Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses.
Identifieur interne : 000416 ( PubMed/Corpus ); précédent : 000415; suivant : 000417Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses.
Auteurs : M. Tamura ; R G Webster ; F A EnnisSource :
- Journal of virology [ 0022-538X ] ; 1994.
English descriptors
- KwdEn :
- Animals, Antibodies, Monoclonal, Antibodies, Viral (biosynthesis), Antigen-Presenting Cells (immunology), Antigen-Presenting Cells (microbiology), Antigens, Viral, Cell Line, Cross Reactions, Humans, Influenza A virus (classification), Influenza A virus (immunology), Influenza, Human (enzymology), Influenza, Human (immunology), Influenza, Human (microbiology), Male, Mice, Mice, Inbred BALB C, Neuraminidase (immunology), Neutralization Tests, Receptors, Fc (metabolism).
- MESH :
- chemical , biosynthesis : Antibodies, Viral.
- chemical , immunology : Neuraminidase.
- chemical , metabolism : Receptors, Fc.
- chemical : Antibodies, Monoclonal, Antigens, Viral.
- classification : Influenza A virus.
- enzymology : Influenza, Human.
- immunology : Antigen-Presenting Cells, Influenza A virus, Influenza, Human.
- microbiology : Antigen-Presenting Cells, Influenza, Human.
- Animals, Cell Line, Cross Reactions, Humans, Male, Mice, Mice, Inbred BALB C, Neutralization Tests.
Abstract
Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from H1N1-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses. These results show that subtype cross-reactive, nonneutralizing antibodies augment the uptake of influenza A virus strains of different subtypes. Antibodies to neuraminidase may contribute to the enhanced uptake of viruses of a different subtype, because N2-specific monoclonal antibodies augmented the uptake of both A/Japan/305/57 (H2N2) and A/Port Chalmers/1/73 (H3N2) viruses.
PubMed: 8189489
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pubmed:8189489Le document en format XML
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<author><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M" last="Tamura">M. Tamura</name>
<affiliation><nlm:affiliation>Department of Medicine, University of Massachusetts Medical Center, Worcester 01655.</nlm:affiliation>
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<author><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R G" last="Webster">R G Webster</name>
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<author><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F A" last="Ennis">F A Ennis</name>
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<author><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M" last="Tamura">M. Tamura</name>
<affiliation><nlm:affiliation>Department of Medicine, University of Massachusetts Medical Center, Worcester 01655.</nlm:affiliation>
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<author><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R G" last="Webster">R G Webster</name>
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<term>Antigen-Presenting Cells (microbiology)</term>
<term>Antigens, Viral</term>
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<term>Influenza A virus (classification)</term>
<term>Influenza A virus (immunology)</term>
<term>Influenza, Human (enzymology)</term>
<term>Influenza, Human (immunology)</term>
<term>Influenza, Human (microbiology)</term>
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<term>Neuraminidase (immunology)</term>
<term>Neutralization Tests</term>
<term>Receptors, Fc (metabolism)</term>
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<keywords scheme="MESH" type="chemical" qualifier="immunology" xml:lang="en"><term>Neuraminidase</term>
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<term>Antigens, Viral</term>
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<keywords scheme="MESH" qualifier="enzymology" xml:lang="en"><term>Influenza, Human</term>
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<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Antigen-Presenting Cells</term>
<term>Influenza A virus</term>
<term>Influenza, Human</term>
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<term>Cross Reactions</term>
<term>Humans</term>
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<front><div type="abstract" xml:lang="en">Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from H1N1-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses. These results show that subtype cross-reactive, nonneutralizing antibodies augment the uptake of influenza A virus strains of different subtypes. Antibodies to neuraminidase may contribute to the enhanced uptake of viruses of a different subtype, because N2-specific monoclonal antibodies augmented the uptake of both A/Japan/305/57 (H2N2) and A/Port Chalmers/1/73 (H3N2) viruses.</div>
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<Abstract><AbstractText>Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from H1N1-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses. These results show that subtype cross-reactive, nonneutralizing antibodies augment the uptake of influenza A virus strains of different subtypes. Antibodies to neuraminidase may contribute to the enhanced uptake of viruses of a different subtype, because N2-specific monoclonal antibodies augmented the uptake of both A/Japan/305/57 (H2N2) and A/Port Chalmers/1/73 (H3N2) viruses.</AbstractText>
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