Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE− and the wild-type parental strain ALD
Identifieur interne : 000B67 ( Istex/Corpus ); précédent : 000B66; suivant : 000B68Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE− and the wild-type parental strain ALD
Auteurs : K. Ishikawa ; H. Nagai ; K. Katayama ; M. Tsutsui ; K. Tanabayashi ; K. Takeuchi ; M. Hishiyama ; A. Saitoh ; M. Takagi ; K. Gotoh ; M. Muramatsu ; A. YamadaSource :
- Archives of Virology [ 0304-8608 ] ; 1995-08-01.
English descriptors
- Teeft :
- Alfort strains, Amino, Amino acid sequence, Amino acid sequences, Amino acid substitutions, Amino acids, Assay laboratory, Attenuated, Cdna, Cholera, Cholera virus, Complete nucleotide sequences, Diarrhea virus, Dideoxy termination method, Entire sequences, Extra nucleotides, Genome, Independent clones, Molecular basis, National institute, Nucleotide, Nucleotide changes, Nucleotide sequence, Nucleotide sequences, Nucleotide substitutions, Open reading frame, Pestivirus genome, Serine protease domain, Sindbis virus, Substitution, Untranslated region, Vaccine, Vaccine strain, Viral, Virology.
Abstract
Summary: We have determined the complete nucleotide sequences of a live attenuated hog cholera virus (HCV) and its progenitor strain. The viral RNA of each strain consisted of 12 298 nucleotides including untranslated regions of 373 and 228 bases at the 5′ and 3′ end, respectively. There was a single large open reading frame spanning 11 697 nucleotides which could encode a large protein of 3 899 amino acids with a calculated molecular weight of 438-kDa. We have found 225 nucleotide difference between the two strains, of which six were located in the untranslated region. Four-sixths of these differences resulted in amino acid substitutions.
Url:
DOI: 10.1007/BF01322665
Links to Exploration step
ISTEX:888F6B8E79E3C3E53AD8F41F4F5726428212D63ELe document en format XML
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We have found 225 nucleotide difference between the two strains, of which six were located in the untranslated region. Four-sixths of these differences resulted in amino acid substitutions.</abstract><qualityIndicators><score>5.751</score><pdfWordCount>2503</pdfWordCount><pdfCharCount>15715</pdfCharCount><pdfVersion>1.3</pdfVersion><pdfPageCount>7</pdfPageCount><pdfPageSize>562 x 774 pts</pdfPageSize><refBibsNative>false</refBibsNative><abstractWordCount>104</abstractWordCount><abstractCharCount>647</abstractCharCount><keywordCount>0</keywordCount></qualityIndicators><title>Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE− and the wild-type parental strain ALD</title><pmid><json:string>7661692</json:string></pmid><genre><json:string>research-article</json:string></genre><host><title>Archives of 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name="istex:docType"></istex:docType><istex:document><Publisher><PublisherInfo><PublisherName>Springer-Verlag</PublisherName><PublisherLocation>Vienna</PublisherLocation></PublisherInfo><Journal><JournalInfo JournalProductType="ArchiveJournal" NumberingStyle="Unnumbered"><JournalID>705</JournalID><JournalPrintISSN>0304-8608</JournalPrintISSN><JournalElectronicISSN>1432-8798</JournalElectronicISSN><JournalTitle>Archives of Virology</JournalTitle><JournalAbbreviatedTitle>Archives of Virology</JournalAbbreviatedTitle><JournalSubjectGroup><JournalSubject Type="Primary">Biomedicine</JournalSubject><JournalSubject Type="Secondary">Medical Microbiology</JournalSubject><JournalSubject Type="Secondary">Virology</JournalSubject><JournalSubject Type="Secondary">Infectious Diseases</JournalSubject></JournalSubjectGroup></JournalInfo><Volume><VolumeInfo VolumeType="Regular" TocLevels="0"><VolumeIDStart>140</VolumeIDStart><VolumeIDEnd>140</VolumeIDEnd><VolumeIssueCount>12</VolumeIssueCount></VolumeInfo><Issue IssueType="Regular"><IssueInfo TocLevels="0"><IssueIDStart>8</IssueIDStart><IssueIDEnd>8</IssueIDEnd><IssueArticleCount>18</IssueArticleCount><IssueHistory><CoverDate><DateString>1995</DateString><Year>1995</Year><Month>8</Month></CoverDate></IssueHistory><IssueCopyright><CopyrightHolderName>Springer-Verlag</CopyrightHolderName><CopyrightYear>1995</CopyrightYear></IssueCopyright></IssueInfo><Article ID="Art4"><ArticleInfo Language="En" ArticleType="OriginalPaper" NumberingStyle="Unnumbered" TocLevels="0" ContainsESM="No"><ArticleID>BF01322665</ArticleID><ArticleDOI>10.1007/BF01322665</ArticleDOI><ArticleSequenceNumber>4</ArticleSequenceNumber><ArticleTitle Language="En">Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE<Superscript>−</Superscript> and the wild-type parental strain ALD</ArticleTitle><ArticleCategory>Original Papers</ArticleCategory><ArticleFirstPage>1385</ArticleFirstPage><ArticleLastPage>1391</ArticleLastPage><ArticleHistory><RegistrationDate><Year>2005</Year><Month>3</Month><Day>4</Day></RegistrationDate><Received><Year>1995</Year><Month>3</Month><Day>22</Day></Received><Accepted><Year>1995</Year><Month>4</Month><Day>21</Day></Accepted></ArticleHistory><ArticleCopyright><CopyrightHolderName>Springer-Verlag</CopyrightHolderName><CopyrightYear>1995</CopyrightYear></ArticleCopyright><ArticleGrants Type="Regular"><MetadataGrant Grant="OpenAccess"></MetadataGrant><AbstractGrant Grant="OpenAccess"></AbstractGrant><BodyPDFGrant Grant="Restricted"></BodyPDFGrant><BodyHTMLGrant Grant="Restricted"></BodyHTMLGrant><BibliographyGrant Grant="Restricted"></BibliographyGrant><ESMGrant Grant="Restricted"></ESMGrant></ArticleGrants><ArticleContext><JournalID>705</JournalID><VolumeIDStart>140</VolumeIDStart><VolumeIDEnd>140</VolumeIDEnd><IssueIDStart>8</IssueIDStart><IssueIDEnd>8</IssueIDEnd></ArticleContext></ArticleInfo><ArticleHeader><AuthorGroup><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>K.</GivenName><FamilyName>Ishikawa</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>H.</GivenName><FamilyName>Nagai</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff2"><AuthorName DisplayOrder="Western"><GivenName>K.</GivenName><FamilyName>Katayama</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>M.</GivenName><FamilyName>Tsutsui</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff3"><AuthorName DisplayOrder="Western"><GivenName>K.</GivenName><FamilyName>Tanabayashi</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff3"><AuthorName DisplayOrder="Western"><GivenName>K.</GivenName><FamilyName>Takeuchi</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff3"><AuthorName DisplayOrder="Western"><GivenName>M.</GivenName><FamilyName>Hishiyama</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>A.</GivenName><FamilyName>Saitoh</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>M.</GivenName><FamilyName>Takagi</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>K.</GivenName><FamilyName>Gotoh</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1"><AuthorName DisplayOrder="Western"><GivenName>M.</GivenName><FamilyName>Muramatsu</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff3"><AuthorName DisplayOrder="Western"><GivenName>A.</GivenName><FamilyName>Yamada</FamilyName></AuthorName></Author><Affiliation ID="Aff1"><OrgName>National Veterinary Assay Laboratory</OrgName><OrgAddress><City>Tokyo</City></OrgAddress></Affiliation><Affiliation ID="Aff2"><OrgDivision>Basic Research Division</OrgDivision><OrgName>Biomedical Laboratories, Inc.</OrgName><OrgAddress><City>Saitama</City></OrgAddress></Affiliation><Affiliation ID="Aff3"><OrgDivision>Department of Viral Disease and Vaccine Control</OrgDivision><OrgName>National Institute of Health</OrgName><OrgAddress><City>Tokyo</City><Country>Japan</Country></OrgAddress></Affiliation></AuthorGroup><Abstract ID="Abs1" Language="En"><Heading>Summary</Heading><Para>We have determined the complete nucleotide sequences of a live attenuated hog cholera virus (HCV) and its progenitor strain. The viral RNA of each strain consisted of 12 298 nucleotides including untranslated regions of 373 and 228 bases at the 5′ and 3′ end, respectively. There was a single large open reading frame spanning 11 697 nucleotides which could encode a large protein of 3 899 amino acids with a calculated molecular weight of 438-kDa. We have found 225 nucleotide difference between the two strains, of which six were located in the untranslated region. Four-sixths of these differences resulted in amino acid substitutions.</Para></Abstract></ArticleHeader><NoBody></NoBody></Article></Issue></Volume></Journal></Publisher></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE− and the wild-type parental strain ALD</title></titleInfo><titleInfo type="alternative" contentType="CDATA"><title>Comparison of the entire nucleotide and deduced amino acid sequences of the attenuated hog cholera vaccine strain GPE− and the wild-type parental strain ALD</title></titleInfo><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Ishikawa</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">H.</namePart><namePart type="family">Nagai</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Katayama</namePart><affiliation>Basic Research Division, Biomedical Laboratories, Inc., Saitama</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">Tsutsui</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Tanabayashi</namePart><affiliation>Department of Viral Disease and Vaccine Control, National Institute of Health, Tokyo, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Takeuchi</namePart><affiliation>Department of Viral Disease and Vaccine Control, National Institute of Health, Tokyo, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">Hishiyama</namePart><affiliation>Department of Viral Disease and Vaccine Control, National Institute of Health, Tokyo, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A.</namePart><namePart type="family">Saitoh</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">Takagi</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">K.</namePart><namePart type="family">Gotoh</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M.</namePart><namePart type="family">Muramatsu</namePart><affiliation>National Veterinary Assay Laboratory, Tokyo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A.</namePart><namePart type="family">Yamada</namePart><affiliation>Department of Viral Disease and Vaccine Control, National Institute of Health, Tokyo, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="OriginalPaper" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-1JC4F85T-7">research-article</genre><originInfo><publisher>Springer-Verlag</publisher><place><placeTerm type="text">Vienna</placeTerm></place><dateCreated encoding="w3cdtf">1995-03-22</dateCreated><dateIssued encoding="w3cdtf">1995-08-01</dateIssued><copyrightDate encoding="w3cdtf">1995</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><abstract lang="en">Summary: We have determined the complete nucleotide sequences of a live attenuated hog cholera virus (HCV) and its progenitor strain. The viral RNA of each strain consisted of 12 298 nucleotides including untranslated regions of 373 and 228 bases at the 5′ and 3′ end, respectively. There was a single large open reading frame spanning 11 697 nucleotides which could encode a large protein of 3 899 amino acids with a calculated molecular weight of 438-kDa. We have found 225 nucleotide difference between the two strains, of which six were located in the untranslated region. Four-sixths of these differences resulted in amino acid substitutions.</abstract><note>Original Papers</note><relatedItem type="host"><titleInfo><title>Archives of Virology</title></titleInfo><titleInfo type="abbreviated"><title>Archives of Virology</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><originInfo><publisher>Springer</publisher><dateIssued encoding="w3cdtf">1995-08-01</dateIssued><copyrightDate encoding="w3cdtf">1995</copyrightDate></originInfo><subject><genre>Biomedicine</genre><topic>Medical Microbiology</topic><topic>Virology</topic><topic>Infectious Diseases</topic></subject><identifier type="ISSN">0304-8608</identifier><identifier type="eISSN">1432-8798</identifier><identifier type="JournalID">705</identifier><identifier type="IssueArticleCount">18</identifier><identifier type="VolumeIssueCount">12</identifier><part><date>1995</date><detail type="volume"><number>140</number><caption>vol.</caption></detail><detail type="issue"><number>8</number><caption>no.</caption></detail><extent unit="pages"><start>1385</start><end>1391</end></extent></part><recordInfo><recordOrigin>Springer-Verlag, 1995</recordOrigin></recordInfo></relatedItem><identifier type="istex">888F6B8E79E3C3E53AD8F41F4F5726428212D63E</identifier><identifier type="ark">ark:/67375/1BB-8PQNFBJB-1</identifier><identifier type="DOI">10.1007/BF01322665</identifier><identifier type="ArticleID">BF01322665</identifier><identifier type="ArticleID">Art4</identifier><accessCondition type="use and reproduction" contentType="copyright">Springer-Verlag, 1995</accessCondition><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-3XSW68JL-F">springer</recordContentSource><recordOrigin>Springer-Verlag, 1995</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/ark:/67375/1BB-8PQNFBJB-1/record.json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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