Perspectives on pandemics : A research agenda
Identifieur interne :
000084 ( PascalFrancis/Corpus );
précédent :
000083;
suivant :
000085
Perspectives on pandemics : A research agenda
Auteurs : E. D. KilbourneSource :
-
The Journal of infectious diseases [ 0022-1899 ] ; 1997.
RBID : Pascal:97-0460472
Descripteurs français
English descriptors
Abstract
During the 20th century, indisputable pandemics of influenza occurred in 1918, 1957, and 1968. The pandemics of 1957 (A/H2N2) and 1968 (A/H3N2) were associated with major antigenic changes in the virus, probably reflecting introduction by recombination of animal virus genes. The 1918 epidemic is beyond the reach of modern virology but, based on seroarcheology, appears to have been caused by a virus very similar to present swine (A/H1N1) influenza viruses. Changes in both principal antigens of the A/H1N1 subtype in 1947 resulted in total vaccine failure and pandemic spread of virus. On the basis of three periods of prevalence in the 20th century, A/H1N1 may be the "default" human virus, although the 39-year persistence of A/H3N2 to the present challenges this view. Only H1, H2, and H3 and N1 and N2 antigens have been found in human influenza viruses, but virologic history is too brief to preclude the contribution of other antigens to future pandemics.
Notice en format standard (ISO 2709)
Pour connaître la documentation sur le format Inist Standard.
| pA |
| A01 | 01 | 1 | | @0 0022-1899 |
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| A02 | 01 | | | @0 JIDIAQ |
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| A03 | | 1 | | @0 J. infect. dis. |
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| A05 | | | | @2 176 |
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| A06 | | | | @3 SUP1 |
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| A08 | 01 | 1 | ENG | @1 Perspectives on pandemics : A research agenda |
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| A11 | 01 | 1 | | @1 KILBOURNE (E. D.) |
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| A12 | 01 | 1 | | @1 MONTO (Arnold S.) @9 ed. |
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| A14 | 01 | | | @1 Department of Microbiology and Immunology, New York Medical College @2 Valhalla, New York @3 USA @Z 1 aut. |
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| A15 | 01 | | | @1 Department of Epidemiology, University of Michigan School of Public Health @2 Ann Arbor, Michigan @3 USA @Z 1 aut. |
|---|
| A18 | 01 | 1 | | @1 National Institutes of Health @2 Bethesda, MD @3 USA @9 patr. |
|---|
| A18 | 02 | 1 | | @1 University of Michigan @2 Ann Arbor, Michigan @3 USA @9 patr. |
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| A20 | | | | @2 S29-S31 |
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| A21 | | | | @1 1997 |
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| A23 | 01 | | | @0 ENG |
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| A43 | 01 | | | @1 INIST @2 2052 @5 354000067739650060 |
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| A44 | | | | @0 0000 @1 © 1997 INIST-CNRS. All rights reserved. |
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| A45 | | | | @0 23 ref. |
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| A47 | 01 | 1 | | @0 97-0460472 |
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| A60 | | | | @1 P @2 C |
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| A61 | | | | @0 A |
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| A64 | 01 | 1 | | @0 The Journal of infectious diseases |
|---|
| A66 | 01 | | | @0 USA |
|---|
| C01 | 01 | | ENG | @0 During the 20th century, indisputable pandemics of influenza occurred in 1918, 1957, and 1968. The pandemics of 1957 (A/H2N2) and 1968 (A/H3N2) were associated with major antigenic changes in the virus, probably reflecting introduction by recombination of animal virus genes. The 1918 epidemic is beyond the reach of modern virology but, based on seroarcheology, appears to have been caused by a virus very similar to present swine (A/H1N1) influenza viruses. Changes in both principal antigens of the A/H1N1 subtype in 1947 resulted in total vaccine failure and pandemic spread of virus. On the basis of three periods of prevalence in the 20th century, A/H1N1 may be the "default" human virus, although the 39-year persistence of A/H3N2 to the present challenges this view. Only H1, H2, and H3 and N1 and N2 antigens have been found in human influenza viruses, but virologic history is too brief to preclude the contribution of other antigens to future pandemics. |
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| C03 | 01 | X | ENG | @0 Influenza @5 01 |
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| C03 | 01 | X | SPA | @0 Gripe @5 01 |
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| C03 | 02 | X | FRE | @0 Homme @5 02 |
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| C03 | 02 | X | ENG | @0 Human @5 02 |
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| C03 | 02 | X | SPA | @0 Hombre @5 02 |
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| C03 | 03 | X | FRE | @0 Epidémiologie @5 03 |
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| C03 | 03 | X | ENG | @0 Epidemiology @5 03 |
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| C03 | 03 | X | SPA | @0 Epidemiología @5 03 |
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| C03 | 04 | X | FRE | @0 Historique @5 04 |
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| C03 | 04 | X | ENG | @0 Case history @5 04 |
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| C03 | 04 | X | GER | @0 Entwicklungsgeschichte @5 04 |
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| C03 | 04 | X | SPA | @0 Histórica @5 04 |
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| C03 | 05 | X | FRE | @0 Perspective @5 05 |
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| C03 | 05 | X | ENG | @0 Perspective @5 05 |
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| C03 | 05 | X | SPA | @0 Perspectiva @5 05 |
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| C03 | 06 | X | FRE | @0 Pandémie @4 INC @5 91 |
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| C07 | 01 | X | FRE | @0 Virose |
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| C07 | 01 | X | ENG | @0 Viral disease |
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| C07 | 01 | X | SPA | @0 Virosis |
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| C07 | 02 | X | FRE | @0 Infection |
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| C07 | 02 | X | ENG | @0 Infection |
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| C07 | 02 | X | SPA | @0 Infección |
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| N21 | | | | @1 279 |
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| pR |
| A30 | 01 | 1 | ENG | @1 Pandemic Influenza: Confronting a Re-emergent Threat @3 Bethesda, Maryland USA @4 1995-12-11 |
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Format Inist (serveur)
| NO : | PASCAL 97-0460472 INIST |
|---|
| ET : | Perspectives on pandemics : A research agenda |
|---|
| AU : | KILBOURNE (E. D.); MONTO (Arnold S.) |
|---|
| AF : | Department of Microbiology and Immunology, New York Medical College/Valhalla, New York/Etats-Unis (1 aut.); Department of Epidemiology, University of Michigan School of Public Health/Ann Arbor, Michigan/Etats-Unis (1 aut.) |
|---|
| DT : | Publication en série; Congrès; Niveau analytique |
|---|
| SO : | The Journal of infectious diseases; ISSN 0022-1899; Coden JIDIAQ; Etats-Unis; Da. 1997; Vol. 176; No. SUP1; S29-S31; Bibl. 23 ref. |
|---|
| LA : | Anglais |
|---|
| EA : | During the 20th century, indisputable pandemics of influenza occurred in 1918, 1957, and 1968. The pandemics of 1957 (A/H2N2) and 1968 (A/H3N2) were associated with major antigenic changes in the virus, probably reflecting introduction by recombination of animal virus genes. The 1918 epidemic is beyond the reach of modern virology but, based on seroarcheology, appears to have been caused by a virus very similar to present swine (A/H1N1) influenza viruses. Changes in both principal antigens of the A/H1N1 subtype in 1947 resulted in total vaccine failure and pandemic spread of virus. On the basis of three periods of prevalence in the 20th century, A/H1N1 may be the "default" human virus, although the 39-year persistence of A/H3N2 to the present challenges this view. Only H1, H2, and H3 and N1 and N2 antigens have been found in human influenza viruses, but virologic history is too brief to preclude the contribution of other antigens to future pandemics. |
|---|
| CC : | 002A05C06 |
|---|
| FD : | Grippe; Homme; Epidémiologie; Historique; Perspective; Pandémie |
|---|
| FG : | Virose; Infection |
|---|
| ED : | Influenza; Human; Epidemiology; Case history; Perspective |
|---|
| EG : | Viral disease; Infection |
|---|
| GD : | Entwicklungsgeschichte |
|---|
| SD : | Gripe; Hombre; Epidemiología; Histórica; Perspectiva |
|---|
| LO : | INIST-2052.354000067739650060 |
|---|
| ID : | 97-0460472 |
|---|
Links to Exploration step
Pascal:97-0460472
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<EA>During the 20th century, indisputable pandemics of influenza occurred in 1918, 1957, and 1968. The pandemics of 1957 (A/H2N2) and 1968 (A/H3N2) were associated with major antigenic changes in the virus, probably reflecting introduction by recombination of animal virus genes. The 1918 epidemic is beyond the reach of modern virology but, based on seroarcheology, appears to have been caused by a virus very similar to present swine (A/H1N1) influenza viruses. Changes in both principal antigens of the A/H1N1 subtype in 1947 resulted in total vaccine failure and pandemic spread of virus. On the basis of three periods of prevalence in the 20th century, A/H1N1 may be the "default" human virus, although the 39-year persistence of A/H3N2 to the present challenges this view. Only H1, H2, and H3 and N1 and N2 antigens have been found in human influenza viruses, but virologic history is too brief to preclude the contribution of other antigens to future pandemics.</EA>
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