1990</Year><Month>12</Month><Day>27</Day></DateCompleted><DateRevised><Year>2008</Year><Month>11</Month><Day>21</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0201-8489</ISSN><JournalIssue CitedMedium="Print"><Volume>36</Volume><Issue>4</Issue><PubDate><MedlineDate>1990 Jul-Aug</MedlineDate></PubDate></JournalIssue><Title>Fiziologicheskii zhurnal</Title><ISOAbbreviation>Fiziol Zh</ISOAbbreviation></Journal><ArticleTitle>[The basic patterns in the development of lymph circulatory failure in the heart].</ArticleTitle><Pagination><MedlinePgn>15-22</MedlinePgn></Pagination><Abstract><AbstractText>63 adult rabbits were used to stimulate single physical exercise, acute and relapsing coronary insufficiency, alimentary atherosclerosis and to study adaptive and pathologic changes in the lymphatic channel (LC) of the heart. LC of the heart in animals with alimentary atherosclerosis after cholesterol exclusion from their diet was studied to determine reversibility of these changes. LC of the heart was injected by differently coloured masses and was investigated by the stereoangioscopic method on clarified preparations, on semithin sections and by the electron-microscopic method. Changes in different elements of a lymphatic net were of the stereotype character, as a whole. Morphofunctional reactions determining the development of the chronic insufficiency of the lymph flow in the heart form as successively following phases which change both due to exposition of the pathological process-initiated effect and due to certain endogenic factors arising during arrangement of LC of the heart.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Gavrish</LastName><ForeName>A S</ForeName><Initials>AS</Initials></Author><Author ValidYN="Y"><LastName>Kravets</LastName><ForeName>S A</ForeName><Initials>SA</Initials></Author></AuthorList><Language>rus</Language><PublicationTypeList><PublicationType UI="D003160">Comparative Study</PublicationType><PublicationType UI="D004740">English Abstract</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList><VernacularTitle>Osnovnye zakonomernosti razvitiia nedostatochnosti limfoobrashcheniia v serdtse.</VernacularTitle></Article><MedlineJournalInfo><Country>Ukraine</Country><MedlineTA>Fiziol Zh</MedlineTA><NlmUniqueID>7806822</NlmUniqueID><ISSNLinking>0201-8489</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D002791">Cholesterol, Dietary</NameOfSubstance></Chemical><Chemical><RegistryNumber>11000-17-2</RegistryNumber><NameOfSubstance UI="D014667">Vasopressins</NameOfSubstance></Chemical></ChemicalList><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000222" MajorTopicYN="N">Adaptation, Physiological</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001161" MajorTopicYN="N">Arteriosclerosis</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="N">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D002791" MajorTopicYN="N">Cholesterol, Dietary</DescriptorName><QualifierName UI="Q000008" MajorTopicYN="N">administration & dosage</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D002908" MajorTopicYN="N">Chronic Disease</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D003327" MajorTopicYN="N">Coronary Disease</DescriptorName><QualifierName UI="Q000139" MajorTopicYN="N">chemically induced</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006321" MajorTopicYN="N">Heart</DescriptorName><QualifierName UI="Q000503" MajorTopicYN="Y">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006937" MajorTopicYN="N">Hypercholesterolemia</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="N">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008196" MajorTopicYN="N">Lymph</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008208" MajorTopicYN="N">Lymphatic System</DescriptorName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008209" MajorTopicYN="N">Lymphedema</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName><QualifierName UI="Q000503" MajorTopicYN="N">physiopathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005082" MajorTopicYN="N">Physical Exertion</DescriptorName><QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011817" MajorTopicYN="N">Rabbits</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D012008" MajorTopicYN="N">Recurrence</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D014667" MajorTopicYN="N">Vasopressins</DescriptorName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1990</Year><Month>7</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1990</Year><Month>7</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1990</Year><Month>7</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">2226941</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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