Causes and consequences of lymphatic disease
Identifieur interne : 007745 ( Istex/Corpus ); précédent : 007744; suivant : 007746Causes and consequences of lymphatic disease
Auteurs : Stanley G. RocksonSource :
- Annals of the New York Academy of Sciences [ 0077-8923 ] ; 2010-10.
Abstract
The visceral manifestations of lymphatic disorders (lymphangiomatosis and lymphangiectasia) are particularly severe. Any pathology of the lymphatic vasculature, whether superficial or internal, regional, or systemic, is predominated by the appearance of lymphedema, the characteristic form of tissue edema that occurs when lymphatic dysfunction supervenes. Disease manifestations may include dysregulation of body fluid homeostasis, immune traffic impairment, and disturbances of lipid and protein reabsorption from the gut lumen. The appearance of lymphatic edema invokes complex biological alterations. Many of these changes seem to relate uniquely to chronic lymphatic edema, including a profound stimulus to collagen and adipose deposition. Despite the recent advances in our understanding of these disorders, substantial knowledge gaps remain; these gaps inhibit our ability to accurately identify, categorize, treat, and prevent these diseases. Future diagnostic, therapeutic, and reproductive decisions for affected individuals require an accurate knowledge of the clinical and laboratory presentation, mode of inheritance, treatment response, outcomes, and prognosis.
Url:
DOI: 10.1111/j.1749-6632.2010.05804.x
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Rockson</name><affiliation><mods:affiliation>Center for Lymphatic and Venous Disorders, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:FE25EDF0017D787AB3F90AE777CBE2FB5A228AF8</idno><date when="2010" year="2010">2010</date><idno type="doi">10.1111/j.1749-6632.2010.05804.x</idno><idno type="url">https://api.istex.fr/document/FE25EDF0017D787AB3F90AE777CBE2FB5A228AF8/fulltext/pdf</idno><idno type="wicri:Area/Istex/Corpus">007745</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">007745</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main">Causes and consequences of lymphatic disease</title><author><name sortKey="Rockson, Stanley G" sort="Rockson, Stanley G" uniqKey="Rockson S" first="Stanley G." last="Rockson">Stanley G. Rockson</name><affiliation><mods:affiliation>Center for Lymphatic and Venous Disorders, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">Annals of the New York Academy of Sciences</title><title level="j" type="alt">ANNALS OF NEW YORK ACADEMY SCIENCES</title><idno type="ISSN">0077-8923</idno><idno type="eISSN">1749-6632</idno><imprint><biblScope unit="vol">1207</biblScope><biblScope unit="page" from="E2">E2</biblScope><biblScope unit="page" to="E6">E6</biblScope><biblScope unit="page-count">5</biblScope><publisher>Blackwell Publishing Inc</publisher><pubPlace>Malden, USA</pubPlace><date type="published" when="2010-10">2010-10</date></imprint><idno type="ISSN">0077-8923</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0077-8923</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">The visceral manifestations of lymphatic disorders (lymphangiomatosis and lymphangiectasia) are particularly severe. Any pathology of the lymphatic vasculature, whether superficial or internal, regional, or systemic, is predominated by the appearance of lymphedema, the characteristic form of tissue edema that occurs when lymphatic dysfunction supervenes. Disease manifestations may include dysregulation of body fluid homeostasis, immune traffic impairment, and disturbances of lipid and protein reabsorption from the gut lumen. The appearance of lymphatic edema invokes complex biological alterations. Many of these changes seem to relate uniquely to chronic lymphatic edema, including a profound stimulus to collagen and adipose deposition. Despite the recent advances in our understanding of these disorders, substantial knowledge gaps remain; these gaps inhibit our ability to accurately identify, categorize, treat, and prevent these diseases. Future diagnostic, therapeutic, and reproductive decisions for affected individuals require an accurate knowledge of the clinical and laboratory presentation, mode of inheritance, treatment response, outcomes, and prognosis.</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>lymphatic</json:string><json:string>lymphedema</json:string><json:string>rockson</json:string><json:string>biol</json:string><json:string>lymphat</json:string><json:string>acad</json:string><json:string>adipose</json:string><json:string>prox1</json:string><json:string>lymphatic edema</json:string><json:string>lymphatic disease</json:string><json:string>york academy</json:string><json:string>tissue edema</json:string><json:string>breast cancer</json:string><json:string>lymphatic diseases</json:string><json:string>edema</json:string><json:string>gastrointestinal tract</json:string><json:string>lymphatic vasculature</json:string><json:string>lymphatic dysfunction</json:string><json:string>secondary lymphedema</json:string><json:string>therapeutic lymphangiogenesis</json:string><json:string>therapeutic options</json:string><json:string>adipose deposition</json:string><json:string>recent advances</json:string><json:string>substantial knowledge gaps</json:string><json:string>reproductive decisions</json:string><json:string>accurate knowledge</json:string><json:string>laboratory presentation</json:string><json:string>treatment response</json:string><json:string>bioimpedance spectroscopy</json:string><json:string>lymphatic hyperplasia</json:string><json:string>venous disorders</json:string><json:string>characteristic form</json:string><json:string>differential diagnosis</json:string><json:string>clinical presentations</json:string><json:string>cardiovascular medicine</json:string><json:string>stanford university school</json:string><json:string>disease prevalence</json:string><json:string>natural history</json:string><json:string>lymphatic dysfunction supervenes</json:string><json:string>disease manifestations</json:string><json:string>immune impairment</json:string><json:string>lymphatic disease rockson</json:string><json:string>primary lymphedema</json:string><json:string>clinical detectability</json:string><json:string>disease risk</json:string><json:string>radionuclide lymphoscintigraphy</json:string><json:string>bioimpedance analysis</json:string><json:string>life expectancy</json:string><json:string>complex biology</json:string><json:string>protein reabsorption</json:string><json:string>early observations</json:string><json:string>lymphatic function</json:string><json:string>visceral manifestations</json:string><json:string>chronic lymphatic edema</json:string><json:string>fontan operation</json:string><json:string>profound stimulus</json:string><json:string>lymphatic disorders</json:string><json:string>lymphatic system</json:string></teeft></keywords><author><json:item><name>Stanley G. 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Future diagnostic, therapeutic, and reproductive decisions for affected individuals require an accurate knowledge of the clinical and laboratory presentation, mode of inheritance, treatment response, outcomes, and prognosis.</p></abstract><textClass><keywords xml:lang="en"><term xml:id="k1">lymphedema</term><term xml:id="k2">lymphangiectasia</term><term xml:id="k3">lymphangiomatosis</term><term xml:id="k4">chylous effusion</term><term xml:id="k5">protein‐losing enteropathy</term><term xml:id="k6">bioimpedance spectroscopy</term><term xml:id="k7">Prox‐1</term></keywords><classCode scheme="tocHeading1">Original Articles</classCode></textClass><langUsage><language ident="EN"></language></langUsage></profileDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/FE25EDF0017D787AB3F90AE777CBE2FB5A228AF8/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Inc</publisherName><publisherLoc>Malden, USA</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1749-6632</doi><issn type="print">0077-8923</issn><issn type="electronic">1749-6632</issn><idGroup><id type="product" value="NYAS"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="ANNALS OF NEW YORK ACADEMY SCIENCES">Annals of the New York Academy of Sciences</title></titleGroup></publicationMeta><publicationMeta level="part" position="12072"><doi origin="wiley">10.1111/nyas.2010.1207.issue-s1</doi><titleGroup><title type="main">S1 Lymphatics in the Digestive System: Physiology, Health, and Disease</title></titleGroup><numberingGroup><numbering type="journalVolume" number="12072">1207</numbering><numbering type="supplement">s1</numbering></numberingGroup><coverDate startDate="2010-10">October 2010</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="2" status="forIssue"><doi origin="wiley">10.1111/j.1749-6632.2010.05804.x</doi><idGroup><id type="unit" value="NYAS5804"></id></idGroup><countGroup><count type="pageTotal" number="5"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><copyright>© 2010 New York Academy of Sciences</copyright><eventGroup><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.4.9 mode:FullText" date="2011-05-18"></event><event type="publishedOnlineFinalForm" date="2010-10-20"></event><event type="firstOnline" date="2010-10-20"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:4.0.1" date="2014-03-19"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-14"></event></eventGroup><numberingGroup><numbering type="pageFirst">E2</numbering><numbering type="pageLast">E6</numbering></numberingGroup><correspondenceTo>Address for correspondence: Stanley G. Rockson, M.D., Center for Lymphatic and Venous Disorders, Division of Cardiovascular Medicine, Stanford University School of Medicine, Falk Cardiovascular Research Center, Stanford, CA 94305. <email>rockson@stanford.edu</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:NYAS.NYAS5804.pdf"></link></linkGroup></publicationMeta><contentMeta><countGroup><count type="figureTotal" number="0"></count><count type="tableTotal" number="0"></count><count type="formulaTotal" number="0"></count><count type="referenceTotal" number="48"></count><count type="linksCrossRef" number="41"></count></countGroup><titleGroup><title type="main">Causes and consequences of lymphatic disease</title><title type="shortAuthors">Rockson</title><title type="short">Causes and consequences of lymphatic disease</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>Stanley G.</givenNames><familyName>Rockson</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="US"><unparsedAffiliation>Center for Lymphatic and Venous Disorders, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, California</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">lymphedema</keyword><keyword xml:id="k2">lymphangiectasia</keyword><keyword xml:id="k3">lymphangiomatosis</keyword><keyword xml:id="k4">chylous effusion</keyword><keyword xml:id="k5">protein‐losing enteropathy</keyword><keyword xml:id="k6">bioimpedance spectroscopy</keyword><keyword xml:id="k7">Prox‐1</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>The visceral manifestations of lymphatic disorders (lymphangiomatosis and lymphangiectasia) are particularly severe. 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Any pathology of the lymphatic vasculature, whether superficial or internal, regional, or systemic, is predominated by the appearance of lymphedema, the characteristic form of tissue edema that occurs when lymphatic dysfunction supervenes. Disease manifestations may include dysregulation of body fluid homeostasis, immune traffic impairment, and disturbances of lipid and protein reabsorption from the gut lumen. The appearance of lymphatic edema invokes complex biological alterations. Many of these changes seem to relate uniquely to chronic lymphatic edema, including a profound stimulus to collagen and adipose deposition. Despite the recent advances in our understanding of these disorders, substantial knowledge gaps remain; these gaps inhibit our ability to accurately identify, categorize, treat, and prevent these diseases. 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