Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses
Identifieur interne : 001F30 ( Main/Merge ); précédent : 001F29; suivant : 001F31Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses
Auteurs : M. Tamura [États-Unis] ; R. G. Webster ; F. A. Ennis [États-Unis]Source :
- Journal of virology [ 0022-538X ] ; 1994.
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- Pascal (Inist)
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Abstract
Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses
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Pascal:94-0535195Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en" level="a">Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses</title><author><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M." last="Tamura">M. Tamura</name><affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Univ. Massachusetts medical cent., dep. medicine, div. infectious diseases</s1><s2>Worcester MA 01655</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>3 aut.</sZ></inist:fA14><country>États-Unis</country><wicri:noRegion>Worcester MA 01655</wicri:noRegion></affiliation></author><author><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R. G." last="Webster">R. G. Webster</name></author><author><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F. A." last="Ennis">F. A. Ennis</name><affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Univ. 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Tamura</name><affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Univ. Massachusetts medical cent., dep. medicine, div. infectious diseases</s1><s2>Worcester MA 01655</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>3 aut.</sZ></inist:fA14><country>États-Unis</country><wicri:noRegion>Worcester MA 01655</wicri:noRegion></affiliation></author><author><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R. G." last="Webster">R. G. Webster</name></author><author><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F. A." last="Ennis">F. A. Ennis</name><affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Univ. Massachusetts medical cent., dep. medicine, div. infectious diseases</s1><s2>Worcester MA 01655</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>3 aut.</sZ></inist:fA14><country>États-Unis</country><wicri:noRegion>Worcester MA 01655</wicri:noRegion></affiliation></author></analytic><series><title level="j" type="main">Journal of virology</title><title level="j" type="abbreviated">J. virol.</title><idno type="ISSN">0022-538X</idno><imprint><date when="1994">1994</date></imprint></series></biblStruct></sourceDesc><seriesStmt><title level="j" type="main">Journal of virology</title><title level="j" type="abbreviated">J. virol.</title><idno type="ISSN">0022-538X</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Antigen presentation</term><term>Cross reaction</term><term>Host virus relation</term><term>Influenzavirus A</term><term>Lymphoid cell</term><term>Mouse</term><term>Type specificity</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Influenzavirus A</term><term>Souris</term><term>Relation hôte virus</term><term>Présentation antigène</term><term>Spécificité type</term><term>Réaction croisée</term><term>Cellule lymphoïde</term><term>Lignée P388D1</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses</div></front></TEI><affiliations><list><country><li>États-Unis</li></country></list><tree><noCountry><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R. G." last="Webster">R. G. Webster</name></noCountry><country name="États-Unis"><noRegion><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M." last="Tamura">M. Tamura</name></noRegion><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F. A." last="Ennis">F. A. 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