Cyclosporin-A inhibits the expression of cyclooxygenase-2 in gingiva.
Identifieur interne : 001D94 ( Ncbi/Curation ); précédent : 001D93; suivant : 001D95Cyclosporin-A inhibits the expression of cyclooxygenase-2 in gingiva.
Auteurs : C-Y Chiang [République populaire de Chine] ; Y-T Chen ; F-M Hung ; H-P Tu ; M M-J Fu ; E. FuSource :
- Journal of periodontal research [ 0022-3484 ] ; 2007.
Descripteurs français
- KwdFr :
- Adulte, Animaux, Antienzymes (pharmacologie), Cellules cultivées, Cellules stromales (), Cellules stromales (enzymologie), Cellules épithéliales (), Cellules épithéliales (enzymologie), Ciclosporine (pharmacologie), Cyclooxygenase 2 (analyse), Dinoprostone (analyse), Facteur de nécrose tumorale alpha (analyse), Femelle, Fibroblastes (), Fibroblastes (enzymologie), Gencive (), Gencive (enzymologie), Humains, Inhibiteurs de la cyclooxygénase 2 (pharmacologie), Interleukine-1 bêta (analyse), Interleukine-6 (analyse), Mâchoire édentée (anatomopathologie), RT-PCR, Rat Sprague-Dawley, Rats, Technique de Western.
- MESH :
- analyse : Cyclooxygenase 2, Dinoprostone, Facteur de nécrose tumorale alpha, Interleukine-1 bêta, Interleukine-6.
- anatomopathologie : Mâchoire édentée.
- enzymologie : Cellules stromales, Cellules épithéliales, Fibroblastes, Gencive.
- pharmacologie : Antienzymes, Ciclosporine, Inhibiteurs de la cyclooxygénase 2.
- Adulte, Animaux, Cellules cultivées, Cellules stromales, Cellules épithéliales, Femelle, Fibroblastes, Gencive, Humains, RT-PCR, Rat Sprague-Dawley, Rats, Technique de Western.
English descriptors
- KwdEn :
- Adult, Animals, Blotting, Western, Cells, Cultured, Cyclooxygenase 2 (analysis), Cyclooxygenase 2 Inhibitors (pharmacology), Cyclosporine (pharmacology), Dinoprostone (analysis), Enzyme Inhibitors (pharmacology), Epithelial Cells (drug effects), Epithelial Cells (enzymology), Female, Fibroblasts (drug effects), Fibroblasts (enzymology), Gingiva (drug effects), Gingiva (enzymology), Humans, Interleukin-1beta (analysis), Interleukin-6 (analysis), Jaw, Edentulous (pathology), Rats, Rats, Sprague-Dawley, Reverse Transcriptase Polymerase Chain Reaction, Stromal Cells (drug effects), Stromal Cells (enzymology), Tumor Necrosis Factor-alpha (analysis).
- MESH :
- chemical , analysis : Cyclooxygenase 2, Dinoprostone, Interleukin-1beta, Interleukin-6, Tumor Necrosis Factor-alpha.
- chemical , pharmacology : Cyclooxygenase 2 Inhibitors, Cyclosporine, Enzyme Inhibitors.
- drug effects : Epithelial Cells, Fibroblasts, Gingiva, Stromal Cells.
- enzymology : Epithelial Cells, Fibroblasts, Gingiva, Stromal Cells.
- pathology : Jaw, Edentulous.
- Adult, Animals, Blotting, Western, Cells, Cultured, Female, Humans, Rats, Rats, Sprague-Dawley, Reverse Transcriptase Polymerase Chain Reaction.
Abstract
Various inflammatory mediators are involved in the development of cyclosporine A-induced gingival overgrowth. In this study, the gingival expression of cyclooxygenase-2 after cyclosporine A therapy was examined in vivo and in vitro.
DOI: 10.1111/j.1600-0765.2006.00967.x
PubMed: 17760822
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pubmed:17760822Le document en format XML
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<author><name sortKey="Chiang, C Y" sort="Chiang, C Y" uniqKey="Chiang C" first="C-Y" last="Chiang">C-Y Chiang</name>
<affiliation wicri:level="1"><nlm:affiliation>Department of Periodontology, School of Dentistry, National Defense Medical Center and Tri-Service General Hospital, Taipei, Taiwan, China.</nlm:affiliation>
<country xml:lang="fr">République populaire de Chine</country>
<wicri:regionArea>Department of Periodontology, School of Dentistry, National Defense Medical Center and Tri-Service General Hospital, Taipei, Taiwan</wicri:regionArea>
<wicri:noRegion>Taiwan</wicri:noRegion>
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<author><name sortKey="Chen, Y T" sort="Chen, Y T" uniqKey="Chen Y" first="Y-T" last="Chen">Y-T Chen</name>
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<author><name sortKey="Hung, F M" sort="Hung, F M" uniqKey="Hung F" first="F-M" last="Hung">F-M Hung</name>
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<term>Cyclooxygenase 2 Inhibitors (pharmacology)</term>
<term>Cyclosporine (pharmacology)</term>
<term>Dinoprostone (analysis)</term>
<term>Enzyme Inhibitors (pharmacology)</term>
<term>Epithelial Cells (drug effects)</term>
<term>Epithelial Cells (enzymology)</term>
<term>Female</term>
<term>Fibroblasts (drug effects)</term>
<term>Fibroblasts (enzymology)</term>
<term>Gingiva (drug effects)</term>
<term>Gingiva (enzymology)</term>
<term>Humans</term>
<term>Interleukin-1beta (analysis)</term>
<term>Interleukin-6 (analysis)</term>
<term>Jaw, Edentulous (pathology)</term>
<term>Rats</term>
<term>Rats, Sprague-Dawley</term>
<term>Reverse Transcriptase Polymerase Chain Reaction</term>
<term>Stromal Cells (drug effects)</term>
<term>Stromal Cells (enzymology)</term>
<term>Tumor Necrosis Factor-alpha (analysis)</term>
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<term>Animaux</term>
<term>Antienzymes (pharmacologie)</term>
<term>Cellules cultivées</term>
<term>Cellules stromales ()</term>
<term>Cellules stromales (enzymologie)</term>
<term>Cellules épithéliales ()</term>
<term>Cellules épithéliales (enzymologie)</term>
<term>Ciclosporine (pharmacologie)</term>
<term>Cyclooxygenase 2 (analyse)</term>
<term>Dinoprostone (analyse)</term>
<term>Facteur de nécrose tumorale alpha (analyse)</term>
<term>Femelle</term>
<term>Fibroblastes ()</term>
<term>Fibroblastes (enzymologie)</term>
<term>Gencive ()</term>
<term>Gencive (enzymologie)</term>
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<term>Inhibiteurs de la cyclooxygénase 2 (pharmacologie)</term>
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<term>Technique de Western</term>
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<term>Interleukin-1beta</term>
<term>Interleukin-6</term>
<term>Tumor Necrosis Factor-alpha</term>
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<term>Cyclosporine</term>
<term>Enzyme Inhibitors</term>
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<term>Dinoprostone</term>
<term>Facteur de nécrose tumorale alpha</term>
<term>Interleukine-1 bêta</term>
<term>Interleukine-6</term>
</keywords>
<keywords scheme="MESH" qualifier="anatomopathologie" xml:lang="fr"><term>Mâchoire édentée</term>
</keywords>
<keywords scheme="MESH" qualifier="drug effects" xml:lang="en"><term>Epithelial Cells</term>
<term>Fibroblasts</term>
<term>Gingiva</term>
<term>Stromal Cells</term>
</keywords>
<keywords scheme="MESH" qualifier="enzymologie" xml:lang="fr"><term>Cellules stromales</term>
<term>Cellules épithéliales</term>
<term>Fibroblastes</term>
<term>Gencive</term>
</keywords>
<keywords scheme="MESH" qualifier="enzymology" xml:lang="en"><term>Epithelial Cells</term>
<term>Fibroblasts</term>
<term>Gingiva</term>
<term>Stromal Cells</term>
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<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Jaw, Edentulous</term>
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<term>Ciclosporine</term>
<term>Inhibiteurs de la cyclooxygénase 2</term>
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<term>Animals</term>
<term>Blotting, Western</term>
<term>Cells, Cultured</term>
<term>Female</term>
<term>Humans</term>
<term>Rats</term>
<term>Rats, Sprague-Dawley</term>
<term>Reverse Transcriptase Polymerase Chain Reaction</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Adulte</term>
<term>Animaux</term>
<term>Cellules cultivées</term>
<term>Cellules stromales</term>
<term>Cellules épithéliales</term>
<term>Femelle</term>
<term>Fibroblastes</term>
<term>Gencive</term>
<term>Humains</term>
<term>RT-PCR</term>
<term>Rat Sprague-Dawley</term>
<term>Rats</term>
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<front><div type="abstract" xml:lang="en">Various inflammatory mediators are involved in the development of cyclosporine A-induced gingival overgrowth. In this study, the gingival expression of cyclooxygenase-2 after cyclosporine A therapy was examined in vivo and in vitro.</div>
</front>
</TEI>
</record>
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