H2N2V1, PubMed, Corpus, bibRecord, 000645

Influenza virus: association of mouse-lung virulence with plaque formation in mouse kidney cells.

Identifieur interne : 000645 ( PubMed/Corpus ); précédent : 000644; suivant : 000646

Influenza virus: association of mouse-lung virulence with plaque formation in mouse kidney cells.

Auteurs : B. Flehmig ; A. Vallbracht ; H J Gerth

Source :

Intervirology [ 0300-5526 ] ; 1976.

RBID : pubmed:1010711

English descriptors

KwdEn :
- Animals, Antigens, Viral (analysis), Culture Techniques, Hemagglutinins, Viral (analysis), Influenza A virus (growth & development), Influenza A virus (immunology), Influenza A virus (pathogenicity), Lung (microbiology), Mice, Neuraminidase (immunology), Recombination, Genetic, Viral Plaque Assay, Virulence, Virus Replication.
MESH :
- chemical , analysis : Antigens, Viral, Hemagglutinins, Viral.
- growth & development : Influenza A virus.
- immunology : Influenza A virus, Neuraminidase.
- microbiology : Lung.
- pathogenicity : Influenza A virus.
- Animals, Culture Techniques, Mice, Recombination, Genetic, Viral Plaque Assay, Virulence, Virus Replication.

Abstract

In genetic recombination experiments with the mouse-lung-adapted human influenza A/Engl/1/61 (H2N2) and an avian influenza strain A/Rostock/34 (FPV) (Hav1N1) which is avirulent for the mouse lung, recombinants in which hemagglutinin and neuraminidase were either segregated (Hav1N2; H2N1) or not segregated (Hav1N1) were selected. The recombinants were studied for mouse-lung virulence and their ability to propagate in mouse kidney cells, mouse embryo fibroblasts, chick embryo kidney cells and chick embryo fibroblasts. An association between plaque formation in mouse kidney cells and mouse-lung virulence was found.

DOI: 10.1159/000149953
PubMed: 1010711

Links to Exploration step

pubmed:1010711

Le document en format XML

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<author><name sortKey="Flehmig, B" sort="Flehmig, B" uniqKey="Flehmig B" first="B" last="Flehmig">B. Flehmig</name>
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<author><name sortKey="Vallbracht, A" sort="Vallbracht, A" uniqKey="Vallbracht A" first="A" last="Vallbracht">A. Vallbracht</name>
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<author><name sortKey="Gerth, H J" sort="Gerth, H J" uniqKey="Gerth H" first="H J" last="Gerth">H J Gerth</name>
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<author><name sortKey="Flehmig, B" sort="Flehmig, B" uniqKey="Flehmig B" first="B" last="Flehmig">B. Flehmig</name>
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<author><name sortKey="Vallbracht, A" sort="Vallbracht, A" uniqKey="Vallbracht A" first="A" last="Vallbracht">A. Vallbracht</name>
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<series><title level="j">Intervirology</title>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term>
<term>Antigens, Viral (analysis)</term>
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<term>Hemagglutinins, Viral (analysis)</term>
<term>Influenza A virus (growth & development)</term>
<term>Influenza A virus (immunology)</term>
<term>Influenza A virus (pathogenicity)</term>
<term>Lung (microbiology)</term>
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<term>Recombination, Genetic</term>
<term>Viral Plaque Assay</term>
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<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Culture Techniques</term>
<term>Mice</term>
<term>Recombination, Genetic</term>
<term>Viral Plaque Assay</term>
<term>Virulence</term>
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<front><div type="abstract" xml:lang="en">In genetic recombination experiments with the mouse-lung-adapted human influenza A/Engl/1/61 (H2N2) and an avian influenza strain A/Rostock/34 (FPV) (Hav1N1) which is avirulent for the mouse lung, recombinants in which hemagglutinin and neuraminidase were either segregated (Hav1N2; H2N1) or not segregated (Hav1N1) were selected. The recombinants were studied for mouse-lung virulence and their ability to propagate in mouse kidney cells, mouse embryo fibroblasts, chick embryo kidney cells and chick embryo fibroblasts. An association between plaque formation in mouse kidney cells and mouse-lung virulence was found.</div>
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<Month>03</Month>
<Day>15</Day>
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<DateRevised><Year>2018</Year>
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<Title>Intervirology</Title>
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<ArticleTitle>Influenza virus: association of mouse-lung virulence with plaque formation in mouse kidney cells.</ArticleTitle>
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<Abstract><AbstractText>In genetic recombination experiments with the mouse-lung-adapted human influenza A/Engl/1/61 (H2N2) and an avian influenza strain A/Rostock/34 (FPV) (Hav1N1) which is avirulent for the mouse lung, recombinants in which hemagglutinin and neuraminidase were either segregated (Hav1N2; H2N1) or not segregated (Hav1N1) were selected. The recombinants were studied for mouse-lung virulence and their ability to propagate in mouse kidney cells, mouse embryo fibroblasts, chick embryo kidney cells and chick embryo fibroblasts. An association between plaque formation in mouse kidney cells and mouse-lung virulence was found.</AbstractText>
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<MeshHeading><DescriptorName UI="D008168" MajorTopicYN="N">Lung</DescriptorName>
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<MeshHeading><DescriptorName UI="D051379" MajorTopicYN="N">Mice</DescriptorName>
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<MeshHeading><DescriptorName UI="D009439" MajorTopicYN="N">Neuraminidase</DescriptorName>
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<MeshHeading><DescriptorName UI="D011995" MajorTopicYN="N">Recombination, Genetic</DescriptorName>
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<MeshHeading><DescriptorName UI="D010948" MajorTopicYN="N">Viral Plaque Assay</DescriptorName>
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Influenza virus: association of mouse-lung virulence with plaque formation in mouse kidney cells.

Influenza virus: association of mouse-lung virulence with plaque formation in mouse kidney cells.

Source :

English descriptors

Abstract

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki