Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses
Identifieur interne : 000094 ( PascalFrancis/Corpus ); précédent : 000093; suivant : 000095Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses
Auteurs : M. Tamura ; R. G. Webster ; F. A. EnnisSource :
- Journal of virology [ 0022-538X ] ; 1994.
Descripteurs français
- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses
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Format Inist (serveur)
NO : | PASCAL 94-0535195 INIST |
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ET : | Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses |
AU : | TAMURA (M.); WEBSTER (R. G.); ENNIS (F. A.) |
AF : | Univ. Massachusetts medical cent., dep. medicine, div. infectious diseases/Worcester MA 01655/Etats-Unis (1 aut., 3 aut.) |
DT : | Publication en série; Niveau analytique |
SO : | Journal of virology; ISSN 0022-538X; Etats-Unis; Da. 1994; Vol. 68; No. 6; Pp. 3499-3504; Bibl. 27 ref. |
LA : | Anglais |
EA : | Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses |
CC : | 002A05C04 |
FD : | Influenzavirus A; Souris; Relation hôte virus; Présentation antigène; Spécificité type; Réaction croisée; Cellule lymphoïde; Lignée P388D1 |
FG : | Influenzavirus; Orthomyxoviridae; Virus; Rodentia; Mammalia; Vertebrata; Réponse immune; Lignée cellulaire |
ED : | Influenzavirus A; Mouse; Host virus relation; Antigen presentation; Type specificity; Cross reaction; Lymphoid cell |
EG : | Influenzavirus; Orthomyxoviridae; Virus; Rodentia; Mammalia; Vertebrata; Immune response; Cell line |
SD : | Influenzavirus A; Ratón; Relación huesped virus; Presentación antígeno; Especificidad tipo; Reacción cruzada; Célula linfoide |
LO : | INIST-13592.354000045748450070 |
ID : | 94-0535195 |
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Pascal:94-0535195Le document en format XML
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<author><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M." last="Tamura">M. Tamura</name>
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<s2>Worcester MA 01655</s2>
<s3>USA</s3>
<sZ>1 aut.</sZ>
<sZ>3 aut.</sZ>
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<author><name sortKey="Webster, R G" sort="Webster, R G" uniqKey="Webster R" first="R. G." last="Webster">R. G. Webster</name>
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<author><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F. A." last="Ennis">F. A. Ennis</name>
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<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses</title>
<author><name sortKey="Tamura, M" sort="Tamura, M" uniqKey="Tamura M" first="M." last="Tamura">M. Tamura</name>
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<author><name sortKey="Ennis, F A" sort="Ennis, F A" uniqKey="Ennis F" first="F. A." last="Ennis">F. A. Ennis</name>
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<series><title level="j" type="main">Journal of virology</title>
<title level="j" type="abbreviated">J. virol.</title>
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<front><div type="abstract" xml:lang="en">Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses</div>
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<ET>Subtype cross-reactive, infection-enhancing antibody responses to influenza A viruses</ET>
<AU>TAMURA (M.); WEBSTER (R. G.); ENNIS (F. A.)</AU>
<AF>Univ. Massachusetts medical cent., dep. medicine, div. infectious diseases/Worcester MA 01655/Etats-Unis (1 aut., 3 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Journal of virology; ISSN 0022-538X; Etats-Unis; Da. 1994; Vol. 68; No. 6; Pp. 3499-3504; Bibl. 27 ref.</SO>
<LA>Anglais</LA>
<EA>Antibody-dependent enhancement of the uptake of influenza A virus by Fc receptor-bearing cells was analyzed by using virus strains of the three human influenza A virus subtypes, A/PR/8/34 (H1N1), A/Japan/305/57 (H2N2), and A/Port Chalmers/1/73 (H3N2). Immune sera obtained from mice following primary infection with an H1N1, H2N2, or H3N2 subtype virus neutralized only virus of the same subtype; however, immune sera augmented the uptake of virus across subtypes. Immune sera from HINI-infected mice augmented uptake of the homologous (H1N1) and H2N2 viruses. Antisera to the H2N2 virus augmented the uptake of virus of all subtypes (H1N1, H2N2, or H3N2). Antisera to the H3N2 virus augmented the uptake of the homologous (H3N2) and H2N2 viruses</EA>
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<SD>Influenzavirus A; Ratón; Relación huesped virus; Presentación antígeno; Especificidad tipo; Reacción cruzada; Célula linfoide</SD>
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